Mengling Chang

701 total citations
25 papers, 573 citations indexed

About

Mengling Chang is a scholar working on Molecular Biology, Surgery and Rehabilitation. According to data from OpenAlex, Mengling Chang has authored 25 papers receiving a total of 573 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Surgery and 5 papers in Rehabilitation. Recurrent topics in Mengling Chang's work include Wound Healing and Treatments (5 papers), Electrospun Nanofibers in Biomedical Applications (5 papers) and Vascular Tumors and Angiosarcomas (2 papers). Mengling Chang is often cited by papers focused on Wound Healing and Treatments (5 papers), Electrospun Nanofibers in Biomedical Applications (5 papers) and Vascular Tumors and Angiosarcomas (2 papers). Mengling Chang collaborates with scholars based in China and Taiwan. Mengling Chang's co-authors include Min Xing, Jiang Chang, Zhiguang Huan, Endian Wang, Q. N. Xu, Zengding Zhou, Lei Yi, Feng Bao, Yu Zhang and Feng Guo and has published in prestigious journals such as ACS Applied Materials & Interfaces, Free Radical Biology and Medicine and Nanoscale.

In The Last Decade

Mengling Chang

23 papers receiving 567 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mengling Chang China 12 155 125 124 107 94 25 573
Ziqi Huang China 15 76 0.5× 230 1.8× 211 1.7× 196 1.8× 118 1.3× 48 832
Yunxiao Zhu China 9 211 1.4× 193 1.5× 211 1.7× 110 1.0× 102 1.1× 21 651
Zhixiao Yao China 18 71 0.5× 104 0.8× 102 0.8× 147 1.4× 176 1.9× 29 786
Meijun Tan China 10 104 0.7× 119 1.0× 248 2.0× 100 0.9× 112 1.2× 16 460
Libing Dai China 15 55 0.4× 213 1.7× 101 0.8× 170 1.6× 54 0.6× 31 646
Linfeng Mao China 12 53 0.3× 149 1.2× 84 0.7× 219 2.0× 71 0.8× 23 658
Hua Zhao China 13 75 0.5× 115 0.9× 89 0.7× 98 0.9× 49 0.5× 65 468
Yunpeng Zhao China 17 77 0.5× 72 0.6× 127 1.0× 204 1.9× 58 0.6× 32 765
Pengfei Tian China 11 101 0.7× 97 0.8× 111 0.9× 134 1.3× 25 0.3× 25 438

Countries citing papers authored by Mengling Chang

Since Specialization
Citations

This map shows the geographic impact of Mengling Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mengling Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mengling Chang more than expected).

Fields of papers citing papers by Mengling Chang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mengling Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mengling Chang. The network helps show where Mengling Chang may publish in the future.

