Matthew K. Lancaster

1.4k total citations
29 papers, 961 citations indexed

About

Matthew K. Lancaster is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Matthew K. Lancaster has authored 29 papers receiving a total of 961 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Cardiology and Cardiovascular Medicine, 19 papers in Molecular Biology and 10 papers in Cellular and Molecular Neuroscience. Recurrent topics in Matthew K. Lancaster's work include Cardiac electrophysiology and arrhythmias (20 papers), Ion channel regulation and function (16 papers) and Neuroscience and Neural Engineering (7 papers). Matthew K. Lancaster is often cited by papers focused on Cardiac electrophysiology and arrhythmias (20 papers), Ion channel regulation and function (16 papers) and Neuroscience and Neural Engineering (7 papers). Matthew K. Lancaster collaborates with scholars based in United Kingdom, Japan and United States. Matthew K. Lancaster's co-authors include Sandra A. Jones, Mark R. Boyett, Halina Dobrzynski, Stephen J. Cook, Ming Lei, Igor R. Efimov, S. Fung, Yoshiko Takagishi, Patrizia Camelliti and Sebastian K.G. Maier and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Circulation Research.

In The Last Decade

Matthew K. Lancaster

28 papers receiving 944 citations

Peers

Countries citing papers authored by Matthew K. Lancaster

Since Specialization

Citations

This map shows the geographic impact of Matthew K. Lancaster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew K. Lancaster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew K. Lancaster more than expected).

Fields of papers citing papers by Matthew K. Lancaster

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew K. Lancaster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew K. Lancaster. The network helps show where Matthew K. Lancaster may publish in the future.

Co-authorship network of co-authors of Matthew K. Lancaster

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew K. Lancaster. A scholar is included among the top collaborators of Matthew K. Lancaster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew K. Lancaster. Matthew K. Lancaster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Ionic current changes underlying action potential repolarization responses to physiological pacing and adrenergic stimulation in adult rat ventricular myocytes 2023 ·Physiological Reports ·(unknown), (unknown), Michael A. Colman, Matthew K. Lancaster, Alan P. Benson	2
2	Reduced cardiac response to the adrenergic system is a key limiting factor for physical capacity in old age 2021 ·Experimental Gerontology ·(unknown), Matthew K. Lancaster	9
3	A Generalizable Framework for Multi-Scale Auditing of Digital Learning Provision in Higher Education 2018 ·Online Learning ·Samuel R. P.‐J. Ross, (unknown), Matthew K. Lancaster, (unknown)	5
4	K_2p3.1 protein is expressed as a transmural gradient across the rat left ventricular free wall 2018 ·Journal of Cardiovascular Electrophysiology ·Sandra A. Jones, Richard D. Walton, Michael J. Morton, Matthew K. Lancaster	1
5	Interactions of Short-Term and Chronic Treadmill Training With Aging of the Left Ventricle of the Heart 2015 ·The Journals of Gerontology Series A ·Richard D. Walton, Sandra A. Jones, (unknown), Anna C. Kayani, Graeme L. Close, Anne McArdle, Matthew K. Lancaster	13
6	Abstract 13130: Age-associated Reductions in Junctophilin-2 and Amphiphysin-2 Associate With Disruption of T-tubules and Intracellular Calcium Regulation in Myocytes From the Mouse Left Ventricle 2015 ·Circulation ·(unknown), Matthew K. Lancaster	1
7	Progressive age-associated activation of JNK associates with conduction disruption in the aged atrium 2015 ·Mechanisms of Ageing and Development ·Sandra A. Jones, Matthew K. Lancaster	14
8	Aging is a primary risk factor for cardiac arrhythmias: disruption of intracellular Ca²⁺regulation as a key suspect 2011 ·Expert Review of Cardiovascular Therapy ·(unknown), Matthew K. Lancaster, Sandra A. Jones	26
9	Ageing‐related changes of connexins and conduction within the sinoatrial node 2004 ·The Journal of Physiology ·Sandra A. Jones, Matthew K. Lancaster, Mark R. Boyett	106
10	Requirement of neuronal‐ and cardiac‐type sodium channels for murine sinoatrial node pacemaking 2004 ·The Journal of Physiology ·Ming Lei, Sandra A. Jones, Jie Liu, Matthew K. Lancaster, S. Fung, Halina Dobrzynski, Patrizia Camelliti, Sebastian K.G. Maier, Denis Noble, Mark R. Boyett	155
11	Intracellular Ca²⁺ and pacemaking within the rabbit sinoatrial node: heterogeneity of role and control 2004 ·The Journal of Physiology ·Matthew K. Lancaster, Sandra A. Jones, Simon M. Harrison, Mark R. Boyett	42
12	Sophisticated Architecture is Required for the Sinoatrial Node to Perform Its Normal Pacemaker Function 2003 ·Journal of Cardiovascular Electrophysiology ·Mark R. Boyett, Halina Dobrzynski, Matthew K. Lancaster, Sandra A. Jones, Haruo Honjo, Itsuo Kodama	44
13	The Gradient Model of the Rabbit Sinoatrial Node 2002 ·Korean Journal of Physiology and Pharmacology ·Halina Dobrzynski, (unknown), Sandra A. Jones, Matthew K. Lancaster, Mark R. Boyett	0
14	Na/K pump α subunit expression in rabbit ventricle and regional variations of intracellular sodium regulation 2000 ·Pflügers Archiv - European Journal of Physiology ·Matthew K. Lancaster, David Bennett, Susan Cook, Stephen O’Neill	8
15	Residues and Mechanisms for Slow Activation and Ba2+Block of the Cardiac Muscarinic K+ Channel, Kir3.1/Kir3.4 2000 ·Journal of Biological Chemistry ·Matthew K. Lancaster, Katharine M. Dibb, (unknown), Robert Leach, Jong‐Kook Lee, John B. C. Findlay, Mark R. Boyett	25
16	Cs+ block of the cardiac muscarinic K+ channel, GIRK1/GIRK4, is not dependent on the aspartate residue at position 173 2000 ·Pflügers Archiv - European Journal of Physiology ·Katharine M. Dibb, Robert Leach, Matthew K. Lancaster, J. B. C. Findlay, Mark R. Boyett	3
17	Changes in contraction, cytosolic Ca2+ and pH during metabolic inhibition and upon restoration of mitochondrial respiration in rat ventricular myocytes 1998 ·Experimental Physiology ·Matthew K. Lancaster, (unknown)	5
18	The effects of levosimendan on [Ca2+] in guinea-pig isolated ventricular myocytes 1997 ·European Journal of Pharmacology ·Matthew K. Lancaster, Stephen J. Cook	54
19	Variability of Spontaneous Ca ²⁺ Release Between Different Rat Ventricular Myocytes Is Correlated With Na ⁺ -Ca ²⁺ Exchange and [Na ⁺ ] _i 1996 ·Circulation Research ·M.E. Díaz, (unknown), Joseph Panashe Chamunorwa, Andrew W. Trafford, Matthew K. Lancaster, Stephen O’Neill, David Eisner	27
20	Laryngeal chondritis in Texel sheep 1987 ·Veterinary Record ·J. G. LANE, Pam Brown, Matthew K. Lancaster, (unknown)	16

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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