Mathias Köppen
Impact in
- Aging top 0.5%
- Genetics, Aging, and Longevity in Model Organisms
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- Circadian rhythm and melatonin
Papers in
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- Developmental Biology and Gene Regulation 4
- Congenital heart defects research 3
- RNA Research and Splicing 2
- CRISPR and Genetic Engineering 1
- Heat shock proteins research 1
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- Cellular Mechanics and Interactions 3
- Hippo pathway signaling and YAP/TAZ 2
- Co-authors
- Carl‐Philipp Heisenberg (5 shared papers)Jeffrey D. Hardin (2 shared papers)Jeffrey S. Simske (2 shared papers)Paul A. Sims (2 shared papers)David H. Hall (1 shared paper)Bonnie L. Firestein (1 shared paper)Anthony D. Radice (1 shared paper)Christopher Rongo (1 shared paper)
- Journals
- Development (3 papers)Nature Cell Biology (2 papers)Gene (1 paper)Mechanisms of Development (1 paper)Developmental Biology (1 paper)
- Partner nations
- GermanyUnited StatesChile
In The Last Decade
Mathias Köppen
8 papers receiving 785 citations
Peers
Comparison fields: 5 of 73
- Aging 384
- Endocrine and Autonomic Systems 119
- Cell Biology 269
- Molecular Biology 446
- Neurology 33
Countries citing papers authored by Mathias Köppen
This map shows the geographic impact of Mathias Köppen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathias Köppen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathias Köppen more than expected).
Fields of papers citing papers by Mathias Köppen
This network shows the impact of papers produced by Mathias Köppen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathias Köppen. The network helps show where Mathias Köppen may publish in the future.
Co-authors
The 25 scholars most cited alongside Mathias Köppen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2001 | 259 | |
| 2 | 2007 | 156 | |
| 3 | 2006 | 127 | |
| 4 | 2008 | 94 | |
| 5 | 2003 | 78 | |
| 6 | 2010 | 55 | |
| 7 | 2008 | 23 | |
| 8 | 2009 | 1 |
About Mathias Köppen
Mathias Köppen is a scholar working on Molecular Biology, Cell Biology, Aging, Infectious Diseases and Organic Chemistry, having authored 8 papers that have together received 793 indexed citations. Recurring topics across this work include Developmental Biology and Gene Regulation (4 papers), Genetics, Aging, and Longevity in Model Organisms (3 papers), Cellular Mechanics and Interactions (3 papers), Congenital heart defects research (3 papers), RNA Research and Splicing (2 papers), Hippo pathway signaling and YAP/TAZ (2 papers), CRISPR and Genetic Engineering (1 paper) and Heat shock proteins research (1 paper). The work is most often cited by research in Aging (384 citations), Endocrine and Autonomic Systems (119 citations), Cell Biology (269 citations), Molecular Biology (446 citations) and Neurology (33 citations). Mathias Köppen has collaborated with scholars based in Germany, United States and Chile. Frequent co-authors include Carl‐Philipp Heisenberg, Jeffrey D. Hardin, Jeffrey S. Simske, Paul A. Sims, David H. Hall, Bonnie L. Firestein, Anthony D. Radice, Christopher Rongo, Pablo Oteíza and Miguel L. Concha. Their work appears in journals such as Development, Nature Cell Biology, Gene, Mechanisms of Development and Developmental Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.