Michael A. Rea

3.4k total citations · 1 hit paper
37 papers, 2.9k citations indexed

About

Michael A. Rea is a scholar working on Endocrine and Autonomic Systems, Cellular and Molecular Neuroscience and Cognitive Neuroscience. According to data from OpenAlex, Michael A. Rea has authored 37 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Endocrine and Autonomic Systems, 24 papers in Cellular and Molecular Neuroscience and 14 papers in Cognitive Neuroscience. Recurrent topics in Michael A. Rea's work include Circadian rhythm and melatonin (26 papers), Photoreceptor and optogenetics research (19 papers) and Sleep and Wakefulness Research (12 papers). Michael A. Rea is often cited by papers focused on Circadian rhythm and melatonin (26 papers), Photoreceptor and optogenetics research (19 papers) and Sleep and Wakefulness Research (12 papers). Michael A. Rea collaborates with scholars based in United States, Netherlands and Japan. Michael A. Rea's co-authors include E. Todd Weber, Gary E. Pickard, Robert L. Gannon, Martha U. Gillette, J. David Glass, Chen Dong, Lia E. Faiman, Jian Ding, Magdi Selim and Luı́s de Lecea and has published in prestigious journals such as Science, Nature Communications and Neuron.

In The Last Decade

Michael A. Rea

36 papers receiving 2.8k citations

Hit Papers

A novel adenylyl cyclase-activating serotonin receptor (5... 1993 2026 2004 2015 1993 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael A. Rea United States 28 1.9k 1.7k 932 625 479 37 2.9k
Rebecca A. Prosser United States 31 2.1k 1.1× 1.6k 0.9× 1.1k 1.2× 498 0.8× 566 1.2× 63 3.0k
Charlotte von Gall Germany 29 2.4k 1.3× 1.1k 0.6× 741 0.8× 476 0.8× 718 1.5× 77 3.1k
Shigenori Watanabe Japan 32 1.0k 0.5× 1.9k 1.1× 778 0.8× 897 1.4× 520 1.1× 158 3.2k
Olivier Bosler France 30 1.0k 0.5× 1.5k 0.9× 478 0.5× 721 1.2× 331 0.7× 81 2.5k
Kunio Kitahama France 38 1.3k 0.7× 1.9k 1.1× 1.1k 1.2× 760 1.2× 402 0.8× 122 3.7k
Johanna E. Chesham United Kingdom 30 3.2k 1.7× 1.4k 0.8× 781 0.8× 597 1.0× 1.2k 2.4× 44 4.0k
Marco Brancaccio United Kingdom 14 1.6k 0.8× 712 0.4× 524 0.6× 272 0.4× 577 1.2× 21 2.1k
Anne Bérod France 34 1.0k 0.5× 2.2k 1.3× 753 0.8× 1.2k 1.9× 426 0.9× 70 3.8k
Hoa A. Lam United States 20 625 0.3× 932 0.5× 768 0.8× 622 1.0× 323 0.7× 27 2.4k
Thomas S. Hnasko United States 35 1.2k 0.6× 3.0k 1.7× 1.3k 1.4× 1.9k 3.1× 745 1.6× 64 5.1k

Countries citing papers authored by Michael A. Rea

Since Specialization
Citations

This map shows the geographic impact of Michael A. Rea's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael A. Rea with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael A. Rea more than expected).

Fields of papers citing papers by Michael A. Rea

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael A. Rea. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael A. Rea. The network helps show where Michael A. Rea may publish in the future.

