Maurizia Valli

1.4k total citations
53 papers, 1.0k citations indexed

About

Maurizia Valli is a scholar working on Genetics, Molecular Biology and Rheumatology. According to data from OpenAlex, Maurizia Valli has authored 53 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Genetics, 16 papers in Molecular Biology and 15 papers in Rheumatology. Recurrent topics in Maurizia Valli's work include Connective tissue disorders research (33 papers), Bone and Dental Protein Studies (12 papers) and Protease and Inhibitor Mechanisms (12 papers). Maurizia Valli is often cited by papers focused on Connective tissue disorders research (33 papers), Bone and Dental Protein Studies (12 papers) and Protease and Inhibitor Mechanisms (12 papers). Maurizia Valli collaborates with scholars based in Italy, Germany and United Kingdom. Maurizia Valli's co-authors include Monica Mottes, Giuseppe Cetta, Franco Antoniazzi, Ruggero Tenni, Marco Trinchera, Anna Bardoni, Antonio Rossi, Simona Viglio, Antonella Forlino and Antonella Sangalli and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and FEBS Letters.

In The Last Decade

Maurizia Valli

52 papers receiving 990 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Maurizia Valli Italy	19	504	426	266	144	132	53	1.0k
Massimiliano Monticone Italy	17	366 0.7×	476 1.1×	190 0.7×	161 1.1×	156 1.2×	34	1.0k
Marnisa Sricholpech United States	9	188 0.4×	420 1.0×	180 0.7×	157 1.1×	109 0.8×	13	902
Phyllis LuValle United States	21	308 0.6×	809 1.9×	472 1.8×	241 1.7×	256 1.9×	31	1.4k
Andrew M. Ho United States	13	272 0.5×	715 1.7×	383 1.4×	157 1.1×	240 1.8×	16	1.7k
Masanao Tsuda United States	9	127 0.3×	536 1.3×	368 1.4×	248 1.7×	64 0.5×	14	940
Takashi Shimoaka Japan	9	131 0.3×	580 1.4×	683 2.6×	211 1.5×	226 1.7×	9	1.3k
B. Font France	20	208 0.4×	596 1.4×	107 0.4×	238 1.7×	58 0.4×	38	1.1k
Chi Faucheu United States	10	132 0.3×	1.0k 2.4×	128 0.5×	89 0.6×	242 1.8×	12	1.3k
Hiromitsu Yamamoto Japan	14	98 0.2×	528 1.2×	214 0.8×	183 1.3×	110 0.8×	36	929
J. Michael Engle United States	12	245 0.5×	230 0.5×	180 0.7×	196 1.4×	98 0.7×	15	910

Countries citing papers authored by Maurizia Valli

Since Specialization

Citations

This map shows the geographic impact of Maurizia Valli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maurizia Valli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maurizia Valli more than expected).

Fields of papers citing papers by Maurizia Valli

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maurizia Valli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maurizia Valli. The network helps show where Maurizia Valli may publish in the future.

Co-authorship network of co-authors of Maurizia Valli

This figure shows the co-authorship network connecting the top 25 collaborators of Maurizia Valli. A scholar is included among the top collaborators of Maurizia Valli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maurizia Valli. Maurizia Valli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Caraffi, Stefano Giuseppe, Ivan Ivanovski, Marzia Pollazzon, et al.. (2019). Severe Peripheral Joint Laxity is a Distinctive Clinical Feature of Spondylodysplastic-Ehlers-Danlos Syndrome (EDS)-B4GALT7 and Spondylodysplastic-EDS-B3GALT6. Genes. 10(10). 799–799. 15 indexed citations

Mangione, P. Patrizia, Guglielmo Verona, Alessandra Corazza, et al.. (2018). Plasminogen activation triggers transthyretin amyloidogenesis in vitro. Journal of Biological Chemistry. 293(37). 14192–14199. 76 indexed citations

Valli, Maurizia, Aileen M. Barnes, Simona Viglio, et al.. (2011). Deficiency of CRTAP in non‐lethal recessive osteogenesis imperfecta reduces collagen deposition into matrix. Clinical Genetics. 82(5). 453–459. 23 indexed citations

Sala, Alberto, Monica Campagnoli, Maurizia Valli, et al.. (2009). Identification of the amniotic fluid insulin‐like growth factor binding protein‐1 phosphorylation sites and propensity to proteolysis of the isoforms. FEBS Journal. 276(20). 6033–6046. 17 indexed citations

Viglio, Simona, Nicoletta Zoppi, Antonella Sangalli, et al.. (2008). Rescue of Migratory Defects of Ehlers–Danlos Syndrome Fibroblasts In Vitro by Type V Collagen but not Insulin-Like Binding Protein-1. Journal of Investigative Dermatology. 128(8). 1915–1919. 24 indexed citations

