Matthew Spite

8.6k citations

65 papers · 6.5k indexed · 3 hit papers · h-index 42

Impact in

Biochemistry top 0.1%
- Eicosanoids and Hypertension Pharmacology
Nutrition and Dietetics top 0.1%
- Fatty Acid Research and Health

Papers in ⓘ

Nutrition and Dietetics 30
- Fatty Acid Research and Health 30
Biochemistry 19
- Eicosanoids and Hypertension Pharmacology 16

Co-authors: Charles N. Serhan (12 shared papers)Brian E. Sansbury (29 shared papers)Jason Hellmann (18 shared papers)Aruni Bhatnagar (14 shared papers)Rong Yang (3 shared papers)Gabrielle Fredman (12 shared papers)Yunan Tang (9 shared papers)Michael J. Zhang (6 shared papers)
Journals: Circulation Research (6 papers)The Journal of Immunology (5 papers)The FASEB Journal (5 papers)Proceedings of the National Academy of Sciences (3 papers)Arteriosclerosis Thrombosis and Vascular Biology (3 papers)
Partner nations: United States Australia Hungary

In The Last Decade

Matthew Spite

64 papers receiving 6.3k citations

Hit Papers

align trajectories log scale

An imbalance between specialized pro-resolving lipid mediators and pro-inflammatory leukotrienes promotes instability of atherosclerotic plaques 2016 · 326 citations

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if any of the following hold:

it has ≥500 total citations;
it reaches ≥1.5× the top-1% citation threshold for papers in the same subfield and year (the threshold is the minimum needed to enter the top 1%, not the average within it);
it reaches the top citation threshold in at least one of its specific research topics.

2016 Nature Communications
2009 Nature
2008 The Journal of Experimental Medicine

Peers

Countries citing papers authored by Matthew Spite

Since Specialization

Citations

This map shows the geographic impact of Matthew Spite's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew Spite with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew Spite more than expected).

Fields of papers citing papers by Matthew Spite

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew Spite. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew Spite. The network helps show where Matthew Spite may publish in the future.

