Melissa Holtz

1.4k total citations
11 papers, 1.1k citations indexed

About

Melissa Holtz is a scholar working on Hematology, Genetics and Rheumatology. According to data from OpenAlex, Melissa Holtz has authored 11 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Hematology, 10 papers in Genetics and 8 papers in Rheumatology. Recurrent topics in Melissa Holtz's work include Chronic Lymphocytic Leukemia Research (10 papers), Chronic Myeloid Leukemia Treatments (10 papers) and Eosinophilic Disorders and Syndromes (8 papers). Melissa Holtz is often cited by papers focused on Chronic Lymphocytic Leukemia Research (10 papers), Chronic Myeloid Leukemia Treatments (10 papers) and Eosinophilic Disorders and Syndromes (8 papers). Melissa Holtz collaborates with scholars based in United States, Switzerland and United Kingdom. Melissa Holtz's co-authors include Ravi Bhatia, Stephen J. Forman, Charles L. Sawyers, Marilyn L. Slovak, Ning Niu, Rachel Gray, David S. Snyder, Daniel A. Arber, Su Chu and Feiyu Zhang and has published in prestigious journals such as Blood, Cancer Research and Leukemia.

In The Last Decade

Melissa Holtz

11 papers receiving 1.1k citations

Peers

Melissa Holtz
Gabriele Gugliotta Italy
Allen Lin United States
F-X Mahon France
Annamaria Galietta Italy
Benjamin Hanfstein Germany
Matthew S. Zabriskie United States
YinWei Ho United States
Sabrina Colarossi Italy
Mahnaz Paktinat United States
Po Yee Mak United States
Gabriele Gugliotta Italy
Melissa Holtz
Citations per year, relative to Melissa Holtz Melissa Holtz (= 1×) peers Gabriele Gugliotta

Countries citing papers authored by Melissa Holtz

Since Specialization
Citations

This map shows the geographic impact of Melissa Holtz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Melissa Holtz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Melissa Holtz more than expected).

Fields of papers citing papers by Melissa Holtz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Melissa Holtz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Melissa Holtz. The network helps show where Melissa Holtz may publish in the future.

Co-authorship network of co-authors of Melissa Holtz

This figure shows the co-authorship network connecting the top 25 collaborators of Melissa Holtz. A scholar is included among the top collaborators of Melissa Holtz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Melissa Holtz. Melissa Holtz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
König, Helmut, Melissa Holtz, Hardik Modi, et al.. (2008). Enhanced BCR-ABL kinase inhibition does not result in increased inhibition of downstream signaling pathways or increased growth suppression in CML progenitors. Leukemia. 22(4). 748–755. 87 indexed citations
2.
Holtz, Melissa, Stephen J. Forman, & Ravi Bhatia. (2007). Growth Factor Stimulation Reduces Residual Quiescent Chronic Myelogenous Leukemia Progenitors Remaining after Imatinib Treatment. Cancer Research. 67(3). 1113–1120. 60 indexed citations
3.
König, Helmut, Melissa Holtz, Hardik Modi, et al.. (2006). Nilotinib Inhibits Bcr-Abl Kinase Activity in CML Progenitor Cells More Effectively Than Imatinib but Is Equipotent in Inducing Growth Inhibition.. Blood. 108(11). 744–744. 1 indexed citations
4.
5.
Holtz, Melissa, Stephen J. Forman, & Ravi Bhatia. (2005). Nonproliferating CML CD34+ progenitors are resistant to apoptosis induced by a wide range of proapoptotic stimuli. Leukemia. 19(6). 1034–1041. 108 indexed citations
6.
7.
Holtz, Melissa, Stephen J. Forman, & Ravi Bhatia. (2004). Effect of Growth Factor Stimulation on Imatinib-Mediated Proliferation Inhibition and Apoptosis of CML CD34+ Cells.. Blood. 104(11). 2967–2967. 1 indexed citations
8.
Chu, Su, Melissa Holtz, Mamta Gupta, & Ravi Bhatia. (2003). BCR/ABL kinase inhibition by imatinib mesylate enhances MAP kinase activity in chronic myelogenous leukemia CD34+ cells. Blood. 103(8). 3167–3174. 142 indexed citations
9.
Holtz, Melissa & Ravi Bhatia. (2003). Effect of Imatinib Mesylate on Chronic Myelogenous Leukemia Hematopoietic Progenitor Cells. Leukemia & lymphoma. 45(2). 237–245. 16 indexed citations
10.
11.
Holtz, Melissa, Marilyn L. Slovak, Feiyu Zhang, et al.. (2002). Imatinib mesylate (STI571) inhibits growth of primitive malignant progenitors in chronic myelogenous leukemia through reversal of abnormally increased proliferation. Blood. 99(10). 3792–3800. 213 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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