Matthew D. Disney
- Molecular Biology top 0.5%
- Cancer Research top 1%
- Cellular and Molecular Neuroscience top 2%
- Organic Chemistry top 2%
- Genetics top 2%
- Co-authors
- Jessica L. Childs‐DisneySai Pradeep VelagapudiPeter H. SeebergerDouglas H. TurnerMichael ZukerDavid H. MathewsSusan J. SchroederLirui Guan
- Topics
- RNA and protein synthesis mechanisms (124 papers)RNA Research and Splicing (101 papers)RNA modifications and cancer (69 papers)
- Partner nations
- United StatesSwitzerlandFrance
In The Last Decade
Matthew D. Disney
188 papers receiving 10.4k citations
Hit Papers
Peers
Comparison fields: 5 of 124
- Molecular Biology 9.5k
- Cancer Research 1.1k
- Cellular and Molecular Neuroscience 1.1k
- Organic Chemistry 832
- Genetics 719
Countries citing papers authored by Matthew D. Disney
This map shows the geographic impact of Matthew D. Disney's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew D. Disney with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew D. Disney more than expected).
Fields of papers citing papers by Matthew D. Disney
This network shows the impact of papers produced by Matthew D. Disney. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew D. Disney. The network helps show where Matthew D. Disney may publish in the future.
Co-authorship network of co-authors of Matthew D. Disney
This figure shows the co-authorship network connecting the top 25 collaborators of Matthew D. Disney. A scholar is included among the top collaborators of Matthew D. Disney based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew D. Disney. Matthew D. Disney is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 4 | |
| 5 | 1 | |
| 6 | 3 | |
| 7 | 28 | |
| 8 | 22 | |
| 9 | 14 | |
| 10 | 41 | |
| 11 | 44 | |
| 12 | 85 | |
| 13 | 91 | |
| 14 | 42 | |
| 15 | 14 | |
| 16 | 13 | |
| 17 | 31 | |
| 18 | 4 | |
| 19 | 165 | |
| 20 | 52 |
About Matthew D. Disney
Matthew D. Disney is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cancer Research, having authored 193 papers that have together received 10.6k indexed citations. Recurring topics across this work include RNA and protein synthesis mechanisms (124 papers), RNA Research and Splicing (101 papers) and RNA modifications and cancer (69 papers). The work is most often cited by research in Molecular Biology (9.5k citations), Cancer Research (1.1k citations) and Cellular and Molecular Neuroscience (1.1k citations). Matthew D. Disney has collaborated with scholars based in United States, Switzerland and France. Frequent co-authors include Jessica L. Childs‐Disney, Sai Pradeep Velagapudi, Peter H. Seeberger, Douglas H. Turner, Michael Zuker, David H. Mathews, Susan J. Schroeder, Lirui Guan, Matthew G. Costales and Tuan Anh Tran. Their work appears in journals such as Science, Chemical Reviews and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.