Masamitsu Iwasa

524 total citations
21 papers, 443 citations indexed

About

Masamitsu Iwasa is a scholar working on Cardiology and Cardiovascular Medicine, Pathology and Forensic Medicine and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Masamitsu Iwasa has authored 21 papers receiving a total of 443 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cardiology and Cardiovascular Medicine, 8 papers in Pathology and Forensic Medicine and 8 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Masamitsu Iwasa's work include Cardiac Ischemia and Reperfusion (8 papers), Cardiovascular Function and Risk Factors (6 papers) and Renin-Angiotensin System Studies (4 papers). Masamitsu Iwasa is often cited by papers focused on Cardiac Ischemia and Reperfusion (8 papers), Cardiovascular Function and Risk Factors (6 papers) and Renin-Angiotensin System Studies (4 papers). Masamitsu Iwasa collaborates with scholars based in Japan and United States. Masamitsu Iwasa's co-authors include Kazuhiko Nishigaki, Shinya Minatoguchi, Yoshihisa Yamada, Genzou Takemura, Itta Kawamura, Hisayoshi Fujiwara, Shinji Yasuda, Takako Fujiwara, Hiroaki Ushikoshi and Masanori Kawasaki and has published in prestigious journals such as PLoS ONE, British Journal of Pharmacology and Cardiovascular Research.

In The Last Decade

Masamitsu Iwasa

21 papers receiving 440 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Masamitsu Iwasa Japan	14	166	153	92	80	76	21	443
Qiangsun Zheng China	15	222 1.3×	242 1.6×	33 0.4×	40 0.5×	65 0.9×	50	666
Nicholas J. Edmunds United Kingdom	11	224 1.3×	140 0.9×	54 0.6×	50 0.6×	22 0.3×	13	451
Nancy Amaral Rebouças Brazil	15	383 2.3×	86 0.6×	208 2.3×	91 1.1×	68 0.9×	40	729
Mariano Schuman Argentina	11	90 0.5×	81 0.5×	128 1.4×	33 0.4×	21 0.3×	20	368
James Simpkin United Kingdom	5	242 1.5×	118 0.8×	31 0.3×	200 2.5×	88 1.2×	6	633
James S. McTaggart United Kingdom	7	306 1.8×	42 0.3×	120 1.3×	164 2.0×	62 0.8×	9	588
Jui Shah United States	12	275 1.7×	106 0.7×	174 1.9×	39 0.5×	16 0.2×	26	533
C Wei United States	8	286 1.7×	271 1.8×	88 1.0×	52 0.7×	41 0.5×	15	609
Nancy Schanze Germany	12	152 0.9×	52 0.3×	87 0.9×	40 0.5×	47 0.6×	22	398

Countries citing papers authored by Masamitsu Iwasa

Since Specialization

Citations

This map shows the geographic impact of Masamitsu Iwasa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masamitsu Iwasa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masamitsu Iwasa more than expected).

Fields of papers citing papers by Masamitsu Iwasa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masamitsu Iwasa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masamitsu Iwasa. The network helps show where Masamitsu Iwasa may publish in the future.

Co-authorship network of co-authors of Masamitsu Iwasa

This figure shows the co-authorship network connecting the top 25 collaborators of Masamitsu Iwasa. A scholar is included among the top collaborators of Masamitsu Iwasa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masamitsu Iwasa. Masamitsu Iwasa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Yoshida, Akihiro, Hiromitsu Kanamori, Shingo Minatoguchi, et al.. (2019). (Pro)renin Receptor Blockade Ameliorates Heart Failure Caused by Chronic Kidney Disease. Journal of Cardiac Failure. 25(4). 286–300. 13 indexed citations

Kanamori, Hiromitsu, Akihiro Yoshida, Shingo Minatoguchi, et al.. (2019). The intestine responds to heart failure by enhanced mitochondrial fusion through glucagon-like peptide-1 signalling. Cardiovascular Research. 115(13). 1873–1885. 17 indexed citations

Iwasa, Masamitsu, Yuka Hayakawa, Shingo Minatoguchi, et al.. (2018). Azilsartan attenuates cardiac damage caused by high salt intake through the downregulation of the cardiac (pro)renin receptor and its downstream signals in spontaneously hypertensive rats. Hypertension Research. 41(11). 886–896. 8 indexed citations

Tanaka, Toshiki, Takuma Aoyama, Shingo Minatoguchi, et al.. (2018). Endogenous Adenosine May Be Related to Left Ventricular Dysfunction, Dilation, and Wall Thinning in Patients With Heart Disease. Circulation Journal. 82(5). 1319–1326. 4 indexed citations

Hayakawa, Yuka, Takuma Aoyama, Shingo Minatoguchi, et al.. (2017). Excessively low salt diet damages the heart through activation of cardiac (pro) renin receptor, renin-angiotensin-aldosterone, and sympatho-adrenal systems in spontaneously hypertensive rats. PLoS ONE. 12(12). e0189099–e0189099. 17 indexed citations

Iwasa, Masamitsu, Shingo Minatoguchi, Yoshihisa Yamada, et al.. (2017). Antidiabetic Drug Alogliptin Protects the Heart Against Ischemia-reperfusion Injury Through GLP-1 Receptor-dependent and Receptor-independent Pathways Involving Nitric Oxide Production in Rabbits. Journal of Cardiovascular Pharmacology. 70(6). 382–389. 8 indexed citations

