Massimo Statuto

647 total citations
17 papers, 549 citations indexed

About

Massimo Statuto is a scholar working on Molecular Biology, Cancer Research and Cell Biology. According to data from OpenAlex, Massimo Statuto has authored 17 papers receiving a total of 549 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Cancer Research and 3 papers in Cell Biology. Recurrent topics in Massimo Statuto's work include Fibroblast Growth Factor Research (5 papers), Connexins and lens biology (3 papers) and Protease and Inhibitor Mechanisms (3 papers). Massimo Statuto is often cited by papers focused on Fibroblast Growth Factor Research (5 papers), Connexins and lens biology (3 papers) and Protease and Inhibitor Mechanisms (3 papers). Massimo Statuto collaborates with scholars based in Italy, United States and France. Massimo Statuto's co-authors include G. Ragnotti, Jeanette A.M. Maier, Marco Rusnati, Marco Presta, Antonella Isacchi, Paolo Sarmientos, R Chiesa, Yvonne Munari‐Silem, Mattia Bertoli and Domenico Albano and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Massimo Statuto

17 papers receiving 541 citations

Peers

Massimo Statuto

IMMUN

PHYSI

Elizabeth Pöhler United Kingdom

Saiphone Webb United States

Hiroto Ishikura Japan

J.P. Salier France

Roberto González‐Amaro Mexico

Fred Elfman United States

Mahalakshmi Ramadass United States

Alejandro Morales United States

Lin Gao China

K Mafune Japan

Elizabeth Pöhler United Kingdom

Massimo Statuto

272 ×1.0

155 ×0.9

IMMUN

133 ×1.4

50 ×0.7

64 ×1.0

PHYSI

Citations per year, relative to Massimo Statuto Massimo Statuto (= 1×) peers Elizabeth Pöhler

Countries citing papers authored by Massimo Statuto

Since Specialization

Citations

This map shows the geographic impact of Massimo Statuto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Massimo Statuto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Massimo Statuto more than expected).

Fields of papers citing papers by Massimo Statuto

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Massimo Statuto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Massimo Statuto. The network helps show where Massimo Statuto may publish in the future.

Co-authorship network of co-authors of Massimo Statuto

This figure shows the co-authorship network connecting the top 25 collaborators of Massimo Statuto. A scholar is included among the top collaborators of Massimo Statuto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Massimo Statuto. Massimo Statuto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Albano, Domenico, Mattia Bertoli, Carlo Rodella, et al.. (2018). Prognostic role of pretreatment 18F-FDG PET/CT in primary brain lymphoma. Annals of Nuclear Medicine. 32(8). 532–541. 45 indexed citations

Giubbini, Raffaele, Alessia Peli, Elisa Milan, et al.. (2017). Comparison between the summed difference score and myocardial blood flow measured by 13N-ammonia. Journal of Nuclear Cardiology. 25(5). 1621–1628. 16 indexed citations

Statuto, Massimo, Elisa Galli, Francesco Bertagna, et al.. (2015). The strange case of the [13N]NH3. Nuclear Medicine Communications. 37(4). 412–421. 6 indexed citations

Statuto, Massimo, Cristina Bianchi, R Perego, & Ugo Del Monte. (2002). Drop of connexin 43 in replicative senescence of human fibroblasts HEL-299 as a possible biomarker of senescence. Experimental Gerontology. 37(8-9). 1113–1120. 26 indexed citations

Callé, Aleth, et al.. (2000). Formation of Three-Dimensional Thyroid Follicle-Like Structures by Polarized FRT Cells Made Communication Competent by Transfection and Stable Expression of the Connexin-32 Gene*. Endocrinology. 141(4). 1403–1413. 25 indexed citations

Statuto, Massimo, et al.. (1997). Restoration of Cell-to-Cell Communication in Thyroid Cell Lines by Transfection with and Stable Expression of the Connexin-32 Gene. Journal of Biological Chemistry. 272(39). 24710–24716. 37 indexed citations

Maier, Jeanette A.M., Massimo Statuto, & G. Ragnotti. (1994). Endogenous interleukin 1 alpha must be transported to the nucleus to exert its activity in human endothelial cells.. Molecular and Cellular Biology. 14(3). 1845–1851. 124 indexed citations

Maier, Jeanette A.M., Massimo Statuto, & G. Ragnotti. (1994). Endogenous Interleukin 1 Alpha Must Be Transported to the Nucleus To Exert Its Activity in Human Endothelial Cells. Molecular and Cellular Biology. 14(3). 1845–1851. 43 indexed citations

Maier, Jeanette A.M., Massimo Statuto, & G. Ragnotti. (1993). Senescence Stimulates U937-Endothelial Cell Interactions. Experimental Cell Research. 208(1). 270–274. 53 indexed citations

10.

Statuto, Massimo, Maria Grazia Ennas, Giuseppe Zamboni, et al.. (1993). Basic fibroblast growth factor in human pheochromocytoma: A biochemical and immunohistochemical study. International Journal of Cancer. 53(1). 5–10. 22 indexed citations

11.

Isacchi, Antonella, Massimo Statuto, R Chiesa, et al.. (1992). Activation of the tyrosine kinase receptor is not sufficient for the full biological activity of bFGF. Proceedings of the Fourth International Symposium on Polarization Phenomena in Nuclear Reactions. 61. 101–106. 1 indexed citations

12.

Presta, Marco, Massimo Statuto, Antonella Isacchi, et al.. (1992). Structure-function relationship of basic fibroblast growth factor: Site-directed mutagenesis of a putative heparin-binding and receptor-binding region. Biochemical and Biophysical Research Communications. 185(3). 1098–1107. 27 indexed citations

13.

Presta, Marco, Marco Rusnati, Chiara Urbinati, et al.. (1992). Basic fibroblast growth factor bound to cell substrate promotes cell adhesion, proliferation, and protease production in cultured endothelial cells. Proceedings of the Fourth International Symposium on Polarization Phenomena in Nuclear Reactions. 61. 205–209. 8 indexed citations

14.

Isacchi, Antonella, Massimo Statuto, Marco Rusnati, et al.. (1991). A Mutant of Basic Fibroblast Growth Factor that Has Lost the Ability to Stimulate Plasminogen Activator Synthesis in Endothelial Cells. Annals of the New York Academy of Sciences. 638(1). 369–377. 3 indexed citations

15.

Presta, Marco, Marco Rusnati, Chiara Urbinati, Massimo Statuto, & G. Ragnotti. (1991). Functional Domains of Basic Fibroblast Growth Factor: Possible Role of Asp‐Gly‐Arg Sequences in the Mitogenic Activity of bFGF. Annals of the New York Academy of Sciences. 638(1). 361–368. 5 indexed citations

16.

Isacchi, Antonella, Massimo Statuto, R Chiesa, et al.. (1991). A six-amino acid deletion in basic fibroblast growth factor dissociates its mitogenic activity from its plasminogen activator-inducing capacity.. Proceedings of the National Academy of Sciences. 88(7). 2628–2632. 73 indexed citations

17.

Presta, Marco, Massimo Statuto, Marco Rusnati, Patrizia Dell’Era, & G. Ragnotti. (1989). Characterization of a Mr 25,000 basic fibroblast growth factor form in adult, regenerating, and fetal rat liver. Biochemical and Biophysical Research Communications. 164(3). 1182–1189. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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