Co-authors

The 25 scholars most cited alongside Matthew Spite, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Matthew Spite Line = papers co-authored together Matthew Spite links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 65 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Maresins: novel macrophage mediators with potent antiinflammatory and proresolving actions The Journal of Experimental Medicine ·Charles N. Serhan, Rong Yang, Kimberly Martinod, Kie Kasuga, Padmini S. Pillai, Timothy F. Porter, Sungwhan F. Oh, Matthew Spite Hit paper breakdown →	2008	694
2	Resolvin D2 is a potent regulator of leukocytes and controls microbial sepsis Nature ·Matthew Spite, Lucy V. Norling, Lisa Summers, Rong Yang, Dianne Cooper, Nicos A. Petasis, Roderick J. Flower, Mauro Perretti, Charles N. Serhan Hit paper breakdown →	2009	563
3	Resolvins, Specialized Proresolving Lipid Mediators, and Their Potential Roles in Metabolic Diseases Cell Metabolism ·Matthew Spite, Joan Clària, Charles N. Serhan	2013	354
4	An imbalance between specialized pro-resolving lipid mediators and pro-inflammatory leukotrienes promotes instability of atherosclerotic plaques Nature Communications ·Gabrielle Fredman, Jason Hellmann, Jonathan D. Proto, George Kuriakose, Romain A. Colas, Bernhard Dorweiler, E. Sander Connolly, Robert A. Solomon, David Jones, Éric Heyer, Matthew Spite, Ira Tabas Hit paper breakdown →	2016	326
5	Novel Lipid Mediators Promote Resolution of Acute Inflammation Circulation Research ·Matthew Spite, Charles N. Serhan	2010	290
6	Resolvin D1 decreases adipose tissue macrophage accumulation and improves insulin sensitivity in obese‐diabetic mice The FASEB Journal ·Jason Hellmann, Yunan Tang, Madhavi Kosuri, Aruni Bhatnagar, Matthew Spite	2011	245
7	MerTK cleavage limits proresolving mediator biosynthesis and exacerbates tissue inflammation Proceedings of the National Academy of Sciences ·Bishuang Cai, Edward B. Thorp, Amanda C. Doran, Manikandan Subramanian, Brian E. Sansbury, Chyuan-Sheng Lin, Matthew Spite, Gabrielle Fredman, Ira Tabas	2016	185
8	MerTK receptor cleavage promotes plaque necrosis and defective resolution in atherosclerosis Journal of Clinical Investigation ·Bishuang Cai, Edward B. Thorp, Amanda C. Doran, Brian E. Sansbury, Mat J.A.P. Daemen, Bernhard Dorweiler, Matthew Spite, Gabrielle Fredman, Ira Tabas	2017	182
9	Regulatory T Cells License Macrophage Pro-Resolving Functions During Atherosclerosis Regression Circulation Research ·Monika Sharma, Martin Schlegel, Milessa Silva Afonso, Emily J. Brown, Karishma Rahman, Ada Weinstock, Brian E. Sansbury, Emma M. Corr, Coen van Solingen, Graeme J. Koelwyn, Lianne C. Shanley, Lauren Beckett, Daniel Peled, Juan J. Lafaille, Matthew Spite, P’ng Loke, Edward A. Fisher, Kathryn J. Moore	2020	182
10	Aspirin-Triggered Lipoxin and Resolvin E1 Modulate Vascular Smooth Muscle Phenotype and Correlate with Peripheral Atherosclerosis American Journal Of Pathology ·Karen J. Ho, Matthew Spite, Christopher Owens, Hope Lancero, Alex Kroemer, Reena L. Pande, Mark A. Creager, Charles N. Serhan, Michael S. Conte	2010	173
11	Resolution of Acute Inflammation and the Role of Resolvins in Immunity, Thrombosis, and Vascular Biology Circulation Research ·Brian E. Sansbury, Matthew Spite	2016	173
12	Cutting Edge: Humanized Nano-Proresolving Medicines Mimic Inflammation-Resolution and Enhance Wound Healing The Journal of Immunology ·Lucy V. Norling, Matthew Spite, Rong Yang, Roderick J. Flower, Mauro Perretti, Charles N. Serhan	2011	168
13	Proresolution Therapy for the Treatment of Delayed Healing of Diabetic Wounds Diabetes ·Yunan Tang, Michael J. Zhang, Jason Hellmann, Madhavi Kosuri, Aruni Bhatnagar, Matthew Spite	2012	162
14	Resolvin D1 limits 5-lipoxygenase nuclear localization and leukotriene B ₄ synthesis by inhibiting a calcium-activated kinase pathway Proceedings of the National Academy of Sciences ·Gabrielle Fredman, Lale Özcan, Stefano Spolitu, Jason Hellmann, Matthew Spite, Johannes Backs, Ira Tabas	2014	160
15	Macrophage PPARγ, a Lipid Activated Transcription Factor Controls the Growth Factor GDF3 and Skeletal Muscle Regeneration Immunity ·Tamás Varga, Rémi Mounier, Andreas Patsalos, Péter Gogolák, Matthew Peloquin, Attila Horváth, Attila Pap, Bence Dániel, Gergely Nagy, Éva Pintye, Szilárd Póliska, Sylvain Cuvellier, Sabrina Ben Larbi, Brian E. Sansbury, Matthew Spite, Chester Brown, Bénédicte Chazaud, László Nagy	2016	138
16	Deficiency of the Leukotriene B4 Receptor, BLT-1, Protects against Systemic Insulin Resistance in Diet-Induced Obesity The Journal of Immunology ·Matthew Spite, Jason Hellmann, Yunan Tang, Steven Mathis, Madhavi Kosuri, Aruni Bhatnagar, Venkatakrishna R. Jala, Bodduluri Haribabu	2011	138
17	Resolvins: Anti-Inflammatory and Proresolving Mediators Derived from Omega-3 Polyunsaturated Fatty Acids Annual Review of Nutrition ·Michael J. Zhang, Matthew Spite	2012	136
18	Metabolic remodeling of white adipose tissue in obesity American Journal of Physiology-Endocrinology and Metabolism ·Timothy D. Cummins, Candice R. Holden, Brian E. Sansbury, Andrew Gibb, Jasmit Shah, Nagma Zafar, Yunan Tang, Jason Hellmann, N. Shesh, Matthew Spite, Aruni Bhatnagar, Bradford G. Hill	2014	134
19	Overexpression of Endothelial Nitric Oxide Synthase Prevents Diet-Induced Obesity and Regulates Adipocyte Phenotype Circulation Research ·Brian E. Sansbury, Timothy D. Cummins, Yunan Tang, Jason Hellmann, Candice R. Holden, Matthew A. Harbeson, Yang Chen, Rakesh P. Patel, Matthew Spite, Aruni Bhatnagar, Bradford G. Hill	2012	122
20	Dynamic changes to lipid mediators support transitions among macrophage subtypes during muscle regeneration Nature Immunology ·Nikolas Giannakis, Brian E. Sansbury, Andreas Patsalos, Tristan T. Hays, Colin O. Riley, Xianlin Han, Matthew Spite, László Nagy	2019	113

About Matthew Spite

Matthew Spite is a scholar working on Nutrition and Dietetics, Biochemistry, Immunology, Epidemiology and Surgery, having authored 65 papers that have together received 6.5k indexed citations. Recurring topics across this work include Fatty Acid Research and Health (30 papers), Eicosanoids and Hypertension Pharmacology (16 papers), Adipokines, Inflammation, and Metabolic Diseases (14 papers), Adipose Tissue and Metabolism (10 papers), Cholesterol and Lipid Metabolism (8 papers), Immune cells in cancer (7 papers), Immune Response and Inflammation (6 papers) and Phagocytosis and Immune Regulation (6 papers). The work is most often cited by research in Biochemistry (1.5k citations), Nutrition and Dietetics (2.5k citations), Immunology (2.0k citations), Physiology (1.2k citations) and Clinical Biochemistry (280 citations). Matthew Spite has collaborated with scholars based in United States, Australia and Hungary. Frequent co-authors include Charles N. Serhan, Brian E. Sansbury, Jason Hellmann, Aruni Bhatnagar, Rong Yang, Gabrielle Fredman, Yunan Tang, Michael J. Zhang, Joan Clària and Lucy V. Norling. Their work appears in journals such as Circulation Research, The Journal of Immunology, The FASEB Journal, Proceedings of the National Academy of Sciences and Arteriosclerosis Thrombosis and Vascular Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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