Kawabe, Tetsuya, et al.. (2016). Attenuation of angiotensin type 2 receptor function in the rostral ventrolateral medullary pressor area of the spontaneously hypertensive rat. Clinical and Experimental Hypertension. 38(2). 209–217. 2 indexed citations

Kawasaki, Masanori, Masamitsu Iwasa, Hiromitsu Kanamori, et al.. (2016). [Relationship between Coronary Plaque Stability Evaluated by Intravascular Ultrasound and Laboratory Parameters].. PubMed. 64(3). 319–26. 1 indexed citations

Yamada, Yoshihisa, Shingo Minatoguchi, Masamitsu Iwasa, et al.. (2015). MicroRNA-145 repairs infarcted myocardium by accelerating cardiomyocyte autophagy. American Journal of Physiology-Heart and Circulatory Physiology. 309(11). H1813–H1826. 68 indexed citations

10.

Hayakawa, Yuka, Takuma Aoyama, Shingo Minatoguchi, et al.. (2015). High Salt Intake Damages the Heart through Activation of Cardiac (Pro) Renin Receptors Even at an Early Stage of Hypertension. PLoS ONE. 10(3). e0120453–e0120453. 39 indexed citations

11.

Iwasa, Masamitsu, et al.. (2013). Activation of melanocortin receptors in the intermediolateral cell column of the upper thoracic cord elicits tachycardia in the rat. American Journal of Physiology-Heart and Circulatory Physiology. 305(6). H885–H893. 19 indexed citations

12.

Yamada, Yoshihisa, Hiroyuki Kobayashi, Masamitsu Iwasa, et al.. (2013). Postinfarct active cardiac-targeted delivery of erythropoietin by liposomes with sialyl Lewis X repairs infarcted myocardium in rabbits. American Journal of Physiology-Heart and Circulatory Physiology. 304(8). H1124–H1133. 22 indexed citations

13.

Iwasa, Masamitsu, Yoshihisa Yamada, Hiroyuki Kobayashi, et al.. (2011). Both stimulation of GLP-1 receptors and inhibition of glycogenolysis additively contribute to a protective effect of oral miglitol against ischaemia-reperfusion injury in rabbits. British Journal of Pharmacology. 164(1). 119–131. 17 indexed citations

14.

Iwasa, Masamitsu, Hiroyuki Kobayashi, Shinji Yasuda, et al.. (2010). Antidiabetic Drug Voglibose Is Protective Against Ischemia—Reperfusion Injury Through Glucagon-Like Peptide 1 Receptors and the Phosphoinositide 3-Kinase-Akt-Endothelial Nitric Oxide Synthase Pathway in Rabbits. Journal of Cardiovascular Pharmacology. 55(6). 625–634. 18 indexed citations

15.

Kobayashi, Hiroyuki, Shinji Yasuda, Masamitsu Iwasa, et al.. (2010). Postconditioning effect of granulocyte colony-stimulating factor is mediated through activation of risk pathway and opening of the mitochondrial K_ATP channels. American Journal of Physiology-Heart and Circulatory Physiology. 299(4). H1174–H1182. 16 indexed citations

16.

Kobayashi, Hiroyuki, Shinji Yasuda, Masamitsu Iwasa, et al.. (2009). Postinfarct Treatment With Oxytocin Improves Cardiac Function and Remodeling via Activating Cell-survival Signals and Angiogenesis. Journal of Cardiovascular Pharmacology. 54(6). 510–519. 55 indexed citations

17.

Minatoguchi, Shinya, Hiroyuki Kobayashi, Shinji Yasuda, et al.. (2009). Acarbose Reduces Myocardial Infarct Size by Preventing Postprandial Hyperglycemia and Hydroxyl Radical Production and Opening Mitochondrial KATP Channels in Rabbits. Journal of Cardiovascular Pharmacology. 54(1). 25–30. 13 indexed citations

18.

Yasuda, Shinji, Hiroyuki Kobayashi, Masamitsu Iwasa, et al.. (2009). Antidiabetic drug pioglitazone protects the heart via activation of PPAR-γ receptors, PI3-kinase, Akt, and eNOS pathway in a rabbit model of myocardial infarction. American Journal of Physiology-Heart and Circulatory Physiology. 296(5). H1558–H1565. 49 indexed citations

19.

Ushikoshi, Hiroaki, Hiroyuki Kobayashi, Shinji Yasuda, et al.. (2008). Simvastatin reduces myocardial infarct size via increased nitric oxide production in normocholesterolemic rabbits. Journal of Cardiology. 53(1). 102–107. 14 indexed citations

20.

Yasuda, Shinji, Hiroyuki Kobayashi, Masamitsu Iwasa, et al.. (2008). Anti-Diabetic Drug Pioglitazone Protects the Heart Via Activation of PPAR-γ Receptors, PI3 Kinase , Akt and eNOS pathway in a Rabbit Model of Myocardial Infarction. Journal of Molecular and Cellular Cardiology. 45(4). S15–S15. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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