Michael Martinez
Impact in
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- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
Papers in
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- Fibroblast Growth Factor Research 1
- Protein Kinase Regulation and GTPase Signaling 1
- RNA regulation and disease 1
- CRISPR and Genetic Engineering 1
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- Metabolism and Genetic Disorders 2
- Co-authors
- Mark Kester (1 shared paper)Emmanuel Coroneos (1 shared paper)Cary O. Harding (2 shared papers)Gerald Schwank (1 shared paper)Beat Thöny (1 shared paper)Andrik J. Aschoff (1 shared paper)Martin I. Resnick (1 shared paper)Gregory T. MacLennan (1 shared paper)
- Journals
- Journal of Inherited Metabolic Disease (2 papers)Cell Reports (1 paper)Hepatology (1 paper)Journal of Biological Chemistry (1 paper)American Journal of Roentgenology (1 paper)
- Partner nations
- United StatesSwedenSwitzerland
In The Last Decade
Michael Martinez
6 papers receiving 300 citations
Peers
Comparison fields: 5 of 52
- Cell Biology 62
- Physiology 17
- Molecular Biology 228
- Clinical Biochemistry 21
- Developmental Neuroscience 8
Countries citing papers authored by Michael Martinez
This map shows the geographic impact of Michael Martinez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Martinez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Martinez more than expected).
Fields of papers citing papers by Michael Martinez
This network shows the impact of papers produced by Michael Martinez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Martinez. The network helps show where Michael Martinez may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael Martinez, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1995 | 197 | |
| 2 | 2019 | 38 | |
| 3 | 2001 | 28 | |
| 4 | 2023 | 20 | |
| 5 | 2021 | 12 | |
| 6 | 2023 | 9 |
About Michael Martinez
Michael Martinez is a scholar working on Molecular Biology, Clinical Biochemistry, Genetics, Genetics and Cancer Research, having authored 6 papers that have together received 304 indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (2 papers), Virus-based gene therapy research (2 papers), Fibroblast Growth Factor Research (1 paper), MicroRNA in disease regulation (1 paper), Protein Kinase Regulation and GTPase Signaling (1 paper), RNA regulation and disease (1 paper), CRISPR and Genetic Engineering (1 paper) and Glioma Diagnosis and Treatment (1 paper). The work is most often cited by research in Cell Biology (62 citations), Physiology (17 citations), Molecular Biology (228 citations), Clinical Biochemistry (21 citations) and Developmental Neuroscience (8 citations). Michael Martinez has collaborated with scholars based in United States, Sweden and Switzerland. Frequent co-authors include Mark Kester, Emmanuel Coroneos, Cary O. Harding, Gerald Schwank, Beat Thöny, Andrik J. Aschoff, Martin I. Resnick, Gregory T. MacLennan, Jonathan S. Lewin and Aaron Sulman. Their work appears in journals such as Journal of Inherited Metabolic Disease, Cell Reports, Hepatology, Journal of Biological Chemistry and American Journal of Roentgenology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.