Michael B. Cohen

559 total citations
13 papers, 440 citations indexed

About

Michael B. Cohen is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Michael B. Cohen has authored 13 papers receiving a total of 440 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Immunology and 3 papers in Oncology. Recurrent topics in Michael B. Cohen's work include Cell death mechanisms and regulation (5 papers), Ubiquitin and proteasome pathways (3 papers) and Cancer-related Molecular Pathways (3 papers). Michael B. Cohen is often cited by papers focused on Cell death mechanisms and regulation (5 papers), Ubiquitin and proteasome pathways (3 papers) and Cancer-related Molecular Pathways (3 papers). Michael B. Cohen collaborates with scholars based in United States. Michael B. Cohen's co-authors include Oskar W. Rokhlin, Rebecca A. Glover, René Feyereisen, Sergei Syrbu, Mary A. Schuler, May R. Berenbaum, Andreas Gewies, Paul M. Heidger, David M. Lubaroff and William C. Powell and has published in prestigious journals such as Gene, Cancer Letters and Journal of Chemical Ecology.

In The Last Decade

Michael B. Cohen

13 papers receiving 428 citations

Peers

Michael B. Cohen
Comparison fields: 5 of 74
  • Molecular Biology 309
  • Insect Science 86
  • Oncology 78
  • Genetics 59
  • Cancer Research 55
Replace Shean‐Jaw Chiou with:
Shean‐Jaw Chiou Taiwan
Çağrı Bodur Türkiye
Meng Lin China
Zhuanzhuan Su United States
Ian J. Forsythe Canada
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Yuqian Feng China
Agnieszka Jeleń Poland
Shean‐Jaw Chiou Taiwan View profile →
Citations per field, relative to Michael B. Cohen
Michael B. Cohen · 1×
Citations per year, relative to Michael B. Cohen
Michael B. Cohen · 1×

Countries citing papers authored by Michael B. Cohen

Since Specialization
Citations

This map shows the geographic impact of Michael B. Cohen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael B. Cohen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael B. Cohen more than expected).

Fields of papers citing papers by Michael B. Cohen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael B. Cohen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael B. Cohen. The network helps show where Michael B. Cohen may publish in the future.

Co-authorship network of co-authors of Michael B. Cohen

This figure shows the co-authorship network connecting the top 25 collaborators of Michael B. Cohen. A scholar is included among the top collaborators of Michael B. Cohen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael B. Cohen. Michael B. Cohen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
# Work Indexed citations
1 35
2
Overexpression of BAD potentiates sensitivity to tumor necrosis factor-related apoptosis-inducing ligand treatment in the prostatic carcinoma cell line LNCaP.
19
3 20
4
Prostatic intraepithelial neoplasia in mice with conditional disruption of the retinoid X receptor alpha allele in the prostate epithelium.
74
5
Cytochrome c is involved in Fas-mediated apoptosis of prostatic carcinoma cell lines.
38
6 6
7
Fas-mediated apoptosis in human prostatic carcinoma cell lines occurs via activation of caspase-8 and caspase-7.
66
8
Dominant nature of the resistance to Fas- and tumor necrosis factor-alpha-mediated apoptosis in human prostatic carcinoma cell lines.
22
9 3
10 28
11 46
12
Gross and microscopic pathology of induced prostatic complex tumors arising in Lobund-Wistar rats.
26
13 57

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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