Mateusz Koptyra

2.3k total citations
35 papers, 958 citations indexed

About

Mateusz Koptyra is a scholar working on Hematology, Molecular Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Mateusz Koptyra has authored 35 papers receiving a total of 958 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Hematology, 15 papers in Molecular Biology and 11 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Mateusz Koptyra's work include Chronic Myeloid Leukemia Treatments (16 papers), Acute Lymphoblastic Leukemia research (11 papers) and Acute Myeloid Leukemia Research (8 papers). Mateusz Koptyra is often cited by papers focused on Chronic Myeloid Leukemia Treatments (16 papers), Acute Lymphoblastic Leukemia research (11 papers) and Acute Myeloid Leukemia Research (8 papers). Mateusz Koptyra collaborates with scholars based in United States, Poland and Germany. Mateusz Koptyra's co-authors include Tomasz Skórski, Margaret Nieborowska-Skorska, Tomasz Stokłosa, Michal O. Nowicki, Artur Słupianek, Janusz Błasiak, Rafal Falinski, Ireneusz Majsterek, Ewa Gloc and Ilona Seferyńska and has published in prestigious journals such as Journal of Biological Chemistry, Blood and Cancer Research.

In The Last Decade

Mateusz Koptyra

31 papers receiving 948 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mateusz Koptyra United States 15 597 424 349 182 179 35 958
Elisabeth Oppliger Leibundgut Switzerland 16 412 0.7× 480 1.1× 311 0.9× 102 0.6× 72 0.4× 38 897
Yuri Kamitsuji Japan 16 449 0.8× 369 0.9× 294 0.8× 174 1.0× 57 0.3× 32 929
Maria Kleppe United States 13 361 0.6× 459 1.1× 302 0.9× 92 0.5× 118 0.7× 24 947
Milada S. Vala United States 14 437 0.7× 349 0.8× 177 0.5× 56 0.3× 84 0.5× 20 860
Carla B. Ripamonti Italy 14 354 0.6× 452 1.1× 146 0.4× 73 0.4× 55 0.3× 29 907
L. H. Hoefsloot Netherlands 8 206 0.3× 240 0.6× 117 0.3× 128 0.7× 109 0.6× 9 645
Yongwei Su China 13 302 0.5× 563 1.3× 82 0.2× 72 0.4× 33 0.2× 27 688
T Fujimoto Japan 11 1.1k 1.8× 633 1.5× 370 1.1× 23 0.1× 391 2.2× 45 1.5k
Hua Dong China 10 228 0.4× 290 0.7× 256 0.7× 127 0.7× 16 0.1× 28 691
Michael Andreef United States 10 342 0.6× 316 0.7× 133 0.4× 20 0.1× 58 0.3× 17 698

Countries citing papers authored by Mateusz Koptyra

Since Specialization
Citations

This map shows the geographic impact of Mateusz Koptyra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mateusz Koptyra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mateusz Koptyra more than expected).

Fields of papers citing papers by Mateusz Koptyra

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mateusz Koptyra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mateusz Koptyra. The network helps show where Mateusz Koptyra may publish in the future.

