Michael L. Phelan
- Molecular Biology top 5%
- Genetics top 10%
- Pathology and Forensic Medicine top 5%
- Immunology
- Oncology
- Co-authors
- Robert E. KingstonGeeta J. NarlikarSaı̈d SifMark FeatherstoneIsabel RambaldiGavin R. SchnitzlerPaul A. WadeTom Owen‐Hughes
- Topics
- Chromatin Remodeling and Cancer (5 papers)Genomics and Chromatin Dynamics (4 papers)interferon and immune responses (3 papers)
- Partner nations
- United StatesCanadaRussia
In The Last Decade
Michael L. Phelan
13 papers receiving 1.5k citations
Hit Papers
Peers
Comparison fields: 5 of 69
- Molecular Biology 1.4k
- Genetics 234
- Pathology and Forensic Medicine 224
- Immunology 107
- Oncology 94
Countries citing papers authored by Michael L. Phelan
This map shows the geographic impact of Michael L. Phelan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael L. Phelan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael L. Phelan more than expected).
Fields of papers citing papers by Michael L. Phelan
This network shows the impact of papers produced by Michael L. Phelan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael L. Phelan. The network helps show where Michael L. Phelan may publish in the future.
Co-authorship network of co-authors of Michael L. Phelan
This figure shows the co-authorship network connecting the top 25 collaborators of Michael L. Phelan. A scholar is included among the top collaborators of Michael L. Phelan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael L. Phelan. Michael L. Phelan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 35 | |
| 3 | 109 | |
| 4 | 94 | |
| 5 | 220 | |
| 6 | 3 | |
| 7 | 178 | |
| 8 | Reconstitution of a Core Chromatin Remodeling Complex from SWI/SNF Subunitsbreakdown → | 513 |
| 9 | 56 | |
| 10 | 193 | |
| 11 | Phelan ML, Rambaldi I, Featherstone MS. Cooperative interactions between HOX and PBX proteins mediated by a conserved peptide motif. Mol Cell Biol 15: 3989-3997 | 25 |
| 12 | 16 | |
| 13 | 39 |
About Michael L. Phelan
Michael L. Phelan is a scholar working on Molecular Biology, Immunology and Pathology and Forensic Medicine, having authored 13 papers that have together received 1.5k indexed citations. Recurring topics across this work include Chromatin Remodeling and Cancer (5 papers), Genomics and Chromatin Dynamics (4 papers) and interferon and immune responses (3 papers). The work is most often cited by research in Molecular Biology (1.4k citations), Pathology and Forensic Medicine (224 citations) and Genetics (234 citations). Michael L. Phelan has collaborated with scholars based in United States, Canada and Russia. Frequent co-authors include Robert E. Kingston, Geeta J. Narlikar, Saı̈d Sif, Mark Featherstone, Isabel Rambaldi, Gavin R. Schnitzler, Paul A. Wade, Tom Owen‐Hughes, Kristina M. Havas and Andrew Flaus. Their work appears in journals such as Cell, Journal of Biological Chemistry and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.