Matthew G.F. Sharp

1.3k total citations
22 papers, 899 citations indexed

About

Matthew G.F. Sharp is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Matthew G.F. Sharp has authored 22 papers receiving a total of 899 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Endocrinology, Diabetes and Metabolism and 6 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Matthew G.F. Sharp's work include Hormonal Regulation and Hypertension (8 papers), Receptor Mechanisms and Signaling (5 papers) and Renin-Angiotensin System Studies (5 papers). Matthew G.F. Sharp is often cited by papers focused on Hormonal Regulation and Hypertension (8 papers), Receptor Mechanisms and Signaling (5 papers) and Renin-Angiotensin System Studies (5 papers). Matthew G.F. Sharp collaborates with scholars based in United Kingdom, Germany and United States. Matthew G.F. Sharp's co-authors include John J. Mullins, Stewart Fleming, Jörg Peters, Joel K. Elmquist, Jeffrey S. Flier, Janice M. Paterson, Christopher J. Kenyon, Hiroshi Shinyama, Jonathan R. Seckl and Hiroaki Masuzaki and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Circulation Research.

In The Last Decade

Matthew G.F. Sharp

20 papers receiving 887 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthew G.F. Sharp United Kingdom 10 499 304 256 151 130 22 899
Elliott J. Meer United States 10 219 0.4× 607 2.0× 242 0.9× 118 0.8× 51 0.4× 11 953
Shin-Ichi Shimoda Japan 17 263 0.5× 305 1.0× 146 0.6× 302 2.0× 34 0.3× 73 898
Emel Songu‐Mize United States 17 213 0.4× 514 1.7× 186 0.7× 208 1.4× 83 0.6× 40 1.0k
Marilyn Dammerman United States 9 317 0.6× 296 1.0× 214 0.8× 126 0.8× 21 0.2× 10 919
D Křenová Czechia 20 292 0.6× 539 1.8× 181 0.7× 347 2.3× 30 0.2× 82 1.2k
I‐Hsien Wu United States 13 172 0.3× 300 1.0× 144 0.6× 154 1.0× 79 0.6× 23 731
Patrizia Borboni Italy 14 314 0.6× 456 1.5× 63 0.2× 158 1.0× 30 0.2× 21 881
Yoshiyuki Sakai Japan 12 326 0.7× 252 0.8× 130 0.5× 121 0.8× 43 0.3× 14 632
Zhong Jian Cheng Finland 12 143 0.3× 195 0.6× 254 1.0× 212 1.4× 57 0.4× 19 712
Aifang Nie China 15 202 0.4× 282 0.9× 60 0.2× 125 0.8× 39 0.3× 31 710

Countries citing papers authored by Matthew G.F. Sharp

Since Specialization
Citations

This map shows the geographic impact of Matthew G.F. Sharp's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew G.F. Sharp with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew G.F. Sharp more than expected).

Fields of papers citing papers by Matthew G.F. Sharp

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew G.F. Sharp. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew G.F. Sharp. The network helps show where Matthew G.F. Sharp may publish in the future.