Co-authorship network of co-authors of Mateusz Koptyra

This figure shows the co-authorship network connecting the top 25 collaborators of Mateusz Koptyra. A scholar is included among the top collaborators of Mateusz Koptyra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mateusz Koptyra. Mateusz Koptyra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Foster, Jessica, Peter J. Madsen, Nikhil Joshi, et al.. (2025). Transient mRNA CAR T cells targeting GD2 provide dose-adjusted efficacy against diffuse midline glioma and high-grade glioma models. Neuro-Oncology. 27(10). 2684–2696. 1 indexed citations
2.
Koptyra, Mateusz, Jo Lynne Rokita, Adam Kraya, et al.. (2024). METB-12. GIFT FROM A CHILD POSTMORTEM SPECIMEN DATASET EXTENSION OF THE PEDIATRIC BRAIN TUMOR ATLAS EMPOWERS PEDIATRIC BRAIN TUMOR RESEARCH. Neuro-Oncology. 26(Supplement_4). 0–0.
3.
Nabavizadeh, Ali, Chao Zhao, Daniel Martı́nez, et al.. (2022). TMET-37. SODIUM-DEPENDENT GLUCOSE TRANSPORTER 2 (SGLT2) IS OVEREXPRESSED IN THE MAJORITY OF ATRTS AND A SUBSET OF PEDIATRIC PATIENTS WITH HIGH-GRADE GLIOMA; A POTENTIAL IMAGING AND THERAPEUTIC TARGET. Neuro-Oncology. 24(Supplement_7). vii270–vii270. 1 indexed citations
4.
Rathi, Komal S., Mateusz Koptyra, Ammar S. Naqvi, et al.. (2020). A transcriptome-based classifier to determine molecular subtypes in medulloblastoma. PLoS Computational Biology. 16(10). e1008263–e1008263. 4 indexed citations
5.
Waanders, Angela J., Joost Wagenaar, Alex Felmeister, et al.. (2017). Abstract 2593: Accelerating pediatric brain tumor research through team science solutions. Cancer Research. 77(13_Supplement). 2593–2593. 1 indexed citations
6.
Park, Tae‐Ju, Mateusz Koptyra, & Tom Curran. (2016). Fibroblast Growth Requires CT10 Regulator of Kinase (Crk) and Crk-like (CrkL). Journal of Biological Chemistry. 291(51). 26273–26290. 14 indexed citations
7.
Dasgupta, Yashodhara, Mateusz Koptyra, Grażyna Hoser, et al.. (2016). Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases. Blood. 127(17). 2131–2143. 32 indexed citations
8.
Koptyra, Mateusz, Tae‐Ju Park, & Tom Curran. (2015). Crk and CrkL are required for cell transformation by v‐fos and v‐ras. Molecular Carcinogenesis. 55(1). 97–104. 5 indexed citations
9.
Hoang, David T., Lei Gu, Zhiyong Liao, et al.. (2015). Inhibition of Stat5a/b Enhances Proteasomal Degradation of Androgen Receptor Liganded by Antiandrogens in Prostate Cancer. Molecular Cancer Therapeutics. 14(3). 713–726. 16 indexed citations
10.
Dasgupta, Yashodhara, Mateusz Koptyra, Margaret Nieborowska-Skorska, et al.. (2013). Normal ABL1 Is a Tumor Suppressor and Therapeutic Target In BCR-ABL1–positive Leukemias. Blood. 122(21). 1466–1466. 1 indexed citations
11.
Schemionek, Mirle, Hans‐Ulrich Klein, Sylwia Flis, et al.. (2013). Genomic instability may originate from imatinib-refractory chronic myeloid leukemia stem cells. Blood. 121(20). 4175–4183. 82 indexed citations
12.
Manrique, A.C. Virgili, Mateusz Koptyra, Yashodhara Dasgupta, et al.. (2011). Imatinib Sensitivity in BCR-ABL1–Positive Chronic Myeloid Leukemia Cells Is Regulated by the Remaining Normal ABL1 Allele. Cancer Research. 71(16). 5381–5386. 10 indexed citations
13.
Koptyra, Mateusz, Tomasz Stokłosa, Grażyna Hoser, et al.. (2011). Monoubiquitinated Fanconi anemia D2 (FANCD2-Ub) is required for BCR-ABL1 kinase-induced leukemogenesis. Leukemia. 25(8). 1259–1267. 4 indexed citations
14.
Koptyra, Mateusz, et al.. (2011). Signal transducer and activator of transcription 5a/b: Biomarker and therapeutic target in prostate and breast cancer. The International Journal of Biochemistry & Cell Biology. 43(10). 1417–1421. 28 indexed citations
15.
Cramer, Kimberly, Margaret Nieborowska-Skorska, Mateusz Koptyra, et al.. (2008). BCR/ABL and Other Kinases from Chronic Myeloproliferative Disorders Stimulate Single-Strand Annealing, an Unfaithful DNA Double-Strand Break Repair. Cancer Research. 68(17). 6884–6888. 74 indexed citations
16.
Stokłosa, Tomasz, Tomasz Popławski, Mateusz Koptyra, et al.. (2008). BCR/ABL Inhibits Mismatch Repair to Protect from Apoptosis and Induce Point Mutations. Cancer Research. 68(8). 2576–2580. 65 indexed citations
17.
Koptyra, Mateusz, Rafal Falinski, Michal O. Nowicki, et al.. (2006). BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance. Blood. 108(1). 319–327. 224 indexed citations
18.
Słupianek, Artur, Michal O. Nowicki, Mateusz Koptyra, & Tomasz Skórski. (2005). BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells. DNA repair. 5(2). 243–250. 53 indexed citations
19.
Stokłosa, Tomasz, Artur Słupianek, Margaret Nieborowska-Skorska, et al.. (2004). BCR/ABL Recruits p53 Tumor Suppressor Protein to Induce Drug Resistance. Cell Cycle. 3(11). 1463–1472. 23 indexed citations
20.
Skórski, Tomasz, Michal O. Nowicki, Rafal Falinski, et al.. (2004). BCR/ABL Oncogenic Kinase Promotes Unfaithful Repair of the Reactive Oxygen Species - Dependent DNA Double-Strand Breaks.. Blood. 104(11). 712–712. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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