Mathias Hohl

4.1k total citations
80 papers, 2.7k citations indexed

About

Mathias Hohl is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Physiology. According to data from OpenAlex, Mathias Hohl has authored 80 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Cardiology and Cardiovascular Medicine, 22 papers in Molecular Biology and 14 papers in Physiology. Recurrent topics in Mathias Hohl's work include Atrial Fibrillation Management and Outcomes (16 papers), Heart Rate Variability and Autonomic Control (16 papers) and Blood Pressure and Hypertension Studies (13 papers). Mathias Hohl is often cited by papers focused on Atrial Fibrillation Management and Outcomes (16 papers), Heart Rate Variability and Autonomic Control (16 papers) and Blood Pressure and Hypertension Studies (13 papers). Mathias Hohl collaborates with scholars based in Germany, Netherlands and Australia. Mathias Hohl's co-authors include Michael Böhm, Dominik Linz, Gerald Thiel, Felix Mahfoud, Ulrich Schotten, Benedikt Linz, Michael Lietz, Christoph Maack, Klaus Wirth and Dobromir Dobrev and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Journal of the American College of Cardiology.

In The Last Decade

Mathias Hohl

75 papers receiving 2.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mathias Hohl Germany 29 1.5k 832 483 342 246 80 2.7k
Naira Metreveli United States 29 831 0.6× 1.1k 1.3× 697 1.4× 280 0.8× 165 0.7× 59 2.8k
Aleksandar Jovanović United Kingdom 31 958 0.6× 1.4k 1.7× 541 1.1× 448 1.3× 156 0.6× 133 3.4k
Emmanuel S. Buys United States 32 862 0.6× 857 1.0× 857 1.8× 202 0.6× 316 1.3× 99 2.7k
Yoshiyuki Toya Japan 31 1.1k 0.7× 1.4k 1.6× 424 0.9× 514 1.5× 189 0.8× 143 3.2k
Pernille Hansen Denmark 34 961 0.6× 1.4k 1.7× 724 1.5× 423 1.2× 330 1.3× 107 3.1k
Johannes Stegbauer Germany 29 997 0.7× 663 0.8× 313 0.6× 339 1.0× 205 0.8× 97 2.4k
Yasuhide Nakashima Japan 29 1.4k 1.0× 749 0.9× 725 1.5× 825 2.4× 315 1.3× 174 3.1k
Giampiero Bricca France 27 873 0.6× 1.0k 1.3× 205 0.4× 270 0.8× 222 0.9× 105 2.5k
Manuel Portolés Spain 28 827 0.6× 901 1.1× 340 0.7× 424 1.2× 147 0.6× 161 2.6k
Heinz‐Gerd Zimmer Germany 30 1.5k 1.0× 1.1k 1.3× 372 0.8× 390 1.1× 209 0.8× 135 2.8k

Countries citing papers authored by Mathias Hohl

Since Specialization
Citations

This map shows the geographic impact of Mathias Hohl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathias Hohl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathias Hohl more than expected).

Fields of papers citing papers by Mathias Hohl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathias Hohl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathias Hohl. The network helps show where Mathias Hohl may publish in the future.