Co-authorship network of co-authors of Michael A. Rea

This figure shows the co-authorship network connecting the top 25 collaborators of Michael A. Rea. A scholar is included among the top collaborators of Michael A. Rea based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael A. Rea. Michael A. Rea is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bollu, Lakshmi Reddy, Fei Su, Rintu Thomas, et al.. (2018). Targeted reduction of the EGFR protein, but not inhibition of its kinase activity, induces mitophagy and death of cancer cells through activation of mTORC2 and Akt. Oncogenesis. 7(1). 5–5. 30 indexed citations
2.
Li, Da‐Qiang, Suresh B. Pakala, Sirigiri Divijendra Natha Reddy, et al.. (2013). Metastasis-associated protein 1 is an integral component of the circadian molecular machinery. Nature Communications. 4(1). 2545–2545. 17 indexed citations
3.
Miki, Takao, Zhi–Xiang Xu, Misty Chen‐Goodspeed, et al.. (2012). PML regulates PER2 nuclear localization and circadian function. The EMBO Journal. 31(6). 1427–1439. 50 indexed citations
4.
Heller, Daniel A., et al.. (2004). Patterned networks of mouse hippocampal neurons on peptide-coated gold surfaces. Biomaterials. 26(8). 883–889. 53 indexed citations
5.
Hallworth, Richard, Matthew J. Cato, Costa M. Colbert, & Michael A. Rea. (2002). Presynaptic adenosine A1 receptors regulate retinohypothalamic neurotransmission in the hamster suprachiasmatic nucleus. Journal of Neurobiology. 52(3). 230–240. 28 indexed citations
6.
Weber, E. Todd, et al.. (2001). Adenosine A1 receptors regulate the response of the hamster circadian clock to light. European Journal of Pharmacology. 414(1). 45–53. 30 indexed citations
7.
Rea, Michael A. & Gary E. Pickard. (2000). A 5-HT1B receptor agonist inhibits light-induced suppression of pineal melatonin production. Brain Research. 858(2). 424–428. 19 indexed citations
8.
9.
Gannon, Robert L., et al.. (1995). GABAergic modulation of optic nerve-evoked field potentials in the rat suprachiasmatic nucleus. Brain Research. 694(1-2). 264–270. 17 indexed citations
10.
Rea, Michael A., Jose Barrera, J. David Glass, & Robert L. Gannon. (1995). Serotonergic potentiation of photic phase shifts of the circadian activity rhythm. Neuroreport. 6(10). 1417–1420. 72 indexed citations
11.
Weber, E. Todd, Robert L. Gannon, & Michael A. Rea. (1995). cGMP-dependent protein kinase inhibitor blocks light-induced phase advances of circadian rhythms in vivo. Neuroscience Letters. 197(3). 227–230. 69 indexed citations
13.
Gannon, Robert L. & Michael A. Rea. (1993). Glutamate receptor immunoreactivity in the rat suprachiasmatic nucleus. Brain Research. 622(1-2). 337–342. 29 indexed citations
14.
Lovenberg, Timothy W., Luı́s de Lecea, Joseph D. Miller, et al.. (1993). A novel adenylyl cyclase-activating serotonin receptor (5-HT7) implicated in the regulation of mammalian circadian rhythms. Neuron. 11(3). 449–458. 590 indexed citations breakdown →
15.
Selim, Magdi, et al.. (1993). Serotonergic inhibition of light-induced fos protein expression and extracellular glutamate in the suprachiasmatic nuclei. Brain Research. 621(2). 181–188. 104 indexed citations
17.
Rea, Michael A.. (1990). VIP-stimulated cyclic AMP accumulation in the suprachiasmatic hypothalamus. Brain Research Bulletin. 25(6). 843–847. 22 indexed citations
18.
Dines, K.A., et al.. (1990). Ultrasound and the Blood-Brain Barrier. Advances in experimental medicine and biology. 267. 369–381. 54 indexed citations
19.
Rea, Michael A.. (1989). Light increases Fos-related protein immunoreactivity in the rat suprachiasmatic nuclei. Brain Research Bulletin. 23(6). 577–581. 219 indexed citations
20.
Claiborne, Brenda J., Michael A. Rea, & David M. Terrian. (1989). Detection of zinc in isolated nerve terminals using a modified Timm's sulfide-silver method. Journal of Neuroscience Methods. 30(1). 17–22. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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