Co-authorship network of co-authors of Matthew G.F. Sharp

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew G.F. Sharp. A scholar is included among the top collaborators of Matthew G.F. Sharp based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew G.F. Sharp. Matthew G.F. Sharp is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Meek, Stephen, Karamjit Singh Dolt, Linda Sutherland, et al.. (2024). Redundancy of p75NTR neurotrophin receptor function in development, growth and fertility in the rat. Transgenic Research. 33(4). 255–266.
2.
Sharp, Matthew G.F., et al.. (2024). Gene model for the ortholog of Glys in Drosophila simulans. PubMed. 2025.
3.
Villalobos, Elisa, Mark Nixon, Lee Murphy, et al.. (2021). Carbonyl reductase 1 amplifies glucocorticoid action in adipose tissue and impairs glucose tolerance in lean mice. Molecular Metabolism. 48. 101225–101225. 6 indexed citations
4.
Villalobos, Elisa, Mark Nixon, Matthew G.F. Sharp, et al.. (2021). Carbonyl Reductase 1 Overexpression in Adipose Amplifies Local Glucocorticoid Action and Impairs Glucose Tolerance in Lean Mice. Journal of the Endocrine Society. 5(Supplement_1). A806–A806. 1 indexed citations
5.
Buckley, Charlotte, Robert Nelson, L. J. Mullins, et al.. (2017). Phenotypic dissection of the mouse Ren1d knockout by complementation with human renin. Journal of Biological Chemistry. 293(4). 1151–1162. 2 indexed citations
6.
Meek, Stephen, Alison J. Thomson, Linda Sutherland, et al.. (2016). Reduced levels of dopamine and altered metabolism in brains of HPRT knock-out rats: a new rodent model of Lesch-Nyhan Disease. Scientific Reports. 6(1). 25592–25592. 20 indexed citations
7.
Hoskins, Peter R., et al.. (2005). B-mode compound imaging in mice. Ultrasound in Medicine & Biology. 32(1). 29–32. 7 indexed citations
8.
Hoskins, Peter R., et al.. (2005). Ultrasound B-mode 360° tomography in mice. 1. 752–755. 6 indexed citations
9.
Masuzaki, Hiroaki, Hiroshi Yamamoto, Christopher J. Kenyon, et al.. (2003). Transgenic amplification of glucocorticoid action in adipose tissue causes high blood pressure in mice. Journal of Clinical Investigation. 112(1). 83–90. 345 indexed citations
10.
Masuzaki, Hiroaki, Hiroshi Yamamoto, Christopher J. Kenyon, et al.. (2003). Transgenic amplification of glucocorticoid action in adipose tissue causes high blood pressure in mice. Journal of Clinical Investigation. 112(1). 83–90. 34 indexed citations
11.
Kazwala, R. R., Matthew G.F. Sharp, Gabriel Shirima, et al.. (2000). The prevalence of bovine tuberculosis in Rift Valley Districts based on single intradermal comparative tuberculin testing.. 20. 136–140. 1 indexed citations
12.
Sharp, Matthew G.F., Surasak Kantachuvesiri, & John J. Mullins. (1997). Genotype and cardiovascular phenotype: lesson from genetically manipulated animals and diseases humans. Current Opinion in Nephrology & Hypertension. 6(1). 51–57. 2 indexed citations
13.
Clark, Allan, Matthew G.F. Sharp, Steven D. Morley, et al.. (1997). Renin-1 Is Essential for Normal Renal Juxtaglomerular Cell Granulation and Macula Densa Morphology. Journal of Biological Chemistry. 272(29). 18185–18190. 76 indexed citations
14.
Sharp, Matthew G.F., et al.. (1996). Dissecting the function of the duplicated mouse renin genes by gene targeting.. Hypertension. 28(4). 708–708. 2 indexed citations
15.
Clark, Allan, Matthew G.F. Sharp, & John J. Mullins. (1996). Gene Targeting and Its Application to Basic Hypertension Research. Clinical Science. 90(6). 435–446. 4 indexed citations
16.
Véniant, Murielle M., et al.. (1995). Developmental studies demonstrate age-dependent elevation of renin activity in TGR(mRen2)27 rats. American Journal of Hypertension. 8(12). 1167–1176. 29 indexed citations
17.
Sharp, Matthew G.F. & John J. Mullins. (1993). Loss of gene function methodology. Journal of Hypertension. 11(4). 339–343. 5 indexed citations
18.
Sharp, Matthew G.F., Susan M. Adams, Rosemary A. Walker, William J. Brammar, & Jennifer M. Varley. (1992). Differential expression of the mitochondrial gene cytochrome oxidase ii in benign and malignant breast tissue. The Journal of Pathology. 168(2). 163–168. 47 indexed citations
19.
Adams, Susan M., Nicholas R. Helps, Matthew G.F. Sharp, et al.. (1992). Isolation and characterization of a novel gene with differential expression in benign and malignant human breast tumours. Human Molecular Genetics. 1(2). 91–96. 46 indexed citations
20.
Kyriacou, C. P., et al.. (1990). Clock mutations alter developmental timing in Drosophila. Heredity. 64(3). 395–401. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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