Michael Frech

897 total citations
12 papers, 230 citations indexed

About

Michael Frech is a scholar working on Immunology, Molecular Biology and Surgery. According to data from OpenAlex, Michael Frech has authored 12 papers receiving a total of 230 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 4 papers in Molecular Biology and 3 papers in Surgery. Recurrent topics in Michael Frech's work include IL-33, ST2, and ILC Pathways (5 papers), Immune Cell Function and Interaction (4 papers) and Eosinophilic Esophagitis (3 papers). Michael Frech is often cited by papers focused on IL-33, ST2, and ILC Pathways (5 papers), Immune Cell Function and Interaction (4 papers) and Eosinophilic Esophagitis (3 papers). Michael Frech collaborates with scholars based in Germany, Japan and Austria. Michael Frech's co-authors include Mario M. Zaiss, Georg Schett, Kerstin Sarter, Jürgen Rech, Melanie Hagen, Ulrike Steffen, Koray Taşçılar, Maria Sokolova, Holger Bang and Yasunori Omata and has published in prestigious journals such as International Journal of Molecular Sciences, Frontiers in Immunology and Nutrients.

In The Last Decade

Michael Frech

11 papers receiving 228 citations

Peers

Michael Frech

MB

IMMUN

RHEUM

SURGE

GENET

Amanda Duhlin Sweden

Mike Fiorino United States

Stacey Bellemore Canada

João Farela Neves Portugal

T. Boyé France

Timothée Fettrelet Switzerland

Shaista Rashid Denmark

Ali Kassem Egypt

Martina Bakele Germany

Alexander Heiseke Germany

Amanda Duhlin Sweden

Michael Frech

78 ×0.8

MB

135 ×1.4

IMMUN

41 ×0.9

RHEUM

20 ×0.4

SURGE

29 ×0.8

GENET

Citations per year, relative to Michael Frech Michael Frech (= 1×) peers Amanda Duhlin

Countries citing papers authored by Michael Frech

Since Specialization

Citations

This map shows the geographic impact of Michael Frech's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Frech with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Frech more than expected).

Fields of papers citing papers by Michael Frech

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Frech. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Frech. The network helps show where Michael Frech may publish in the future.

Co-authorship network of co-authors of Michael Frech

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Frech. A scholar is included among the top collaborators of Michael Frech based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Frech. Michael Frech is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

1.

Schälter, Fabian, Vugar Azizov, Michael Frech, et al.. (2024). CCL19‐Positive Lymph Node Stromal Cells Govern the Onset of Inflammatory Arthritis via Tropomyosin Receptor Kinase. Arthritis & Rheumatology. 76(6). 857–868.

2.

Azizov, Vugar, Michael Frech, Jörg Hofmann, et al.. (2023). Alcohol-sourced acetate impairs T cell function by promoting cortactin acetylation. iScience. 26(7). 107230–107230. 3 indexed citations

3.

Frech, Michael, Heike Danzer, Vugar Azizov, et al.. (2023). Butyrophilin 2a2 (Btn2a2) expression on thymic epithelial cells promotes central T cell tolerance and prevents autoimmune disease. Journal of Autoimmunity. 139. 103071–103071. 5 indexed citations

4.

Dürholz, Kerstin, Eva Schmid, Michael Frech, et al.. (2022). Microbiota-Derived Propionate Modulates Megakaryopoiesis and Platelet Function. Frontiers in Immunology. 13. 908174–908174. 14 indexed citations

5.

Frech, Michael, Yasunori Omata, Stefan Wirtz, et al.. (2022). Btn2a2 Regulates ILC2–T Cell Cross Talk in Type 2 Immune Responses. Frontiers in Immunology. 13. 757436–757436. 8 indexed citations

6.

Schälter, Fabian, Michael Frech, Kerstin Dürholz, et al.. (2022). Acetate, a metabolic product of Heligmosomoides polygyrus, facilitates intestinal epithelial barrier breakdown in a FFAR2-dependent manner. International Journal for Parasitology. 52(9). 591–601. 6 indexed citations

7.

Omata, Yasunori, Michael Frech, Taku Saito, et al.. (2022). Inflammatory Arthritis and Bone Metabolism Regulated by Type 2 Innate and Adaptive Immunity. International Journal of Molecular Sciences. 23(3). 1104–1104. 8 indexed citations

8.

Frech, Michael, et al.. (2021). RANKL-Induced Btn2a2 – A T Cell Immunomodulatory Molecule – During Osteoclast Differentiation Fine-Tunes Bone Resorption. Frontiers in Endocrinology. 12. 685060–685060. 2 indexed citations

9.

Frech, Michael, et al.. (2020). An in vivo gene delivery approach for the isolation of reasonable numbers of type 2 innate lymphoid cells. MethodsX. 7. 101054–101054. 4 indexed citations

10.

Omata, Yasunori, Michael Frech, Sébastien Lucas, et al.. (2020). Type 2 innate lymphoid cells inhibit the differentiation of osteoclasts and protect from ovariectomy-induced bone loss. Bone. 136. 115335–115335. 22 indexed citations

11.

Bang, Holger, Melanie Hagen, Michael Frech, et al.. (2019). The Role of Dietary Fiber in Rheumatoid Arthritis Patients: A Feasibility Study. Nutrients. 11(10). 2392–2392. 100 indexed citations

12.

Omata, Yasunori, Michael Frech, Sébastien Lucas, et al.. (2018). Group 2 Innate Lymphoid Cells Attenuate Inflammatory Arthritis and Protect from Bone Destruction in Mice. Cell Reports. 24(1). 169–180. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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