Mateusz Kolanczyk

1.6k total citations
23 papers, 680 citations indexed

About

Mateusz Kolanczyk is a scholar working on Molecular Biology, Neurology and Rheumatology. According to data from OpenAlex, Mateusz Kolanczyk has authored 23 papers receiving a total of 680 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 10 papers in Neurology and 8 papers in Rheumatology. Recurrent topics in Mateusz Kolanczyk's work include Neurofibromatosis and Schwannoma Cases (10 papers), Soft tissue tumor case studies (4 papers) and Sarcoma Diagnosis and Treatment (4 papers). Mateusz Kolanczyk is often cited by papers focused on Neurofibromatosis and Schwannoma Cases (10 papers), Soft tissue tumor case studies (4 papers) and Sarcoma Diagnosis and Treatment (4 papers). Mateusz Kolanczyk collaborates with scholars based in Germany, United States and Poland. Mateusz Kolanczyk's co-authors include Stefan Mundlos, Tomasz Żemojtel, Marlena Duchniewicz, Jirko Kühnisch, Jürgen Scheele, Nadine Kossler, Fried Zwartkruis, Steffen Großmann, Sigmar Stricker and Uwe Kornak and has published in prestigious journals such as Nucleic Acids Research, PLoS ONE and Molecular and Cellular Biology.

In The Last Decade

Mateusz Kolanczyk

23 papers receiving 673 citations

Peers

Mateusz Kolanczyk

NEURO

GENET

RHEUM

Catherine Sweeney Ireland

Hiroto Obata Japan

Debbie Hicks United Kingdom

Rob Dee Netherlands

Aaron Proweller United States

Heli Ylä‐Outinen Finland

Takuyu Taki Japan

Nina Schumacher Germany

Lauren M. Goddard United States

Patricia Thompson United States

Catherine Sweeney Ireland

Mateusz Kolanczyk

493 ×1.6

107 ×0.5

NEURO

80 ×0.7

GENET

116 ×1.0

RHEUM

79 ×1.0

Citations per year, relative to Mateusz Kolanczyk Mateusz Kolanczyk (= 1×) peers Catherine Sweeney

Countries citing papers authored by Mateusz Kolanczyk

Since Specialization

Citations

This map shows the geographic impact of Mateusz Kolanczyk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mateusz Kolanczyk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mateusz Kolanczyk more than expected).

Fields of papers citing papers by Mateusz Kolanczyk

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mateusz Kolanczyk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mateusz Kolanczyk. The network helps show where Mateusz Kolanczyk may publish in the future.

Co-authorship network of co-authors of Mateusz Kolanczyk

This figure shows the co-authorship network connecting the top 25 collaborators of Mateusz Kolanczyk. A scholar is included among the top collaborators of Mateusz Kolanczyk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mateusz Kolanczyk. Mateusz Kolanczyk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Hartl, Daniela, Claus‐Eric Ott, Monika Osswald, et al.. (2015). Double NF1 Inactivation Affects Adrenocortical Function in NF1Prx1 Mice and a Human Patient. PLoS ONE. 10(3). e0119030–e0119030. 9 indexed citations

Kühnisch, Jirko, Jong Seto, Claudia Lange, et al.. (2014). Neurofibromin inactivation impairs osteocyte development in Nf1Prx1 and Nf1Col1 mouse models. Bone. 66. 155–162. 8 indexed citations

Żemojtel, Tomasz, Jochen Hecht, Peter Krawitz, et al.. (2014). Somatic neurofibromatosis type 1 (NF1) inactivation events in cutaneous neurofibromas of a single NF1 patient. European Journal of Human Genetics. 23(6). 870–873. 16 indexed citations

Kolanczyk, Mateusz, Peter Krawitz, Jochen Hecht, et al.. (2014). Missense variant in CCDC22 causes X-linked recessive intellectual disability with features of Ritscher-Schinzel/3C syndrome. European Journal of Human Genetics. 23(5). 633–638. 43 indexed citations

Stevenson, David A., David G. Little, Linlea Armstrong, et al.. (2013). Approaches to Treating NF1 Tibial Pseudarthrosis. Journal of Pediatric Orthopaedics. 33(3). 269–275. 44 indexed citations

