Michael C. Pride

1.7k total citations
16 papers, 885 citations indexed

About

Michael C. Pride is a scholar working on Genetics, Cognitive Neuroscience and Molecular Biology. According to data from OpenAlex, Michael C. Pride has authored 16 papers receiving a total of 885 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Genetics, 8 papers in Cognitive Neuroscience and 5 papers in Molecular Biology. Recurrent topics in Michael C. Pride's work include Genetics and Neurodevelopmental Disorders (9 papers), Autism Spectrum Disorder Research (7 papers) and Epigenetics and DNA Methylation (3 papers). Michael C. Pride is often cited by papers focused on Genetics and Neurodevelopmental Disorders (9 papers), Autism Spectrum Disorder Research (7 papers) and Epigenetics and DNA Methylation (3 papers). Michael C. Pride collaborates with scholars based in United States, Canada and United Kingdom. Michael C. Pride's co-authors include Jill L. Silverman, Jacqueline N. Crawley, Kyle Puhger, Brian C. Trainor, Salah A. Baker, Nycole A. Copping, Rosalina Villalon Landeros, Nicholas Knoblauch, Janine M. LaSalle and Mu Yang and has published in prestigious journals such as PLoS ONE, Scientific Reports and Human Molecular Genetics.

In The Last Decade

Michael C. Pride

16 papers receiving 880 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael C. Pride United States 13 376 333 307 246 243 16 885
Natallia V. Riddick United States 17 226 0.6× 210 0.6× 299 1.0× 238 1.0× 261 1.1× 21 849
Hewlet G. McFarlane United States 8 328 0.9× 483 1.5× 319 1.0× 264 1.1× 317 1.3× 13 1.1k
Christopher L. Muller United States 10 276 0.7× 493 1.5× 261 0.9× 146 0.6× 229 0.9× 10 929
Ksenia Meyza Poland 18 245 0.7× 446 1.3× 234 0.8× 347 1.4× 281 1.2× 29 965
Carmen Agustín‐Pavón Spain 19 189 0.5× 252 0.8× 308 1.0× 328 1.3× 421 1.7× 40 1.1k
Daniel G. Smith United States 9 266 0.7× 331 1.0× 271 0.9× 157 0.6× 255 1.0× 15 768
Hala Harony‐Nicolas United States 17 547 1.5× 572 1.7× 380 1.2× 347 1.4× 185 0.8× 29 1.2k
Alexia M. Thomas United States 11 615 1.6× 644 1.9× 456 1.5× 128 0.5× 349 1.4× 13 1.2k
Amy E. Clipperton‐Allen United States 14 308 0.8× 273 0.8× 276 0.9× 509 2.1× 103 0.4× 20 1.1k
Caterina Michetti Italy 14 218 0.6× 151 0.5× 252 0.8× 186 0.8× 155 0.6× 22 639

Countries citing papers authored by Michael C. Pride

Since Specialization
Citations

This map shows the geographic impact of Michael C. Pride's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael C. Pride with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael C. Pride more than expected).

Fields of papers citing papers by Michael C. Pride

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael C. Pride. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael C. Pride. The network helps show where Michael C. Pride may publish in the future.

