Masaya Matsuda

436 total citations
35 papers, 339 citations indexed

About

Masaya Matsuda is a scholar working on Physiology, Immunology and Surgery. According to data from OpenAlex, Masaya Matsuda has authored 35 papers receiving a total of 339 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Physiology, 17 papers in Immunology and 9 papers in Surgery. Recurrent topics in Masaya Matsuda's work include Asthma and respiratory diseases (16 papers), IL-33, ST2, and ILC Pathways (13 papers) and Eosinophilic Esophagitis (7 papers). Masaya Matsuda is often cited by papers focused on Asthma and respiratory diseases (16 papers), IL-33, ST2, and ILC Pathways (13 papers) and Eosinophilic Esophagitis (7 papers). Masaya Matsuda collaborates with scholars based in Japan, United States and Germany. Masaya Matsuda's co-authors include Takeshi Nabe, Kazuyuki Kitatani, Ryo Kawata, Tetsuya Terada, Koichi Okutani, Nobuaki Mizutani, Xuewei Zhang, Nobuo Yaegashi, Kenichi Sogawa and Hideo Ayame and has published in prestigious journals such as SHILAP Revista de lepidopterología, The Journal of Immunology and The FASEB Journal.

In The Last Decade

Masaya Matsuda

32 papers receiving 327 citations

Peers

Masaya Matsuda
Elizabeth Townsend United Kingdom
Rafeul Alam United States
Qi Tan China
Miranda L. Curtiss United States
Swati Agrawal United States
Susanne Grond United States
Lorna Wood Canada
Elizabeth Townsend United Kingdom
Masaya Matsuda
Citations per year, relative to Masaya Matsuda Masaya Matsuda (= 1×) peers Elizabeth Townsend

Countries citing papers authored by Masaya Matsuda

Since Specialization
Citations

This map shows the geographic impact of Masaya Matsuda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masaya Matsuda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masaya Matsuda more than expected).

Fields of papers citing papers by Masaya Matsuda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masaya Matsuda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masaya Matsuda. The network helps show where Masaya Matsuda may publish in the future.

Co-authorship network of co-authors of Masaya Matsuda

This figure shows the co-authorship network connecting the top 25 collaborators of Masaya Matsuda. A scholar is included among the top collaborators of Masaya Matsuda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masaya Matsuda. Masaya Matsuda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Matsuda, Masaya, et al.. (2025). Inhibition of cyclin-dependent kinase 4/6 attenuates airway remodeling in a murine severe asthma model by suppressing group 2 innate lymphoid cells proliferation. Journal of Pharmacology and Experimental Therapeutics. 392(11). 103726–103726.
2.
Matsuda, Masaya, et al.. (2025). Essential roles of mechanistic target of rapamycin in the induction of steroid resistance in group 2 innate lymphoid cells and severe asthma. Journal of Pharmacology and Experimental Therapeutics. 392(11). 103744–103744.
3.
Matsuda, Masaya, K. Matsuo, Kazuyuki Kitatani, et al.. (2025). Cyclin-dependent kinase (CDK) 8 and its paralog CDK19 develop group 2 innate lymphoid cell–related lung fibrosis by activating STAT5. The Journal of Immunology. 214(12). 3238–3249. 2 indexed citations
4.
Matsuda, Masaya, et al.. (2024). Involvement of CCR5 on interstitial macrophages in the development of lung fibrosis in severe asthma. International Immunopharmacology. 135. 112331–112331. 2 indexed citations
5.
Matsuda, Masaya, et al.. (2024). Involvement of Janus kinase‐dependent Bcl‐xL overexpression in steroid resistance of group 2 innate lymphoid cells in asthma. Immunology. 172(4). 653–668. 7 indexed citations
6.
Matsuda, Masaya, et al.. (2024). Steroid-Insensitive Gene Expression of Extracellular Matrix Components and Pro-fibrotic Factors in the Lung Associated with Airway Hyperresponsiveness in Murine Asthma. Biological and Pharmaceutical Bulletin. 47(1). 227–231. 2 indexed citations
7.
Ohya, Yuki, Masaya Matsuda, Kentaro Nishida, et al.. (2024). Pronecroptotic Therapy Using Ceramide Nanoliposomes Is Effective for Triple-Negative Breast Cancer Cells. Cells. 13(5). 405–405. 5 indexed citations
8.
Matsuda, Masaya, et al.. (2022). Local IL-10 replacement therapy was effective for steroid-insensitive asthma in mice. International Immunopharmacology. 110. 109037–109037. 10 indexed citations
9.
Matsuda, Masaya, Tetsuya Terada, Kazuyuki Kitatani, Ryo Kawata, & Takeshi Nabe. (2022). Roles of type 1 regulatory T (Tr1) cells in allergen-specific immunotherapy. SHILAP Revista de lepidopterología. 3. 981126–981126. 15 indexed citations
10.
Terada, Tetsuya, et al.. (2021). Sublingual immunotherapy for 4 years increased the number of Foxp3+ Treg cells, which correlated with clinical effects. Inflammation Research. 70(5). 581–589. 12 indexed citations
11.
Matsumoto, Misaki, Junjie Liu, K Iwata, et al.. (2021). NOX1/NADPH oxidase is involved in the LPS-induced exacerbation of collagen-induced arthritis. Journal of Pharmacological Sciences. 146(2). 88–97. 7 indexed citations
12.
Terada, Tetsuya, et al.. (2020). Sustained effects of intralymphatic pollen-specific immunotherapy on Japanese cedar pollinosis. Rhinology Journal. 0(0). 0–0. 20 indexed citations
13.
Matsuda, Masaya, et al.. (2019). Regulatory T and B Cells in Peripheral Blood of Subcutaneous Immunotherapy-Treated Japanese Cedar Pollinosis Patients. Immunotherapy. 11(6). 473–482. 11 indexed citations
14.
Matsuda, Masaya, et al.. (2019). Adoptive transfer of type 1 regulatory T cells suppressed the development of airway hyperresponsiveness in ovalbumin-induced airway inflammation model mice. Journal of Pharmacological Sciences. 141(4). 139–145. 13 indexed citations
15.
Matsuda, Masaya, et al.. (2018). Phenotype analyses of IL-10-producing Foxp3 − CD4 + T cells increased by subcutaneous immunotherapy in allergic airway inflammation. International Immunopharmacology. 61. 297–305. 20 indexed citations
16.
Matsuda, Masaya, et al.. (2017). Regulation of allergic airway inflammation by adoptive transfer of CD4 + T cells preferentially producing IL-10. European Journal of Pharmacology. 812. 38–47. 22 indexed citations
17.
Nabe, Takeshi, Akihiro Nishiguchi, Masaya Matsuda, et al.. (2015). Production of interleukin (IL)-33 in the lungs during multiple antigen challenge-induced airway inflammation in mice, and its modulation by a glucocorticoid. European Journal of Pharmacology. 757. 34–41. 39 indexed citations
18.
Matsuda, Masaya, et al.. (2001). [Low dose intrathecal morphine and postoperative pain relief in elderly patients].. PubMed. 50(10). 1096–100. 7 indexed citations
19.
Ikezawa, Zenrō, Hideo Ogura, Hiroshi Odajima, et al.. (1992). Mass trial of hypoallergenic rice (HRS-1) produced by enzymatic digestion in atopic dermatitis with suspected rice allergy. 108–112. 10 indexed citations
20.
Tomimoto, Hidekazu, Keith H. Ogawa, Masaya Matsuda, & K Shimada. (1984). [A case report of infarction of the bilateral thalamus and the midbrain].. PubMed. 36(6). 553–7. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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