Maxine Tevendale

485 total citations
8 papers, 417 citations indexed

About

Maxine Tevendale is a scholar working on Molecular Biology, Genetics and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Maxine Tevendale has authored 8 papers receiving a total of 417 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Genetics and 3 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Maxine Tevendale's work include Genetic Syndromes and Imprinting (5 papers), Epigenetics and DNA Methylation (5 papers) and Prenatal Screening and Diagnostics (3 papers). Maxine Tevendale is often cited by papers focused on Genetic Syndromes and Imprinting (5 papers), Epigenetics and DNA Methylation (5 papers) and Prenatal Screening and Diagnostics (3 papers). Maxine Tevendale collaborates with scholars based in United Kingdom and Italy. Maxine Tevendale's co-authors include Anne C. Ferguson‐Smith, Shuji Takada, Pantelis Georgiades, Martina Paulsen, Tom C. Freeman, Elizabeth A. Campbell, Martin H. Johnson, Marie Watkins, E. J. Knowles and Simão Teixeira da Rocha and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Current Biology and Developmental Biology.

In The Last Decade

Maxine Tevendale

8 papers receiving 411 citations

Peers

Maxine Tevendale

MB

GENET

PPCH

CR

ONCOL

Liora Z. Strichman-Almashanu United States

Stephanie Dallaire United States

Uta‐Dorothee Immel Germany

Shane C. Quinonez United States

Jennifer M. Huang United States

Pishun Li China

Steven Schonberg United States

Hideaki Chiyo Japan

Kamilla Schlade‐Bartusiak Canada

Erina Inoue Japan

Liora Z. Strichman-Almashanu United States

Maxine Tevendale

289 ×0.8

MB

156 ×0.6

GENET

50 ×0.4

PPCH

28 ×0.5

CR

38 ×0.8

ONCOL

Citations per year, relative to Maxine Tevendale Maxine Tevendale (= 1×) peers Liora Z. Strichman-Almashanu

Countries citing papers authored by Maxine Tevendale

Since Specialization

Citations

This map shows the geographic impact of Maxine Tevendale's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maxine Tevendale with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maxine Tevendale more than expected).

Fields of papers citing papers by Maxine Tevendale

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maxine Tevendale. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maxine Tevendale. The network helps show where Maxine Tevendale may publish in the future.

Co-authorship network of co-authors of Maxine Tevendale

This figure shows the co-authorship network connecting the top 25 collaborators of Maxine Tevendale. A scholar is included among the top collaborators of Maxine Tevendale based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maxine Tevendale. Maxine Tevendale is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Li, Wenjing, Brian J. Ferguson, Walid T. Khaled, et al.. (2009). PML depletion disrupts normal mammary gland development and skews the composition of the mammary luminal cell progenitor pool. Proceedings of the National Academy of Sciences. 106(12). 4725–4730. 46 indexed citations

2.

Rocha, Simão Teixeira da, Maxine Tevendale, E. J. Knowles, et al.. (2007). Restricted co-expression of Dlk1 and the reciprocally imprinted non-coding RNA, Gtl2: Implications for cis-acting control. Developmental Biology. 306(2). 810–823. 69 indexed citations

3.

Tevendale, Maxine, et al.. (2007). The Beginning of the End: Death Signaling in Early Involution. Journal of Mammary Gland Biology and Neoplasia. 12(1). 3–13. 50 indexed citations

4.

Matthews, James R., et al.. (2007). Caspase-dependent proteolytic cleavage of STAT3α in ES cells, in mammary glands undergoing forced involution and in breast cancer cell lines. BMC Cancer. 7(1). 29–29. 9 indexed citations

5.

Tevendale, Maxine, Marie Watkins, C. Rasberry, B.M. Cattanach, & Anne C. Ferguson‐Smith. (2006). Analysis of mouse conceptuses with uniparental duplication/deficiency for distal chromosome 12: comparison with chromosome 12 uniparental disomy and implications for genomic imprinting. Cytogenetic and Genome Research. 113(1-4). 215–222. 24 indexed citations

6.

Ferguson‐Smith, Anne C., et al.. (2003). Genomic imprinting—insights from studies in mice. Seminars in Cell and Developmental Biology. 14(1). 43–49. 19 indexed citations

7.

Ferguson‐Smith, Anne C., Maxine Tevendale, Pantelis Georgiades, & Valérie Grandjean. (2002). Balanced Translocations for the Analysis of Imprinted Regions of the Mouse Genome. Humana Press eBooks. 181. 41–54. 5 indexed citations

8.

Takada, Shuji, Maxine Tevendale, Pantelis Georgiades, et al.. (2000). Delta-like and Gtl2 are reciprocally expressed, differentially methylated linked imprinted genes on mouse chromosome 12. Current Biology. 10(18). 1135–1138. 195 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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