Midori Masuda
- Molecular Biology
- Immunology top 10%
- Cellular and Molecular Neuroscience top 10%
- Endocrinology, Diabetes and Metabolism top 10%
- Cardiology and Cardiovascular Medicine top 10%
- Co-authors
- Yutaka KomiyamaHakuo TakahashiNoriko NishimuraTakashi MurakamiXian DongHiroshi EgawaHiroya MasakiMasamichi Yoshika
- Topics
- Coagulation, Bradykinin, Polyphosphates, and Angioedema (9 papers)Monoclonal and Polyclonal Antibodies Research (8 papers)Aldose Reductase and Taurine (7 papers)
- Journals
- NeurologyDiabetesJournal of Virology
- Partner nations
- JapanUnited StatesAustria
In The Last Decade
Midori Masuda
71 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 104
- Molecular Biology 575
- Immunology 221
- Cellular and Molecular Neuroscience 178
- Endocrinology, Diabetes and Metabolism 176
- Cardiology and Cardiovascular Medicine 158
Countries citing papers authored by Midori Masuda
This map shows the geographic impact of Midori Masuda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Midori Masuda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Midori Masuda more than expected).
Fields of papers citing papers by Midori Masuda
This network shows the impact of papers produced by Midori Masuda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Midori Masuda. The network helps show where Midori Masuda may publish in the future.
Co-authorship network of co-authors of Midori Masuda
This figure shows the co-authorship network connecting the top 25 collaborators of Midori Masuda. A scholar is included among the top collaborators of Midori Masuda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Midori Masuda. Midori Masuda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 93 | |
| 3 | 7 | |
| 4 | 3 | |
| 5 | 23 | |
| 6 | 37 | |
| 7 | 81 | |
| 8 | 5 | |
| 9 | 2 | |
| 10 | 11 | |
| 11 | 50 | |
| 12 | 55 | |
| 13 | 99 | |
| 14 | 10 | |
| 15 | 3 | |
| 16 | 5 | |
| 17 | 25 | |
| 18 | 9 | |
| 19 | [Role of taurine in neutrophil function]. | 4 |
| 20 | 3 |
About Midori Masuda
Midori Masuda is a scholar working on Genetics, Immunology and Hematology, having authored 73 papers that have together received 1.4k indexed citations. Recurring topics across this work include Coagulation, Bradykinin, Polyphosphates, and Angioedema (9 papers), Monoclonal and Polyclonal Antibodies Research (8 papers) and Aldose Reductase and Taurine (7 papers). The work is most often cited by research in Hematology (151 citations), Endocrinology, Diabetes and Metabolism (176 citations) and Immunology (221 citations). Midori Masuda has collaborated with scholars based in Japan, United States and Austria. Frequent co-authors include Yutaka Komiyama, Hakuo Takahashi, Noriko Nishimura, Takashi Murakami, Xian Dong, Hiroshi Egawa, Hiroya Masaki, Masamichi Yoshika, Nobuhiro Yuki and Michiaki Koga. Their work appears in journals such as Neurology, Diabetes and Journal of Virology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.