Masahiro Itonaga
- Oncology top 5%
- Surgery top 5%
- Pulmonary and Respiratory Medicine top 5%
- Epidemiology
- Radiology, Nuclear Medicine and Imaging
- Co-authors
- Masayuki KitanoYasunobu YamashitaTakashi TamuraKeiichi HatamaruTakeichi YoshidaYuki KawajiTakao MaekitaMasao Ichinose
- Topics
- Pancreatic and Hepatic Oncology Research (49 papers)Gallbladder and Bile Duct Disorders (35 papers)Pediatric Hepatobiliary Diseases and Treatments (18 papers)
- Cited by
- OncologySurgeryGastroenterology
- Partner nations
- Japan
In The Last Decade
Masahiro Itonaga
70 papers receiving 985 citations
Peers
Comparison fields: 5 of 73
- Oncology 689
- Surgery 684
- Pulmonary and Respiratory Medicine 455
- Epidemiology 182
- Radiology, Nuclear Medicine and Imaging 105
Countries citing papers authored by Masahiro Itonaga
This map shows the geographic impact of Masahiro Itonaga's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masahiro Itonaga with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masahiro Itonaga more than expected).
Fields of papers citing papers by Masahiro Itonaga
This network shows the impact of papers produced by Masahiro Itonaga. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masahiro Itonaga. The network helps show where Masahiro Itonaga may publish in the future.
Co-authorship network of co-authors of Masahiro Itonaga
This figure shows the co-authorship network connecting the top 25 collaborators of Masahiro Itonaga. A scholar is included among the top collaborators of Masahiro Itonaga based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masahiro Itonaga. Masahiro Itonaga is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 1 | |
| 4 | 1 | |
| 5 | 2 | |
| 6 | 0 | |
| 7 | 2 | |
| 8 | 3 | |
| 9 | 0 | |
| 10 | 7 | |
| 11 | 7 | |
| 12 | 5 | |
| 13 | 11 | |
| 14 | 9 | |
| 15 | 18 | |
| 16 | 41 | |
| 17 | 9 | |
| 18 | 5 | |
| 19 | 5 | |
| 20 | CHOLECYSTOKININ ANTAGONISM BY BENZODIAZEPINES : A NEW APPROACH TO THE MECHANISM OF ACTION OF BENZODIAZEPINES | 1 |
About Masahiro Itonaga
Masahiro Itonaga is a scholar working on Oncology, Gastroenterology and Pulmonary and Respiratory Medicine, having authored 77 papers that have together received 1.0k indexed citations. Recurring topics across this work include Pancreatic and Hepatic Oncology Research (49 papers), Gallbladder and Bile Duct Disorders (35 papers) and Pediatric Hepatobiliary Diseases and Treatments (18 papers). The work is most often cited by research in Oncology (689 citations), Surgery (684 citations) and Gastroenterology (77 citations). Masahiro Itonaga has collaborated with scholars based in Japan. Frequent co-authors include Masayuki Kitano, Yasunobu Yamashita, Takashi Tamura, Keiichi Hatamaru, Takeichi Yoshida, Yuki Kawaji, Takao Maekita, Masao Ichinose, Mikitaka Iguchi and Reiko Ashida. Their work appears in journals such as SHILAP Revista de lepidopterología, PLoS ONE and Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.