Sullivan, Kathleen E., Kate Quinlan, Fleur C. Garton, et al.. (2013). NF1 is a critical regulator of muscle development and metabolism. Human Molecular Genetics. 23(5). 1250–1259. 35 indexed citations

Jamsheer, Aleksander, Tomasz Żemojtel, Mateusz Kolanczyk, et al.. (2013). Whole exome sequencing identifies FGF16 nonsense mutations as the cause of X-linked recessive metacarpal 4/5 fusion. Journal of Medical Genetics. 50(9). 579–584. 25 indexed citations

Khassawna, Thaqif El, Daniel Toben, Mateusz Kolanczyk, et al.. (2012). Deterioration of fracture healing in the mouse model of NF1 long bone dysplasia. Bone. 51(4). 651–660. 21 indexed citations

Lange, Claudia, C. Li, I. Manjubala, et al.. (2011). Fetal and postnatal mouse bone tissue contains more calcium than is present in hydroxyapatite. Journal of Structural Biology. 176(2). 159–167. 35 indexed citations

10.

Kossler, Nadine, Sigmar Stricker, Christian Rödelsperger, et al.. (2011). Neurofibromin (Nf1) is required for skeletal muscle development. Human Molecular Genetics. 20(14). 2697–2709. 47 indexed citations

11.

Kolanczyk, Mateusz, Victor Mautner, Nadine Kossler, et al.. (2011). MIA is a potential biomarker for tumour load in neurofibromatosis type 1. BMC Medicine. 9(1). 82–82. 12 indexed citations

12.

Rödelsperger, Christian, Gao Guo, Mateusz Kolanczyk, et al.. (2010). Integrative analysis of genomic, functional and protein interaction data predicts long-range enhancer-target gene interactions. Nucleic Acids Research. 39(7). 2492–2502. 18 indexed citations

13.

Jamsheer, Aleksander, Mateusz Kolanczyk, Denise Horn, et al.. (2010). Osteopoikilosis and multiple exostoses caused by novel mutations in LEMD3 and EXT1 genes respectively - coincidence within one family. BMC Medical Genetics. 11(1). 110–110. 19 indexed citations

14.

Scheele, Jürgen, et al.. (2009). The Spt-Ada-Gcn5-acetyltransferase complex interaction motif of E2a is essential for a subset of transcriptional and oncogenic properties of E2a-Pbx1. Leukemia & lymphoma. 50(5). 816–828. 8 indexed citations

15.

Kolanczyk, Mateusz, Tomasz Żemojtel, Urs Lichtenauer, et al.. (2009). PBX1 is dispensable for neural commitment of RA-treated murine ES cells. In Vitro Cellular & Developmental Biology - Animal. 45(5-6). 252–263. 8 indexed citations

16.

Kolanczyk, Mateusz, Jirko Kühnisch, Nadine Kossler, et al.. (2008). Modelling neurofibromatosis type 1 tibial dysplasia and its treatment with lovastatin. BMC Medicine. 6(1). 21–21. 35 indexed citations

17.

Kolanczyk, Mateusz, Nadine Kossler, Jirko Kühnisch, et al.. (2007). Multiple roles for neurofibromin in skeletal development and growth. Human Molecular Genetics. 16(8). 874–886. 102 indexed citations

18.

Lichtenauer, Urs, Marlena Duchniewicz, Mateusz Kolanczyk, et al.. (2006). Pre-B-Cell Transcription Factor 1 and Steroidogenic Factor 1 Synergistically Regulate Adrenocortical Growth and Steroidogenesis. Endocrinology. 148(2). 693–704. 44 indexed citations

19.

Żemojtel, Tomasz, Mateusz Kolanczyk, Nadine Kossler, et al.. (2005). Mammalian mitochondrial nitric oxide synthase: Characterization of a novel candidate. FEBS Letters. 580(2). 455–462. 36 indexed citations

20.

Duchniewicz, Marlena, Tomasz Żemojtel, Mateusz Kolanczyk, et al.. (2005). Rap1A-Deficient T and B Cells Show Impaired Integrin-Mediated Cell Adhesion. Molecular and Cellular Biology. 26(2). 643–653. 101 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact