Mauricio Barrero

2.3k total citations
8 papers, 1.0k citations indexed

About

Mauricio Barrero is a scholar working on Molecular Biology, Orthopedics and Sports Medicine and Oncology. According to data from OpenAlex, Mauricio Barrero has authored 8 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Orthopedics and Sports Medicine and 5 papers in Oncology. Recurrent topics in Mauricio Barrero's work include Bone Metabolism and Diseases (7 papers), Bone health and osteoporosis research (6 papers) and Bone health and treatments (5 papers). Mauricio Barrero is often cited by papers focused on Bone Metabolism and Diseases (7 papers), Bone health and osteoporosis research (6 papers) and Bone health and treatments (5 papers). Mauricio Barrero collaborates with scholars based in United States and Hong Kong. Mauricio Barrero's co-authors include Michael S. Ominsky, Paul J. Kostenuik, W. Scott Simonet, Frank Asuncion, Hua Zhu Ke, Marina Stolina, Denise Dwyer, Xiaodong Li, Chris Pászty and Kelly Warmington and has published in prestigious journals such as Endocrinology, Journal of Bone and Mineral Research and Bone.

In The Last Decade

Mauricio Barrero

8 papers receiving 977 citations

Peers

Mauricio Barrero

OSM

MB

ONCOL

SURGE

EPIDE

Elizabeth C. Wahl United States

Satoshi Komatsubara Japan

Qing Zeng United States

Saburo Nishida Japan

Val Mann United Kingdom

Alyson Morse Australia

A. John Yates United States

Naoki Haraguchi Japan

Atsushi Arai Japan

John C. Tiffee United States

Elizabeth C. Wahl United States

Mauricio Barrero

278 ×0.5

OSM

407 ×0.7

MB

194 ×0.4

ONCOL

107 ×0.5

SURGE

82 ×0.6

EPIDE

Citations per year, relative to Mauricio Barrero Mauricio Barrero (= 1×) peers Elizabeth C. Wahl

Countries citing papers authored by Mauricio Barrero

Since Specialization

Citations

This map shows the geographic impact of Mauricio Barrero's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mauricio Barrero with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mauricio Barrero more than expected).

Fields of papers citing papers by Mauricio Barrero

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mauricio Barrero. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mauricio Barrero. The network helps show where Mauricio Barrero may publish in the future.

Co-authorship network of co-authors of Mauricio Barrero

This figure shows the co-authorship network connecting the top 25 collaborators of Mauricio Barrero. A scholar is included among the top collaborators of Mauricio Barrero based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mauricio Barrero. Mauricio Barrero is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Li, Xiaodong, Michael S. Ominsky, Kelly Warmington, et al.. (2011). Increased Bone Formation and Bone Mass Induced by Sclerostin Antibody Is Not Affected by Pretreatment or Cotreatment with Alendronate in Osteopenic, Ovariectomized Rats. Endocrinology. 152(9). 3312–3322. 71 indexed citations

2.

Ominsky, Michael S., Hong‐Lin Tan, Mauricio Barrero, et al.. (2011). Increased callus mass and enhanced strength during fracture healing in mice lacking the sclerostin gene. Bone. 49(6). 1178–1185. 61 indexed citations

3.

Li, Xiaodong, Kelly Warmington, Qing‐Tian Niu, et al.. (2010). Inhibition of sclerostin by monoclonal antibody increases bone formation, bone mass, and bone strength in aged male rats. Journal of Bone and Mineral Research. 25(12). 2647–2656. 191 indexed citations

4.

Ominsky, Michael S., Chaoyang Li, Xiaodong Li, et al.. (2010). Inhibition of sclerostin by monoclonal antibody enhances bone healing and improves bone density and strength of nonfractured bones. Journal of Bone and Mineral Research. 26(5). 1012–1021. 209 indexed citations

5.

Ominsky, Michael S., Marina Stolina, Xiaodong Li, et al.. (2009). One Year of Transgenic Overexpression of Osteoprotegerin in Rats Suppressed Bone Resorption and Increased Vertebral Bone Volume, Density, and Strength. Journal of Bone and Mineral Research. 24(7). 1234–1246. 36 indexed citations

6.

Gerstenfeld, Louis C., Zachary Mason, Dana T. Graves, et al.. (2008). Comparison of Effects of the Bisphosphonate Alendronate Versus the RANKL Inhibitor Denosumab on Murine Fracture Healing. Journal of Bone and Mineral Research. 24(2). 196–208. 181 indexed citations

7.

Ominsky, Michael S., Xiaodong Li, Frank Asuncion, et al.. (2008). RANKL Inhibition with Osteoprotegerin Increases Bone Strength by Improving Cortical and Trabecular bone Architecture in Ovariectomized Rats. Journal of Bone and Mineral Research. 23(5). 672–682. 104 indexed citations

8.

Alberta, Frank G., Michael S. Aronow, Mauricio Barrero, et al.. (2005). Ligamentous Lisfranc Joint Injuries: A Biomechanical Comparison of Dorsal Plate and Transarticular Screw Fixation. Foot & Ankle International. 26(6). 462–473. 153 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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