Co-authorship network of co-authors of Michael C. Pride

This figure shows the co-authorship network connecting the top 25 collaborators of Michael C. Pride. A scholar is included among the top collaborators of Michael C. Pride based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael C. Pride. Michael C. Pride is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Pride, Michael C., et al.. (2024). Surgical restabilization reduces the progression of post-traumatic osteoarthritis initiated by ACL rupture in mice. Osteoarthritis and Cartilage. 32(8). 909–920. 1 indexed citations
2.
Berg, Elizabeth L., Michael C. Pride, Stela P. Petkova, et al.. (2020). Developmental exposure to near roadway pollution produces behavioral phenotypes relevant to neurodevelopmental disorders in juvenile rats. Translational Psychiatry. 10(1). 289–289. 21 indexed citations
3.
Petkova, Stela P., Michael C. Pride, Carolyn Klocke, et al.. (2020). Cyclin D2-knock-out mice with attenuated dentate gyrus neurogenesis have robust deficits in long-term memory formation. Scientific Reports. 10(1). 8204–8204. 6 indexed citations
4.
Adhikari, Anna, Nycole A. Copping, Michael C. Pride, et al.. (2018). Cognitive deficits in the Snord116 deletion mouse model for Prader-Willi syndrome. Neurobiology of Learning and Memory. 165. 106874–106874. 45 indexed citations
5.
Matt, Lucas, Lyndsey M. Kirk, George Chenaux, et al.. (2018). SynDIG4/Prrt1 Is Required for Excitatory Synapse Development and Plasticity Underlying Cognitive Function. Cell Reports. 22(9). 2246–2253. 44 indexed citations
6.
Jones, Karen L., Michael C. Pride, Mu Yang, et al.. (2018). Autism-specific maternal autoantibodies produce behavioral abnormalities in an endogenous antigen-driven mouse model of autism. Molecular Psychiatry. 25(11). 2994–3009. 44 indexed citations
7.
Berg, Elizabeth L., Nycole A. Copping, Michael C. Pride, et al.. (2018). Developmental social communication deficits in the Shank3 rat model of phelan‐mcdermid syndrome and autism spectrum disorder. Autism Research. 11(4). 587–601. 69 indexed citations
8.
Ciernia, Annie Vogel, Dag H. Yasui, Michael C. Pride, et al.. (2018). MeCP2 isoform e1 mutant mice recapitulate motor and metabolic phenotypes of Rett syndrome. Human Molecular Genetics. 27(23). 4077–4093. 9 indexed citations
9.
Dhamne, Sameer C., Jill L. Silverman, Mustafa Q. Hameed, et al.. (2017). Replicable in vivo physiological and behavioral phenotypes of the Shank3B null mutant mouse model of autism. Molecular Autism. 8(1). 26–26. 105 indexed citations
10.
Copping, Nycole A., Mohammad Saharul Islam, Dorota Żółkowska, et al.. (2017). Neuronal overexpression of Ube3a isoform 2 causes behavioral impairments and neuroanatomical pathology relevant to 15q11.2-q13.3 duplication syndrome. Human Molecular Genetics. 26(20). 3995–4010. 52 indexed citations
11.
Ciernia, Annie Vogel, Michael C. Pride, Blythe Durbin‐Johnson, et al.. (2017). Early motor phenotype detection in a female mouse model of Rett syndrome is improved by cross-fostering. Human Molecular Genetics. 26(10). 1839–1854. 25 indexed citations
12.
Leach, Prescott T., et al.. (2016). Normal Performance of Fmr1 Mice on a Touchscreen Delayed Nonmatching to Position Working Memory Task. eNeuro. 3(1). ENEURO.0143–15.2016. 18 indexed citations
13.
Silverman, Jill L., et al.. (2015). GABAB Receptor Agonist R-Baclofen Reverses Social Deficits and Reduces Repetitive Behavior in Two Mouse Models of Autism. Neuropsychopharmacology. 40(9). 2228–2239. 172 indexed citations
14.
Bales, Karen L., Marjorie Solomon, Suma Jacob, et al.. (2014). Long-term exposure to intranasal oxytocin in a mouse autism model. Translational Psychiatry. 4(11). e480–e480. 102 indexed citations
15.
McMackin, Marissa Z., et al.. (2014). Effects of kappa opioid receptors on conditioned place aversion and social interaction in males and females. Behavioural Brain Research. 262. 84–93. 47 indexed citations
16.
Trainor, Brian C., et al.. (2011). Sex Differences in Social Interaction Behavior Following Social Defeat Stress in the Monogamous California Mouse (Peromyscus californicus). PLoS ONE. 6(2). e17405–e17405. 125 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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