Mathias Köppen

1.0k total citations
8 papers, 791 citations indexed

About

Mathias Köppen is a scholar working on Molecular Biology, Cell Biology and Aging. According to data from OpenAlex, Mathias Köppen has authored 8 papers receiving a total of 791 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Cell Biology and 3 papers in Aging. Recurrent topics in Mathias Köppen's work include Developmental Biology and Gene Regulation (4 papers), Congenital heart defects research (3 papers) and Cellular Mechanics and Interactions (3 papers). Mathias Köppen is often cited by papers focused on Developmental Biology and Gene Regulation (4 papers), Congenital heart defects research (3 papers) and Cellular Mechanics and Interactions (3 papers). Mathias Köppen collaborates with scholars based in Germany, United States and Chile. Mathias Köppen's co-authors include Carl‐Philipp Heisenberg, Jeffrey D. Hardin, Paul A. Sims, Jeffrey S. Simske, Christopher Rongo, David H. Hall, Anthony D. Radice, Bonnie L. Firestein, Lara Carvalho and Pablo Oteíza and has published in prestigious journals such as Nature Cell Biology, Development and Developmental Biology.

In The Last Decade

Mathias Köppen

8 papers receiving 783 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mathias Köppen Germany 7 460 389 276 122 91 8 791
Jeffrey D. Hardin United States 8 483 1.1× 537 1.4× 212 0.8× 167 1.4× 94 1.0× 10 906
Martha C. Soto United States 16 581 1.3× 526 1.4× 336 1.2× 117 1.0× 121 1.3× 24 982
Lihsia Chen United States 14 539 1.2× 466 1.2× 203 0.7× 106 0.9× 163 1.8× 20 893
Brian H. Lee United States 16 811 1.8× 279 0.7× 495 1.8× 182 1.5× 95 1.0× 22 1.2k
Suhong Xu China 15 473 1.0× 288 0.7× 160 0.6× 85 0.7× 72 0.8× 31 826
Baris Tursun Germany 20 944 2.1× 509 1.3× 121 0.4× 144 1.2× 81 0.9× 36 1.2k
Dana T. Byrd United States 7 415 0.9× 360 0.9× 196 0.7× 104 0.9× 46 0.5× 10 668
Jeffrey S. Simske United States 13 729 1.6× 894 2.3× 319 1.2× 324 2.7× 156 1.7× 20 1.4k
Anton Karabinoš Germany 17 581 1.3× 175 0.4× 295 1.1× 77 0.6× 65 0.7× 33 804
Sumeet Sarin United States 12 565 1.2× 335 0.9× 65 0.2× 85 0.7× 151 1.7× 12 928

Countries citing papers authored by Mathias Köppen

Since Specialization
Citations

This map shows the geographic impact of Mathias Köppen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathias Köppen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathias Köppen more than expected).

Fields of papers citing papers by Mathias Köppen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathias Köppen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathias Köppen. The network helps show where Mathias Köppen may publish in the future.

Co-authorship network of co-authors of Mathias Köppen

This figure shows the co-authorship network connecting the top 25 collaborators of Mathias Köppen. A scholar is included among the top collaborators of Mathias Köppen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mathias Köppen. Mathias Köppen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Oteíza, Pablo, Mathias Köppen, Michael Krieg, et al.. (2010). Planar cell polarity signalling regulates cell adhesion properties in progenitors of the zebrafish laterality organ. Development. 137(20). 3459–3468. 55 indexed citations
2.
Oteíza, Pablo, Mathias Köppen, Michael Krieg, et al.. (2009). S10-05 Linking organ formation to left–right patterning in the embryonic zebrafish. Mechanisms of Development. 126. S11–S11. 1 indexed citations
3.
Vervenne, Hilke, Lara Carvalho, Mathias Köppen, et al.. (2008). Lpp is involved in Wnt/PCP signaling and acts together with Scrib to mediate convergence and extension movements during zebrafish gastrulation. Developmental Biology. 320(1). 267–277. 23 indexed citations
4.
Oteíza, Pablo, Mathias Köppen, Miguel L. Concha, & Carl‐Philipp Heisenberg. (2008). Origin and shaping of the laterality organ in zebrafish. Development. 135(16). 2807–2813. 93 indexed citations
5.
Qadota, Hiroshi, Makiko Inoue, Takao Hikita, et al.. (2007). Establishment of a tissue-specific RNAi system in C. elegans. Gene. 400(1-2). 166–173. 154 indexed citations
6.
Köppen, Mathias, Beatriz García Fernández, Lara Carvalho, António Jacinto, & Carl‐Philipp Heisenberg. (2006). Coordinated cell-shape changes control epithelial movement in zebrafish and Drosophila. Development. 133(14). 2671–2681. 126 indexed citations
7.
Simske, Jeffrey S., et al.. (2003). The cell junction protein VAB-9 regulates adhesion and epidermal morphology in C. elegans. Nature Cell Biology. 5(7). 619–625. 79 indexed citations
8.
Köppen, Mathias, Jeffrey S. Simske, Paul A. Sims, et al.. (2001). Cooperative regulation of AJM-1 controls junctional integrity in Caenorhabditis elegans epithelia. Nature Cell Biology. 3(11). 983–991. 260 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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