Mattia Schmid

434 total citations
10 papers, 350 citations indexed

About

Mattia Schmid is a scholar working on Epidemiology, Immunology and Molecular Biology. According to data from OpenAlex, Mattia Schmid has authored 10 papers receiving a total of 350 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Epidemiology, 4 papers in Immunology and 3 papers in Molecular Biology. Recurrent topics in Mattia Schmid's work include Adipokines, Inflammation, and Metabolic Diseases (4 papers), Immune Response and Inflammation (2 papers) and S100 Proteins and Annexins (2 papers). Mattia Schmid is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (4 papers), Immune Response and Inflammation (2 papers) and S100 Proteins and Annexins (2 papers). Mattia Schmid collaborates with scholars based in Switzerland, Germany and Italy. Mattia Schmid's co-authors include Martin Hersberger, Claudio Gemperle, Nicole Rimann, R. H. Largo, Jacqueline Marti-Jaun, Viola Günther, Paulina Wawrzyniak, Peter Wiesli, Gustav K. von Schulthess and Jan A. Ehses and has published in prestigious journals such as The Journal of Immunology, PLoS ONE and Atherosclerosis.

In The Last Decade

Mattia Schmid

9 papers receiving 346 citations

Peers

Mattia Schmid

IMMUN

ND

EPIDE

MB

BIOCH

Tomasz Śliwa Poland

Mizuki Endo Japan

Kelly Hester United States

Hee Young Kim South Korea

Monan Angela Zhang Canada

Mingtong Xu China

Jiro Toyonaga Japan

Alexis Cutchins United States

Kazuhiro Osanai Japan

Tomasz Śliwa Poland

Mattia Schmid

134 ×1.0

IMMUN

54 ×0.5

ND

42 ×0.5

EPIDE

77 ×0.9

MB

9 ×0.1

BIOCH

Citations per year, relative to Mattia Schmid Mattia Schmid (= 1×) peers Tomasz Śliwa

Countries citing papers authored by Mattia Schmid

Since Specialization

Citations

This map shows the geographic impact of Mattia Schmid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mattia Schmid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mattia Schmid more than expected).

Fields of papers citing papers by Mattia Schmid

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mattia Schmid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mattia Schmid. The network helps show where Mattia Schmid may publish in the future.

Co-authorship network of co-authors of Mattia Schmid

This figure shows the co-authorship network connecting the top 25 collaborators of Mattia Schmid. A scholar is included among the top collaborators of Mattia Schmid based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mattia Schmid. Mattia Schmid is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Jungo, Alain, Mattia Schmid, Raphael Sznitman, et al.. (2025). GEYEDANCE: An OCT-Enhanced Multi-Modal Feedback Platform for Robot-Assisted Ophthalmic Surgery. IEEE Transactions on Medical Robotics and Bionics. 7(3). 1017–1028. 2 indexed citations

2.

Gemperle, Claudio, Mattia Schmid, Nicole Rimann, et al.. (2021). Resolvin D1 reduces inflammation in co-cultures of primary human macrophages and adipocytes by triggering macrophages. Prostaglandins Leukotrienes and Essential Fatty Acids. 174. 102363–102363. 13 indexed citations

3.

Schmid, Mattia, Claudio Gemperle, Nicole Rimann, & Martin Hersberger. (2016). Resolvin D1 Polarizes Primary Human Macrophages toward a Proresolution Phenotype through GPR32. The Journal of Immunology. 196(8). 3429–3437. 84 indexed citations

4.

Schmid, Mattia, et al.. (2015). ChemR23, the Receptor for Chemerin and Resolvin E1, Is Expressed and Functional on M1 but Not on M2 Macrophages. The Journal of Immunology. 194(5). 2330–2337. 132 indexed citations

5.

Schmid, Mattia, et al.. (2014). Low dose aspirin is associated with plasma chemerin levels and may reduce adipose tissue inflammation. Atherosclerosis. 235(2). 256–262. 27 indexed citations

6.

Gemperle, Claudio, et al.. (2012). Regulation of the Formyl Peptide Receptor 1 (FPR1) Gene in Primary Human Macrophages. PLoS ONE. 7(11). e50195–e50195. 37 indexed citations

7.

Schmid, Mattia, et al.. (2012). Characterization of the Promoter and the Transcriptional Regulation of the Lipoxin A4 Receptor (FPR2/ALX) Gene in Human Monocytes and Macrophages. The Journal of Immunology. 188(4). 1856–1867. 30 indexed citations

8.

Wiesli, Peter, Mattia Schmid, Christoph Schmid, et al.. (2007). TNF-alpha -308G>A Polymorphism Modulates Cytokine Serum Concentrations and Macrovascular Complications in Diabetic Patients on Aspirin. Experimental and Clinical Endocrinology & Diabetes. 115(5). 322–326. 5 indexed citations

9.

Südkamp, Michael, Mattia Schmid, H. Geißler, et al.. (2005). VDD-Pacemaker in Children - A Long-Term Therapy?. The Thoracic and Cardiovascular Surgeon. 53(3). 158–161.

10.

Schmid, Mattia & R. H. Largo. (1986). Visual acuity and stereopsis between the ages of 5 and 10 years. European Journal of Pediatrics. 145(6). 475–479. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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