Michael C. Elia
- Molecular Biology
- Cancer Research top 10%
- Plant Science
- Health, Toxicology and Mutagenesis top 10%
- Oncology
- Co-authors
- Matthews O. BradleyJohn G. DeLucaRichard D. StorerTroy W. McKelveyAndrew R. KraynakWarren W. NicholsEvangelos N. MoudrianakisJohn E. Barnum
- Topics
- DNA Repair Mechanisms (4 papers)Carcinogens and Genotoxicity Assessment (4 papers)DNA and Nucleic Acid Chemistry (3 papers)
- Partner nations
- United States
In The Last Decade
Michael C. Elia
12 papers receiving 471 citations
Peers
Comparison fields: 5 of 79
- Molecular Biology 310
- Cancer Research 218
- Plant Science 94
- Health, Toxicology and Mutagenesis 79
- Oncology 53
Countries citing papers authored by Michael C. Elia
This map shows the geographic impact of Michael C. Elia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael C. Elia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael C. Elia more than expected).
Fields of papers citing papers by Michael C. Elia
This network shows the impact of papers produced by Michael C. Elia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael C. Elia. The network helps show where Michael C. Elia may publish in the future.
Co-authorship network of co-authors of Michael C. Elia
This figure shows the co-authorship network connecting the top 25 collaborators of Michael C. Elia. A scholar is included among the top collaborators of Michael C. Elia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael C. Elia. Michael C. Elia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 83 | |
| 2 | 84 | |
| 3 | 8 | |
| 4 | 40 | |
| 5 | 13 | |
| 6 | 42 | |
| 7 | Influence of chromatin structure on the induction of DNA double strand breaks by ionizing radiation. | 89 |
| 8 | 65 | |
| 9 | 2 | |
| 10 | Induction of sister-chromatid exchanges by polycyclic aromatic hydrocarbons following metabolic activation with phorbol ester-stimulated human polymorphonuclear leukocytes. | 10 |
| 11 | 32 | |
| 12 | 31 |
About Michael C. Elia
Michael C. Elia is a scholar working on Cancer Research, Biotechnology and Plant Science, having authored 12 papers that have together received 499 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (4 papers), Carcinogens and Genotoxicity Assessment (4 papers) and DNA and Nucleic Acid Chemistry (3 papers). The work is most often cited by research in Cancer Research (218 citations), Chemical Health and Safety (5 citations) and Health, Toxicology and Mutagenesis (79 citations). Michael C. Elia has collaborated with scholars based in United States. Frequent co-authors include Matthews O. Bradley, John G. DeLuca, Richard D. Storer, Troy W. McKelvey, Andrew R. Kraynak, Warren W. Nichols, Evangelos N. Moudrianakis, John E. Barnum, Tamra Goodrow and Thomas H. Eickbush. Their work appears in journals such as Journal of Biological Chemistry, Analytical Biochemistry and Pharmacology & Therapeutics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.