Masaaki Ochi
About
Masaaki Ochi is a scholar working on Epidemiology, Physiology and Endocrine and Autonomic Systems.
According to data from OpenAlex, Masaaki Ochi has authored 29 papers receiving a total of 884 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Epidemiology, 15 papers in Physiology and 13 papers in Endocrine and Autonomic Systems. Recurrent topics in Masaaki Ochi's work include Adipokines, Inflammation, and Metabolic Diseases (17 papers), Adipose Tissue and Metabolism (15 papers) and Regulation of Appetite and Obesity (13 papers). Masaaki Ochi is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (17 papers), Adipose Tissue and Metabolism (15 papers) and Regulation of Appetite and Obesity (13 papers). Masaaki Ochi collaborates with scholars based in Japan and United States. Masaaki Ochi's co-authors include Haruhiko Osawa, Hideichi Makino, Hiroshi Onuma, Jun Ohashi, Wataru Nishida, Yasuhisa Fujii, Ikki Shimizu, Akiko Murakami, Tatsuya Nishimiya and Kazuya Yamada and has published in prestigious journals such as PLoS ONE, The Journal of Clinical Endocrinology & Metabolism and Diabetes Care.
In The Last Decade
Masaaki Ochi
29 papers receiving 847 citations
Peers
Masaaki Ochi
Hirokazu Yago Japan
Shuji Oguchi Japan
Steve Hurel United Kingdom
Soo Jeong Koh South Korea
Maria Rosaria Manigrasso Italy
D Melina Italy
K.-D. Palitzsch Germany
Eun Shil Hong South Korea
Rafał Nikodem Wlazeł Poland
Hiroshi Tatsukawa Japan
Citations per year, relative to Masaaki Ochi Masaaki Ochi (= 1×)
peers
Hirokazu Yago
Countries citing papers authored by Masaaki Ochi
Since
Specialization
Citations
This map shows the geographic impact of Masaaki Ochi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masaaki Ochi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masaaki Ochi more than expected).
Fields of papers citing papers by Masaaki Ochi
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Masaaki Ochi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masaaki Ochi. The network helps show where Masaaki Ochi may publish in the future.
Co-authorship network of co-authors of Masaaki Ochi
This figure shows the co-authorship network connecting the top 25 collaborators of Masaaki Ochi. A scholar is included among the top collaborators of Masaaki Ochi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masaaki Ochi. Masaaki Ochi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Onuma, Hiroshi, Yasuharu Tabara, Ryoichi Kawamura, et al.. (2016). Dual Effects of a RETN Single Nucleotide Polymorphism (SNP) at –420 on Plasma Resistin: Genotype and DNA Methylation. The Journal of Clinical Endocrinology & Metabolism. 102(3). 884–892.
2.
Onuma, Hiroshi, Yasuharu Tabara, Ryoichi Kawamura, et al.. (2012). Plasma Resistin Is Associated With Single Nucleotide Polymorphisms of a Possible Resistin Receptor, the Decorin Gene, in the General Japanese Population. Diabetes. 62(2). 649–652.
3.
Onuma, Hiroshi, Yasuharu Tabara, Ryoichi Kawamura, et al.. (2010). A at Single Nucleotide Polymorphism-358 Is Required for G at -420 to Confer the Highest Plasma Resistin in the General Japanese Population. PLoS ONE. 5(3). e9718–e9718.
4.
Osawa, Haruhiko, Yasuharu Tabara, Ryuichi Kawamoto, et al.. (2009). PPARγ Pro12Ala Pro/Pro and resistin SNP‐420 G/G genotypes are synergistically associated with plasma resistin in the Japanese general population. Clinical Endocrinology. 71(3). 341–345.
5.
Yamauchi, Junko, Haruhiko Osawa, Masaaki Ochi, et al.. (2008). Serum resistin is reduced by glucose and meal loading in healthy human subjects. Metabolism. 57(2). 149–156.
6.
Osawa, Haruhiko, Kazuya Yamada, Yasuharu Tabara, et al.. (2008). The G/G genotype of a single nucleotide polymorphism at –1066 of c‐Jun N‐terminal kinase 1 gene (MAPK8) does not affect type 2 diabetes susceptibility despite the specific binding of AP2α. Clinical Endocrinology. 69(1). 36–44.
7.