Tedeschi, Elisa, Monica Mottes, Maurizia Valli, et al.. (2006). Osteogenesis imperfecta: clinical, biochemical and molecular findings. Clinical Genetics. 70(2). 131–139. 40 indexed citations

Genta, Ida, C. Colonna, Paola Perugini, et al.. (2005). Evaluation of bioadhesive performance of chitosan derivatives as films for buccal application. Journal of Drug Delivery Science and Technology. 15(6). 459–463. 10 indexed citations

Salvini, Roberta, Anna Bardoni, Maurizia Valli, & Marco Trinchera. (2001). β1,3-Galactosyltransferase β3Gal-T5 Acts on the GlcNAcβ1→3Galβ1→4GlcNAcβ1→R Sugar Chains of Carcinoembryonic Antigen and Other N-Linked Glycoproteins and Is Down-regulated in Colon Adenocarcinomas. Journal of Biological Chemistry. 276(5). 3564–3573. 50 indexed citations

Bardoni, Anna, Maurizia Valli, & Marco Trinchera. (1999). Differential expression of β1,3galactosyltransferases in human colon cells derived from adenocarcinomas or normal mucosa¹. FEBS Letters. 451(1). 75–80. 17 indexed citations

10.

Valli, Maurizia, et al.. (1998). β‐1,3‐galactosyltransferase and α‐1,2‐fucosyltransferase involved in the biosynthesis of type‐1‐chain carbohydrate antigens in human colon adenocarcinoma cell lines. European Journal of Biochemistry. 256(2). 494–501. 15 indexed citations

11.

Mottes, Monica, et al.. (1998). Four new cases of lethal osteogenesis imperfecta due to glycine substitutions in COL1A1 and genes. Human Mutation. 12(1). 71–72.

12.

Mottes, Monica, et al.. (1998). Four new cases of lethal Osteogenesis Imperfecta due to glycine substitutions in the COL1A1 and COL1A2 genes. 12. 71–72. 2 indexed citations

13.

Zolezzi, Francesca, Maurizia Valli, Massimo Clementi, et al.. (1997). Mutation producing alternative splicing of exon 26 in the COL1A2 gene causes type IV osteogenesis imperfecta with intrafamilial clinical variability. American Journal of Medical Genetics. 71(3). 366–370. 14 indexed citations

14.

Zanaboni, Giuseppe, Simona Viglio, Katharine M. Dyne, et al.. (1997). Direct monitoring of prolidase activity in cultured skin fibroblasts using capillary electrophoresis. Journal of Chromatography B Biomedical Sciences and Applications. 695(1). 77–84. 13 indexed citations

15.

Zolezzi, Francesca, Antonella Forlino, Monica Mottes, et al.. (1995). A 931 + 2T → C transition in one COL1A2 allele causes exon 16 skipping in PROα2(I) mRNA and produces moderately severe OI. Human Mutation. 6(3). 268–271. 4 indexed citations

16.

Mottes, Monica, Antonella Sangalli, Maurizia Valli, et al.. (1994). A base substitution at IVS-19 3?-end splice junction causes exon 20 skipping in pro?2(I) collagen mRNA and produces mild osteogenesis imperfecta. Human Genetics. 93(6). 681–7. 13 indexed citations

17.

Forlino, Antonella, Francesca Zolezzi, Maurizia Valli, et al.. (1994). Severe (type III) osteogenesis imperfecta due to glycine substitutions in the central domain of the collagen triple helix. Human Molecular Genetics. 3(12). 2201–2206. 12 indexed citations

18.

Mottes, Monica, Antonella Sangalli, Maurizia Valli, et al.. (1992). Mild dominant osteogenesis imperfecta with intrafamilial variability: the cause is a serine for glycine ?1(I) 901 substitution in a type-I collagen gene. Human Genetics. 89(5). 480–4. 24 indexed citations

19.

Valli, Maurizia, Monica Mottes, Ruggero Tenni, et al.. (1991). A de novo G to T transversion in a pro-alpha 1 (I) collagen gene for a moderate case of osteogenesis imperfecta. Substitution of cysteine for glycine 178 in the triple helical domain. Journal of Biological Chemistry. 266(3). 1872–1878. 40 indexed citations

20.

Valli, Maurizia, Ruggero Tenni, & Giuseppe Cetta. (1990). Moderately Severe Osteogenesis Imperfecta: Biochemical Studies Showing Variable Defect Localization in the Triple-Helical Domain of Type I Collagen. Matrix. 10(3). 200–205. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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