Giuseppe Cetta

2.7k total citations
105 papers, 2.1k citations indexed

About

Giuseppe Cetta is a scholar working on Genetics, Molecular Biology and Oncology. According to data from OpenAlex, Giuseppe Cetta has authored 105 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 48 papers in Genetics, 40 papers in Molecular Biology and 26 papers in Oncology. Recurrent topics in Giuseppe Cetta's work include Connective tissue disorders research (47 papers), Peptidase Inhibition and Analysis (22 papers) and Protease and Inhibitor Mechanisms (20 papers). Giuseppe Cetta is often cited by papers focused on Connective tissue disorders research (47 papers), Peptidase Inhibition and Analysis (22 papers) and Protease and Inhibitor Mechanisms (20 papers). Giuseppe Cetta collaborates with scholars based in Italy, United States and Germany. Giuseppe Cetta's co-authors include Antonio Rossi, Antonella Forlino, Ruggero Tenni, Giuseppe Zanaboni, Paolo Iadarola, Maurizia Valli, Simona Viglio, Katharine M. Dyne, Andrea Superti‐Furga and Anna Lupi and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and Blood.

In The Last Decade

Giuseppe Cetta

105 papers receiving 2.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Giuseppe Cetta Italy 28 671 631 489 363 338 105 2.1k
John M. McPherson United States 34 1.1k 1.7× 285 0.5× 471 1.0× 138 0.4× 509 1.5× 66 3.1k
Katsuto Tamai Japan 32 1.7k 2.5× 445 0.7× 462 0.9× 250 0.7× 182 0.5× 129 3.6k
Darrell H. Carney United States 36 1.4k 2.1× 226 0.4× 457 0.9× 655 1.8× 119 0.4× 77 3.7k
Ruggero Tenni Italy 23 388 0.6× 320 0.5× 218 0.4× 229 0.6× 258 0.8× 59 1.3k
Carmelo Mavilia Italy 27 572 0.9× 284 0.5× 515 1.1× 132 0.4× 135 0.4× 49 2.3k
Shingo Tajima Japan 30 907 1.4× 681 1.1× 185 0.4× 362 1.0× 321 0.9× 187 3.6k
Muhammad M. Bashir United States 27 648 1.0× 565 0.9× 162 0.3× 231 0.6× 279 0.8× 61 1.9k
Ryu‐Ichiro Hata Japan 33 1.6k 2.4× 281 0.4× 532 1.1× 361 1.0× 303 0.9× 87 3.4k
Tsunao Kishida Japan 28 1.1k 1.6× 387 0.6× 385 0.8× 204 0.6× 260 0.8× 92 2.6k
Zeenat Gunja‐Smith United States 22 562 0.8× 254 0.4× 416 0.9× 527 1.5× 401 1.2× 31 2.1k

Countries citing papers authored by Giuseppe Cetta

Since Specialization
Citations

This map shows the geographic impact of Giuseppe Cetta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giuseppe Cetta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giuseppe Cetta more than expected).

Fields of papers citing papers by Giuseppe Cetta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Giuseppe Cetta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giuseppe Cetta. The network helps show where Giuseppe Cetta may publish in the future.