Osawa, Haruhiko, Masaaki Ochi, Yasuharu Tabara, et al.. (2008). Serum resistin is positively correlated with the accumulation of metabolic syndrome factors in type 2 diabetes. Clinical Endocrinology. 69(1). 74–80.
8.
Kato, Kenichi, Haruhiko Osawa, Masaaki Ochi, et al.. (2007). Serum total and high molecular weight adiponectin levels are correlated with the severity of diabetic retinopathy and nephropathy. Clinical Endocrinology. 68(3). 442–449.
9.
Osawa, Haruhiko, Masaaki Ochi, Kenichi Kato, et al.. (2007). Serum resistin is associated with the severity of microangiopathies in type 2 diabetes. Biochemical and Biophysical Research Communications. 355(2). 342–346.
10.
Ochi, Masaaki, Haruhiko Osawa, Yushi Hirota, et al.. (2007). Frequency of the G/G Genotype of Resistin Single Nucleotide Polymorphism at −420 Appears to Be Increased in Younger-Onset Type 2 Diabetes. Diabetes. 56(11). 2834–2838.
11.
Osawa, Haruhiko, Hiroshi Onuma, Masaaki Ochi, et al.. (2005). Resistin SNP-420 determines its monocyte mRNA and serum levels inducing type 2 diabetes. Biochemical and Biophysical Research Communications. 335(2). 596–602.
12.
Yoshioka, Yuki, Kaori Hashimoto, Masaaki Ochi, et al.. (2005). A simple precipitation method for measuring sialic acid in apolipoprotein B-containing lipoproteins in plasma. Annals of Clinical Biochemistry International Journal of Laboratory Medicine. 42(4). 298–300.
13.
Osawa, Haruhiko, Kazuya Yamada, Hiroshi Onuma, et al.. (2004). The G/G Genotype of a Resistin Single-Nucleotide Polymorphism at −420 Increases Type 2 Diabetes Mellitus Susceptibility by Inducing Promoter Activity through Specific Binding of Sp1/3. The American Journal of Human Genetics. 75(4). 678–686.
14.
Osawa, Haruhiko, Toshiyuki Niiya, Hiroshi Onuma, et al.. (2003). Systematic search for single nucleotide polymorphisms in the 5′ flanking region of the human phosphodiesterase 3B gene: absence of evidence for major effects of identified polymorphisms on susceptibility to Japanese type 2 diabetes. Molecular Genetics and Metabolism. 79(1). 43–51.
15.
Ogura, Takahiro, Haruhiko Osawa, Yan Tang, et al.. (2003). Reduction of phosphodiesterase 3B gene expression in peroxisome proliferator‐activated receptor γ (+/−) mice independent of adipocyte size. FEBS Letters. 542(1-3). 65–68.
16.
Ochi, Masaaki, Haruhiko Osawa, Hiroshi Onuma, et al.. (2003). The absence of evidence for major effects of the frequent SNP +299G>A in the resistin gene on susceptibility to insulin resistance syndrome associated with Japanese type 2 diabetes. Diabetes Research and Clinical Practice. 61(3). 191–198.
17.
Hasegawa, Masaaki, Yan Tang, Haruhiko Osawa, et al.. (2002). Differential regulation of gene expression and insulin-induced activation of phosphodiesterase 3B in adipocytes of lean insulin-resistant IRS-1 (−/−) mice. Diabetes Research and Clinical Practice. 58(2). 79–85.
18.
Tang, Yan, Haruhiko Osawa, Hiroshi Onuma, et al.. (2001). Phosphodiesterase 3B gene expression is enhanced in the liver but reduced in the adipose tissue of obese insulin resistant db/db mouse. Diabetes Research and Clinical Practice. 54(3). 145–155.
19.
Osawa, Haruhiko, Hiroshi Onuma, Akiko Murakami, et al.. (2001). Systematic Search for Single Nucleotide Polymorphisms in the Insulin Gene: Evidence for a High Frequency of −23T→A in Japanese Subjects. Biochemical and Biophysical Research Communications. 286(3). 451–455.
20.
Saheki, Shuichi, Masaaki Ochi, Takahiko Horiuchi, et al.. (1992). Intestinal type alkaline phosphatase hyperphosphatasemia associated with liver cirrhosis. Clinica Chimica Acta. 210(1-2). 63–73.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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