Co-authorship network of co-authors of Mengling Chang

This figure shows the co-authorship network connecting the top 25 collaborators of Mengling Chang. A scholar is included among the top collaborators of Mengling Chang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mengling Chang. Mengling Chang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chen, Siyuan, Lu Lin, Mengling Chang, et al.. (2025). Thermal-crosslinked acellular dermal matrix combined with adipose-derived stem cells to regenerate vascularized adipose tissue. Biomedical Materials. 20(2). 25020–25020.
2.
Chang, Mengling, Yi Lei, Zengding Zhou, et al.. (2023). GEF-H1/RhoA signaling pathway mediates pro-inflammatory effects of NF-κB on CD40L-induced pulmonary endothelial cells. Molecular Immunology. 157. 42–52. 2 indexed citations
3.
Wei, Chiu‐Chi, et al.. (2023). Assessing Knowledge Quality Using Fuzzy MCDM Model. Mathematics. 11(17). 3673–3673. 3 indexed citations
4.
Yi, Xin, Mengling Chang, Zengding Zhou, et al.. (2023). LPS induces SGPP2 to participate metabolic reprogramming in endothelial cells. Free Radical Biology and Medicine. 208. 780–793. 3 indexed citations
5.
Chang, Mengling, et al.. (2022). Adipose-Derived Stem Cells for the Treatment of Diabetic Wound: From Basic Study to Clinical Application. Frontiers in Endocrinology. 13. 882469–882469. 19 indexed citations
6.
Ding, Yi, et al.. (2022). A Comparative Study on Two Types of Porcine Acellular Dermal Matrix Sponges Prepared by Thermal Crosslinking and Thermal-Glutaraldehyde Crosslinking Matrix Microparticles. Frontiers in Bioengineering and Biotechnology. 10. 938798–938798. 9 indexed citations
7.
Yu, Xin, et al.. (2021). Construction of a prognostic model for lung squamous cell carcinoma based on seven N6-methylandenosine-related autophagy genes. Mathematical Biosciences & Engineering. 18(5). 6709–6723. 3 indexed citations
8.
Chang, Mengling, Zengding Zhou, Lei Yi, et al.. (2020). Factors Influencing Length of Hospital Stay and Predictors Affecting Probability of Requiring Surgery in Severely Pediatric Burn Patients. Journal of Burn Care & Research. 41(6). 1165–1171. 3 indexed citations
9.
Yi, Lei, et al.. (2019). Suppressive Effects of GSS on Lipopolysaccharide-Induced Endothelial Cell Injury and ALI via TNF-αand IL-6. Mediators of Inflammation. 2019. 1–13. 14 indexed citations
10.
Yi, Lei, et al.. (2019). Genistein‐3′‐sodium sulphonate protects against lipopolysaccharide‐induced lung vascular endothelial cell apoptosis and acute lung injury via BCL‐2 signalling. Journal of Cellular and Molecular Medicine. 24(1). 1022–1035. 18 indexed citations
11.
Chang, Mengling, et al.. (2019). Exosome-delivered syndecan-1 rescues acute lung injury via a FAK/p190RhoGAP/RhoA/ROCK/NF-κB signaling axis and glycocalyx enhancement. Experimental Cell Research. 384(1). 111596–111596. 51 indexed citations
13.
Yi, Lei, et al.. (2017). GSK-3Beta-Dependent Activation of GEF-H1/ROCK Signaling Promotes LPS-Induced Lung Vascular Endothelial Barrier Dysfunction and Acute Lung Injury. Frontiers in Cellular and Infection Microbiology. 7. 357–357. 17 indexed citations
15.
Huang, Xiaoqin, et al.. (2017). Relation between dynamic changes of platelet counts and 30-day mortality in severely burned patients. Platelets. 30(2). 158–163. 10 indexed citations
16.
Yi, Lei, et al.. (2016). Lipopolysaccharide Induces Human Pulmonary Micro-Vascular Endothelial Apoptosis via the YAP Signaling Pathway. Frontiers in Cellular and Infection Microbiology. 6. 133–133. 27 indexed citations
17.
Chang, Mengling, Tianxiang Ouyang, Jun Lin, et al.. (2015). Potential Molecular Mechanisms Involved in 5-Aminolevulinic Acid–Based Photodynamic Therapy against Human Hypertrophic Scars. Plastic & Reconstructive Surgery. 136(4). 715–727. 20 indexed citations
18.
Chang, Mengling, Tianxiang Ouyang, Miao Xu, et al.. (2015). Combination of propranolol and sclerotherapy for treatment of infantile parotid hemangiomas.. PubMed. 8(7). 10865–74. 5 indexed citations
19.
Ouyang, Tianxiang, et al.. (2014). Propranolol enhanced adipogenesis instead of induction of apoptosis of hemangiomas stem cells.. PubMed. 7(7). 3809–17. 23 indexed citations
20.
Chang, Mengling, et al.. (1994). [Bacteriostasis of rhizoma Coptidis combined with trimethoprim (TMPO)].. PubMed. 19(12). 748–9, 764.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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