Co-authorship network of co-authors of Giuseppe Cetta

This figure shows the co-authorship network connecting the top 25 collaborators of Giuseppe Cetta. A scholar is included among the top collaborators of Giuseppe Cetta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giuseppe Cetta. Giuseppe Cetta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Viglio, Simona, Laura Annovazzi, Bice Conti, et al.. (2006). The role of emerging techniques in the investigation of prolidase deficiency: From diagnosis to the development of a possible therapeutical approach. Journal of Chromatography B. 832(1). 1–8. 27 indexed citations
2.
Iadarola, Paolo, Giuseppe Cetta, Maurizio Luisetti, et al.. (2005). Micellar electrokinetic chromatographic and capillary zone electrophoretic methods for screening urinary biomarkers of human disorders: A critical review of the state‐of‐the‐art. Electrophoresis. 26(4-5). 752–766. 21 indexed citations
3.
Lupi, Anna, et al.. (2005). N-benzyloxycarbonyl-l-proline: An in vitro and in vivo inhibitor of prolidase. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1744(2). 157–163. 19 indexed citations
4.
5.
Annovazzi, Laura, Simona Viglio, Dealba Gheduzzi, et al.. (2004). High levels of desmosines in urine and plasma of patients with pseudoxanthoma elasticum. European Journal of Clinical Investigation. 34(2). 156–164. 34 indexed citations
6.
Perugini, Paola, Ida Genta, Tiziana Modena, et al.. (2004). Intracellular delivery of liposome-encapsulated prolidase in cultured fibroblasts from prolidase-deficient patients. Journal of Controlled Release. 102(1). 181–190. 19 indexed citations
7.
Rossi, Antonio, et al.. (2003). IN VITRO PROTEOGLYCAN SULFATION DERIVED FROM SULFHYDRYL COMPOUNDS IN SULFATE TRANSPORTER CHONDRODYSPLASIAS. Pediatric Pathology & Molecular Medicine. 22(4). 311–321. 10 indexed citations
11.
Grimm, Rudi, Giuseppe Zanaboni, Simona Viglio, et al.. (1997). Complete resolution of imidodipeptide mixtures in urine of prolidase-deficient patients using micellar electrokinetic chromatography. Journal of Chromatography A. 768(1). 57–66. 12 indexed citations
12.
Zolezzi, Francesca, Maurizia Valli, Massimo Clementi, et al.. (1997). Mutation producing alternative splicing of exon 26 in the COL1A2 gene causes type IV osteogenesis imperfecta with intrafamilial clinical variability. American Journal of Medical Genetics. 71(3). 366–370. 14 indexed citations
13.
Rossi, Antonio, Jacky Bonaventure, Anne‐Lise Delezoide, Giuseppe Cetta, & Andrea Superti‐Furga. (1996). Undersulfation of Proteoglycans Synthesized by Chondrocytes from a Patient with Achondrogenesis Type 1B Homozygous for an L483P Substitution in the Diastrophic Dysplasia Sulfate Transporter. Journal of Biological Chemistry. 271(31). 18456–18464. 65 indexed citations
14.
Zanaboni, Giuseppe, Rudi Grimm, Katharine M. Dyne, et al.. (1996). Use of capillary zone electrophoresis for analysis of imidodipeptides in urine of prolidase-deficient patients. Journal of Chromatography B Biomedical Sciences and Applications. 683(1). 97–107. 18 indexed citations
15.
Zolezzi, Francesca, Antonella Forlino, Monica Mottes, et al.. (1995). A 931 + 2T → C transition in one COL1A2 allele causes exon 16 skipping in PROα2(I) mRNA and produces moderately severe OI. Human Mutation. 6(3). 268–271. 4 indexed citations
16.
Mottes, Monica, Antonella Sangalli, Maurizia Valli, et al.. (1994). A base substitution at IVS-19 3?-end splice junction causes exon 20 skipping in pro?2(I) collagen mRNA and produces mild osteogenesis imperfecta. Human Genetics. 93(6). 681–7. 13 indexed citations
17.
Berardesca, Enzo, et al.. (1992). Blood transfusions in the therapy of a case of prolidase deficiency. British Journal of Dermatology. 126(2). 193–195. 22 indexed citations
18.
Pozzi, Ernesto, et al.. (1989). Relationship between Changes in Alveolar Surfactant Levels and Lung Defence Mechanisms. Respiration. 55(1). 53–59. 12 indexed citations
19.
Cetta, Giuseppe, Francesco Ramirez, & Petros Tsipouras. (1988). Third International Conference on Osteogenesis Imperfecta. New York Academy of Sciences eBooks. 4 indexed citations
20.
Cetta, Giuseppe, Ruggero Tenni, Giuseppe Zanaboni, et al.. (1982). Biochemical and morphological modifications in rabbit Achilles tendon during maturation and ageing. Biochemical Journal. 204(1). 61–67. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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