Co-authorship network of co-authors of Mathias Hohl

This figure shows the co-authorship network connecting the top 25 collaborators of Mathias Hohl. A scholar is included among the top collaborators of Mathias Hohl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mathias Hohl. Mathias Hohl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pepin, Mark E., Sumra Nazir, Christoph Maack, et al.. (2025). Mitochondrial NNT Promotes Diastolic Dysfunction in Cardiometabolic HFpEF. Circulation Research. 136(12). 1564–1578. 5 indexed citations
2.
Hohl, Mathias, Felix Götzinger, S. Jäger, et al.. (2025). Assessing phototoxic drug properties of hydrochlorothiazide using human skin biopsies. Communications Biology. 8(1). 705–705.
3.
Maas, Sanne L., Mathias Hohl, Matthias Bartneck, et al.. (2024). The calcium-sensing-receptor (CaSR) in adipocytes contributes to sex-differences in the susceptibility to high fat diet induced obesity and atherosclerosis. EBioMedicine. 107. 105293–105293. 5 indexed citations
4.
Wong, Dickson W.L., et al.. (2023). Semiautomated pipeline for quantitative analysis of heart histopathology. Journal of Translational Medicine. 21(1). 666–666. 5 indexed citations
5.
Hohl, Mathias, Lucas Lauder, Lea Wagmann, et al.. (2023). Efficacy of Antihypertensive Drugs of Different Classes After Renal Denervation in Spontaneously Hypertensive Rats. Hypertension. 80(6). e90–e100. 4 indexed citations
6.
Selejan, Simina‐Ramona, Dominik Linz, Mathias Hohl, et al.. (2022). Renal denervation reduces atrial remodeling in hypertensive rats with metabolic syndrome. Basic Research in Cardiology. 117(1). 36–36. 13 indexed citations
7.
Scott, Larry, Anke C. Fender, Arnela Saljic, et al.. (2021). NLRP3 inflammasome is a key driver of obesity-induced atrial arrhythmias. Cardiovascular Research. 117(7). 1746–1759. 107 indexed citations
8.
9.
Soppert, Josefin, Janina Frisch, Julia J. Wirth, et al.. (2021). A systematic review and meta-analysis of murine models of uremic cardiomyopathy. Kidney International. 101(2). 256–273. 23 indexed citations
10.
Hohl, Mathias, Manuel Mayr, Lisa A. Lang, et al.. (2020). Cathepsin A contributes to left ventricular remodeling by degrading extracellular superoxide dismutase in mice. Journal of Biological Chemistry. 295(36). 12605–12617. 16 indexed citations
11.
Linz, Benedikt, Mathias Hohl, Ricardo S. Mishima, et al.. (2020). Pharmacological inhibition of sodium-proton-exchanger subtype 3-mediated sodium absorption in the gut reduces atrial fibrillation susceptibility in obese spontaneously hypertensive rats. IJC Heart & Vasculature. 28. 100534–100534. 6 indexed citations
12.
13.
Linz, Dominik, Adrian D. Elliott, Mathias Hohl, et al.. (2018). Role of autonomic nervous system in atrial fibrillation. International Journal of Cardiology. 287. 181–188. 106 indexed citations
14.
Linz, Dominik, Mathias Hohl, Adrian D. Elliott, et al.. (2018). Modulation of renal sympathetic innervation: recent insights beyond blood pressure control. Clinical Autonomic Research. 28(4). 375–384. 16 indexed citations
15.
Linz, Dominik, Mathias Hohl, Stefan Dhein, et al.. (2016). Cathepsin A mediates susceptibility to atrial tachyarrhythmia and impairment of atrial emptying function in Zucker diabetic fatty rats. Cardiovascular Research. 110(3). 371–380. 31 indexed citations
16.
Linz, Dominik, Mathias Hohl, Felix Mahfoud, et al.. (2012). Cardiac remodeling and myocardial dysfunction in obese spontaneously hypertensive rats. Journal of Translational Medicine. 10(1). 187–187. 20 indexed citations
17.
Ekici, Myriam, et al.. (2011). Chromatin structure and expression of the AMPA receptor subunit Glur2 in human glioma cells: Major regulatory role of REST and Sp1. Journal of Cellular Biochemistry. 113(2). 528–543. 24 indexed citations
18.
Reil, Jan‐Christian, Mathias Hohl, Martin Oberhofer, et al.. (2010). Cardiac Rac1 overexpression in mice creates a substrate for atrial arrhythmias characterized by structural remodelling. Cardiovascular Research. 87(3). 485–493. 50 indexed citations
19.
Seeland, Ute, Simina‐Ramona Selejan, Mathias Hohl, et al.. (2009). Effects of AT1‐ and β‐adrenergic receptor antagonists on TGF‐β1‐induced fibrosis in transgenic mice. European Journal of Clinical Investigation. 39(10). 851–859. 25 indexed citations
20.
Richter, Ruth, et al.. (1977). Significance of oscillation frequency in intrapartum fetal monitoring.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 50(6). 694–700. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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