Jan A. Ehses

9.9k total citations · 2 hit papers
51 papers, 7.1k citations indexed

About

Jan A. Ehses is a scholar working on Surgery, Genetics and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Jan A. Ehses has authored 51 papers receiving a total of 7.1k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Surgery, 31 papers in Genetics and 26 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Jan A. Ehses's work include Pancreatic function and diabetes (39 papers), Diabetes and associated disorders (30 papers) and Diabetes Treatment and Management (13 papers). Jan A. Ehses is often cited by papers focused on Pancreatic function and diabetes (39 papers), Diabetes and associated disorders (30 papers) and Diabetes Treatment and Management (13 papers). Jan A. Ehses collaborates with scholars based in Switzerland, Canada and France. Jan A. Ehses's co-authors include Marc Y. Donath, Mirjam Faulenbach, Helga Ellingsgaard, Claus M. Larsen, Thomas Mandrup‐Poulsen, Allan Vaag, Marianne Böni‐Schnetzler, Burkhardt Seifert, Aage Vølund and Clara Westwell‐Roper and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Jan A. Ehses

51 papers receiving 6.9k citations

Hit Papers

Interleukin-1–Receptor Antagonist in Type 2 Diabetes Mell... 2007 2026 2013 2019 2007 2007 400 800 1.2k

Peers

Jan A. Ehses
Jan A. Ehses
Citations per year, relative to Jan A. Ehses Jan A. Ehses (= 1×) peers Kathrin Maedler

Countries citing papers authored by Jan A. Ehses

Since Specialization
Citations

This map shows the geographic impact of Jan A. Ehses's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jan A. Ehses with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jan A. Ehses more than expected).

Fields of papers citing papers by Jan A. Ehses

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jan A. Ehses. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jan A. Ehses. The network helps show where Jan A. Ehses may publish in the future.

