Michel Pons
- Genetics top 5%
- Molecular Biology
- Health, Toxicology and Mutagenesis top 5%
- Endocrinology, Diabetes and Metabolism top 10%
- Oncology
- Co-authors
- S. Stoney SimonsDidier GagnePatrick BalaguerAnne‐Marie BoussiouxJean‐Claude NicolasClaude CasellasHélène FenetFranck Comunale
- Topics
- Estrogen and related hormone effects (21 papers)Receptor Mechanisms and Signaling (6 papers)Free Radicals and Antioxidants (4 papers)
- Journals
- The Science of The Total EnvironmentJournal of Agricultural and Food ChemistryBiochemical and Biophysical Research Communications
- Partner nations
- FranceUnited StatesJapan
In The Last Decade
Michel Pons
34 papers receiving 943 citations
Peers
Comparison fields: 5 of 97
- Genetics 499
- Molecular Biology 470
- Health, Toxicology and Mutagenesis 223
- Endocrinology, Diabetes and Metabolism 122
- Oncology 120
Countries citing papers authored by Michel Pons
This map shows the geographic impact of Michel Pons's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michel Pons with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michel Pons more than expected).
Fields of papers citing papers by Michel Pons
This network shows the impact of papers produced by Michel Pons. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michel Pons. The network helps show where Michel Pons may publish in the future.
Co-authorship network of co-authors of Michel Pons
This figure shows the co-authorship network connecting the top 25 collaborators of Michel Pons. A scholar is included among the top collaborators of Michel Pons based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michel Pons. Michel Pons is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 12 | |
| 2 | 16 | |
| 3 | 11 | |
| 4 | 7 | |
| 5 | 8 | |
| 6 | 37 | |
| 7 | 10 | |
| 8 | 26 | |
| 9 | 24 | |
| 10 | 29 | |
| 11 | 16 | |
| 12 | 9 | |
| 13 | 45 | |
| 14 | 9 | |
| 15 | 6 | |
| 16 | 14 | |
| 17 | 36 | |
| 18 | 26 | |
| 19 | 81 | |
| 20 | 32 |
About Michel Pons
Michel Pons is a scholar working on Toxicology, Genetics and Molecular Biology, having authored 34 papers that have together received 995 indexed citations. Recurring topics across this work include Estrogen and related hormone effects (21 papers), Receptor Mechanisms and Signaling (6 papers) and Free Radicals and Antioxidants (4 papers). The work is most often cited by research in Genetics (499 citations), Health, Toxicology and Mutagenesis (223 citations) and Toxicology (37 citations). Michel Pons has collaborated with scholars based in France, United States and Japan. Frequent co-authors include S. Stoney Simons, Didier Gagne, Patrick Balaguer, Anne‐Marie Boussioux, Jean‐Claude Nicolas, Claude Casellas, Hélène Fenet, Franck Comunale, David Johnson and Ediz Demirpençe. Their work appears in journals such as The Science of The Total Environment, Journal of Agricultural and Food Chemistry and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.