Mathilde Huyghe

1.9k total citations · 1 hit paper
14 papers, 1.4k citations indexed

About

Mathilde Huyghe is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Mathilde Huyghe has authored 14 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Oncology and 3 papers in Cancer Research. Recurrent topics in Mathilde Huyghe's work include Cancer Cells and Metastasis (10 papers), Developmental Biology and Gene Regulation (6 papers) and Hedgehog Signaling Pathway Studies (3 papers). Mathilde Huyghe is often cited by papers focused on Cancer Cells and Metastasis (10 papers), Developmental Biology and Gene Regulation (6 papers) and Hedgehog Signaling Pathway Studies (3 papers). Mathilde Huyghe collaborates with scholars based in France, United States and United Kingdom. Mathilde Huyghe's co-authors include Silvia Fré, Daniel Louvard, Spyros Artavanis‐Tsakonas, Sylvie Robine, Philippos Mourikis, Daniel Lafkas, Verónica Rodilla, S. K. Pallavi, Marick Laé and Klaus‐Peter Janssen and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Journal of Cell Biology.

In The Last Decade

Mathilde Huyghe

13 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Notch signals control the fate of immature progenitor cells in the intestine

2005 708 citations Silvia Fré, Mathilde Huyghe et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mathilde Huyghe France	9	973	615	309	187	110	14	1.4k
Tae-Hee Kim Canada	16	741 0.8×	485 0.8×	375 1.2×	117 0.6×	273 2.5×	40	1.3k
Alexandra Bell United Kingdom	10	704 0.7×	689 1.1×	168 0.5×	218 1.2×	92 0.8×	13	1.3k
Charbel Darido Australia	21	1.1k 1.2×	409 0.7×	313 1.0×	309 1.7×	156 1.4×	47	1.7k
Walid T. Khaled United Kingdom	18	951 1.0×	655 1.1×	209 0.7×	291 1.6×	76 0.7×	28	1.5k
Doug J. Winton United Kingdom	5	1.2k 1.2×	722 1.2×	430 1.4×	154 0.8×	226 2.1×	7	1.8k
Stephanie M. Bartley United States	12	1.5k 1.5×	464 0.8×	201 0.7×	223 1.2×	65 0.6×	15	1.8k
Duanduan Ma United States	17	1.1k 1.1×	459 0.7×	110 0.4×	211 1.1×	70 0.6×	28	1.6k
Lars Jønson Denmark	21	878 0.9×	214 0.3×	256 0.8×	338 1.8×	96 0.9×	36	1.5k
Eyal Reinstein Israel	16	1.0k 1.0×	324 0.5×	359 1.2×	89 0.5×	82 0.7×	41	1.5k
Krishna S. Ghanta United States	10	747 0.8×	333 0.5×	143 0.5×	108 0.6×	66 0.6×	12	1.1k

Countries citing papers authored by Mathilde Huyghe

Since Specialization

Citations

This map shows the geographic impact of Mathilde Huyghe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathilde Huyghe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathilde Huyghe more than expected).

Fields of papers citing papers by Mathilde Huyghe

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathilde Huyghe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathilde Huyghe. The network helps show where Mathilde Huyghe may publish in the future.

Co-authorship network of co-authors of Mathilde Huyghe

This figure shows the co-authorship network connecting the top 25 collaborators of Mathilde Huyghe. A scholar is included among the top collaborators of Mathilde Huyghe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mathilde Huyghe. Mathilde Huyghe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Huyghe, Mathilde, et al.. (2025). Conserved signals control self-organization and symmetry breaking of murine bilayered epithelia during development and regeneration. Developmental Cell. 60(19). 2576–2593.e6.

Sun, Wenjie, et al.. (2024). Spatially distinct epithelial and mesenchymal cell subsets along progressive lineage restriction in the branching embryonic mammary gland. The EMBO Journal. 43(12). 2308–2336. 6 indexed citations

Sun, Wenjie, Mathilde Huyghe, Marisa M. Faraldo, et al.. (2024). Extracting, filtering and simulating cellular barcodes using CellBarcode tools. Nature Computational Science. 4(2). 128–143. 1 indexed citations

Lloyd‐Lewis, Bethan, et al.. (2022). In vivo imaging of mammary epithelial cell dynamics in response to lineage-biased Wnt/β-catenin activation. Cell Reports. 38(10). 110461–110461. 9 indexed citations

Jacquemin, Guillaume, Mathilde Huyghe, Wenjie Sun, et al.. (2022). Paracrine signalling between intestinal epithelial and tumour cells induces a regenerative programme. eLife. 11. 7 indexed citations

Jacquemin, Guillaume, et al.. (2019). Lineage tracing of Notch1-expressing cells in intestinal tumours reveals a distinct population of cancer stem cells. Scientific Reports. 9(1). 888–888. 12 indexed citations

Lilja, Anna M., Verónica Rodilla, Mathilde Huyghe, et al.. (2018). Clonal analysis of Notch1-expressing cells reveals the existence of unipotent stem cells that retain long-term plasticity in the embryonic mammary gland. Nature Cell Biology. 20(6). 677–687. 99 indexed citations

Rodilla, Verónica, Mathilde Huyghe, Daniel Lafkas, et al.. (2015). Luminal Progenitors Restrict Their Lineage Potential during Mammary Gland Development. PLoS Biology. 13(2). e1002069–e1002069. 91 indexed citations

Germann, Markus, Huiling Xu, Jordane Malaterre, et al.. (2014). Tripartite interactions between Wnt signaling, Notch and Myb for stem/progenitor cell functions during intestinal tumorigenesis. Stem Cell Research. 13(3). 355–366. 14 indexed citations

10.

Lafkas, Daniel, et al.. (2013). Notch3 marks clonogenic mammary luminal progenitor cells in vivo. The Journal of Cell Biology. 203(1). 47–56. 78 indexed citations

11.

Fré, Silvia, Édouard Hannezo, Sanja Šale, et al.. (2011). Notch Lineages and Activity in Intestinal Stem Cells Determined by a New Set of Knock-In Mice. PLoS ONE. 6(10). e25785–e25785. 107 indexed citations

12.

Fré, Silvia, S. K. Pallavi, Mathilde Huyghe, et al.. (2009). Notch and Wnt signals cooperatively control cell proliferation and tumorigenesis in the intestine. Proceedings of the National Academy of Sciences. 106(15). 6309–6314. 263 indexed citations

13.

Fré, Silvia, Mathilde Huyghe, Philippos Mourikis, et al.. (2005). Notch signals control the fate of immature progenitor cells in the intestine. Nature. 435(7044). 964–968. 708 indexed citations breakdown →

14.

Robine, Sylvie, Silvia Fré, Mathilde Huyghe, Spyros Artavanis‐Tsakonas, & Daniel Louvard. (2005). Notch, un gène clé de l’équilibre entre prolifération et différenciation des cellules épithéliales de l’intestin. médecine/sciences. 21(8-9). 780–782. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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