Co-authorship network of co-authors of Jan A. Ehses

This figure shows the co-authorship network connecting the top 25 collaborators of Jan A. Ehses. A scholar is included among the top collaborators of Jan A. Ehses based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jan A. Ehses. Jan A. Ehses is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lehrskov, Louise Lang, Mark Lyngbæk, Grit Elster Legaard, et al.. (2018). Interleukin-6 Delays Gastric Emptying in Humans with Direct Effects on Glycemic Control. Cell Metabolism. 27(6). 1201–1211.e3. 88 indexed citations
2.
Oprescu, Andrei I., Khajag Koulajian, Yusaku Mori, et al.. (2017). IKKβ inhibition prevents fat-induced beta cell dysfunction in vitro and in vivo in rodents. Diabetologia. 60(10). 2021–2032. 14 indexed citations
3.
Westwell‐Roper, Clara, Heather C. Denroche, Jan A. Ehses, & C. Bruce Verchere. (2016). Differential Activation of Innate Immune Pathways by Distinct Islet Amyloid Polypeptide (IAPP) Aggregates. Journal of Biological Chemistry. 291(17). 8908–8917. 39 indexed citations
4.
Weisser, Shelley B., Young Lo, Lisa K. Kozicky, et al.. (2015). Activity of SHIP, Which Prevents Expression of Interleukin 1β, Is Reduced in Patients With Crohn’s Disease. Gastroenterology. 150(2). 465–476. 27 indexed citations
5.
Westwell‐Roper, Clara, et al.. (2014). IL-1 mediates amyloid-associated islet dysfunction and inflammation in human islet amyloid polypeptide transgenic mice. Diabetologia. 58(3). 575–585. 62 indexed citations
6.
Nąckiewicz, Dominika, Meixia Dan, Wei He, et al.. (2014). TLR2/6 and TLR4-activated macrophages contribute to islet inflammation and impair beta cell insulin gene expression via IL-1 and IL-6. Diabetologia. 57(8). 1645–1654. 93 indexed citations
7.
Giroix, M H, J.C. Irminger, Grégory Lacraz, et al.. (2011). Hypercholesterolaemia, signs of islet microangiopathy and altered angiogenesis precede onset of type 2 diabetes in the Goto–Kakizaki (GK) rat. Diabetologia. 54(9). 2451–2462. 22 indexed citations
8.
Noll, Christophe, Grégory Lacraz, Jan A. Ehses, et al.. (2011). Early reduction of circulating homocysteine levels in Goto–Kakizaki rat, a spontaneous nonobese model of type 2 diabetes. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1812(6). 699–702. 11 indexed citations
9.
Donath, Marc Y., Marianne Böni‐Schnetzler, Helga Ellingsgaard, Philippe A. Halban, & Jan A. Ehses. (2010). Cytokine production by islets in health and diabetes: cellular origin, regulation and function. Trends in Endocrinology and Metabolism. 21(5). 261–267. 170 indexed citations
10.
Beauquis, Juan, Françoise Homo‐Delarche, Marie-Hélène Giroix, et al.. (2010). Hippocampal neurovascular and hypothalamic–pituitary–adrenal axis alterations in spontaneously type 2 diabetic GK rats. Experimental Neurology. 222(1). 125–134. 57 indexed citations
11.
Ehses, Jan A., Grégory Lacraz, M.H. Giroix, et al.. (2009). IL-1 antagonism reduces hyperglycemia and tissue inflammation in the type 2 diabetic GK rat. Proceedings of the National Academy of Sciences. 106(33). 13998–14003. 291 indexed citations
12.
Lacraz, Grégory, Marie-Hélène Giroix, Nadim Kassis, et al.. (2009). Islet Endothelial Activation and Oxidative Stress Gene Expression Is Reduced by IL-1Ra Treatment in the Type 2 Diabetic GK Rat. PLoS ONE. 4(9). e6963–e6963. 58 indexed citations
13.
Larsen, Claus M., Mirjam Faulenbach, Allan Vaag, et al.. (2009). Sustained Effects of Interleukin-1 Receptor Antagonist Treatment in Type 2 Diabetes. Diabetes Care. 32(9). 1663–1668. 308 indexed citations
14.
Böni‐Schnetzler, Marianne, Jan A. Ehses, Mirjam Faulenbach, & Marc Y. Donath. (2008). Insulitis in type 2 diabetes. Diabetes Obesity and Metabolism. 10(s4). 201–204. 50 indexed citations
15.
Larsen, Claus M., Mirjam Faulenbach, Allan Vaag, et al.. (2007). Interleukin-1–Receptor Antagonist in Type 2 Diabetes Mellitus. New England Journal of Medicine. 356(15). 1517–1526. 1390 indexed citations breakdown →
16.
Wiesli, Peter, Mattia Schmid, Christoph Schmid, et al.. (2007). TNF-alpha -308G>A Polymorphism Modulates Cytokine Serum Concentrations and Macrovascular Complications in Diabetic Patients on Aspirin. Experimental and Clinical Endocrinology & Diabetes. 115(5). 322–326. 5 indexed citations
17.
Hinke, Simon A., Francis C. Lynn, Jan A. Ehses, et al.. (2005). Glucose-dependent Insulinotropic Polypeptide (GIP): Development of DP IV-Resistant Analogues with Therapeutic Potential. Kluwer Academic Publishers eBooks. 524. 293–301. 9 indexed citations
18.
Maedler, Kathrin, П. В. Сергеев, Jan A. Ehses, et al.. (2004). Leptin modulates β cell expression of IL-1 receptor antagonist and release of IL-1β in human islets. Proceedings of the National Academy of Sciences. 101(21). 8138–8143. 214 indexed citations
19.
Pospisilik, J. Andrew, et al.. (2004). Dipeptidyl Peptidase IV Inhibition in Animal Models of Diabetes. Advances in experimental medicine and biology. 524. 281–291. 17 indexed citations
20.
Wheeler, Michael B., Richard W. Gelling, Simon A. Hinke, et al.. (1999). Characterization of the Carboxyl-terminal Domain of the Rat Glucose-dependent Insulinotropic Polypeptide (GIP) Receptor. Journal of Biological Chemistry. 274(35). 24593–24601. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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