Mary Y. K. Lee

851 total citations
16 papers, 694 citations indexed

About

Mary Y. K. Lee is a scholar working on Physiology, Pathology and Forensic Medicine and Genetics. According to data from OpenAlex, Mary Y. K. Lee has authored 16 papers receiving a total of 694 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Physiology, 3 papers in Pathology and Forensic Medicine and 3 papers in Genetics. Recurrent topics in Mary Y. K. Lee's work include Nitric Oxide and Endothelin Effects (4 papers), Cardiovascular, Neuropeptides, and Oxidative Stress Research (2 papers) and Sirtuins and Resveratrol in Medicine (2 papers). Mary Y. K. Lee is often cited by papers focused on Nitric Oxide and Endothelin Effects (4 papers), Cardiovascular, Neuropeptides, and Oxidative Stress Research (2 papers) and Sirtuins and Resveratrol in Medicine (2 papers). Mary Y. K. Lee collaborates with scholars based in Hong Kong, China and United States. Mary Y. K. Lee's co-authors include Paul M. Vanhoutte, Yu Wang, Ricky Y.K. Man, Yan Liang, Cheng Xu, Ling Liu, Aimin Xu, Huiying Li, Yang Xiao and Zhiguang Zhou and has published in prestigious journals such as Circulation, Circulation Research and Journal of Applied Physiology.

In The Last Decade

Mary Y. K. Lee

16 papers receiving 688 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mary Y. K. Lee Hong Kong 11 253 237 166 136 80 16 694
Ruifeng Yang China 10 317 1.3× 174 0.7× 212 1.3× 114 0.8× 76 0.9× 20 669
Yuchen Ge China 8 340 1.3× 210 0.9× 178 1.1× 152 1.1× 49 0.6× 12 781
Nicola Schäfer Switzerland 7 181 0.7× 163 0.7× 163 1.0× 119 0.9× 97 1.2× 7 681
Keita Inanaga Japan 9 187 0.7× 162 0.7× 126 0.8× 61 0.4× 60 0.8× 10 532
Francesca Saba Italy 18 243 1.0× 164 0.7× 97 0.6× 466 3.4× 54 0.7× 31 970
Kazuo Fujisawa Japan 10 474 1.9× 300 1.3× 55 0.3× 153 1.1× 73 0.9× 12 925
Tongshuai Chen China 12 237 0.9× 158 0.7× 222 1.3× 174 1.3× 42 0.5× 32 683
Jin A. Shin South Korea 16 258 1.0× 99 0.4× 105 0.6× 80 0.6× 60 0.8× 27 710
Audrey Ayer France 14 298 1.2× 188 0.8× 99 0.6× 79 0.6× 28 0.3× 23 602
Katharina Pfeil Germany 8 326 1.3× 141 0.6× 159 1.0× 125 0.9× 30 0.4× 14 656

Countries citing papers authored by Mary Y. K. Lee

Since Specialization
Citations

This map shows the geographic impact of Mary Y. K. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary Y. K. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary Y. K. Lee more than expected).

Fields of papers citing papers by Mary Y. K. Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary Y. K. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary Y. K. Lee. The network helps show where Mary Y. K. Lee may publish in the future.

Co-authorship network of co-authors of Mary Y. K. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Mary Y. K. Lee. A scholar is included among the top collaborators of Mary Y. K. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mary Y. K. Lee. Mary Y. K. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Loong, Herbert H., et al.. (2020). Cost-effectiveness analysis of pembrolizumab compared to standard of care as first line treatment for patients with advanced melanoma in Hong Kong. Cost Effectiveness and Resource Allocation. 18(1). 2–2. 8 indexed citations
2.
Yuen, Man‐Fung, Wai‐Kay Seto, Lung‐Yi Mak, et al.. (2020). Cost–Utility of All-Oral Direct-Acting Antiviral Regimens for the Treatment of Genotype 1 Chronic Hepatitis C Virus-Infected Patients in Hong Kong. Digestive Diseases and Sciences. 66(4). 1315–1326. 6 indexed citations
3.
Loong, Herbert H., Carlos King Ho Wong, Praveen Dhankhar, et al.. (2019). Cost Effectiveness of PD-L1-Based Test-and-Treat Strategy with Pembrolizumab as the First-Line Treatment for Metastatic NSCLC in Hong Kong. PharmacoEconomics - Open. 4(2). 235–247. 21 indexed citations
4.
Wang, Yan, Jcw Mak, Mary Y. K. Lee, Aimin Xu, & Msm Ip. (2018). Low‐Frequency Intermittent Hypoxia Promotes Subcutaneous Adipogenic Differentiation. Oxidative Medicine and Cellular Longevity. 2018(1). 4501757–4501757. 8 indexed citations
5.
Lee, Mary Y. K., et al.. (2018). Low but not high frequency of intermittent hypoxia suppresses endothelium-dependent, oxidative stress-mediated contractions in carotid arteries of obese mice. Journal of Applied Physiology. 125(5). 1384–1395. 7 indexed citations
6.
Lee, Mary Y. K., Yan Wang, Jcw Mak, & Msm Ip. (2016). Intermittent hypoxia induces NF-κB-dependent endothelial activation via adipocyte-derived mediators. American Journal of Physiology-Cell Physiology. 310(6). C446–C455. 34 indexed citations
7.
8.
Bai, Bo, Yan Liang, Cheng Xu, et al.. (2012). Cyclin-Dependent Kinase 5–Mediated Hyperphosphorylation of Sirtuin-1 Contributes to the Development of Endothelial Senescence and Atherosclerosis. Circulation. 126(6). 729–740. 85 indexed citations
9.
Lee, Mary Y. K., Yu Cai, Yu Wang, et al.. (2012). Differential genomic changes caused by cholesterol- and PUFA-rich diets in regenerated porcine coronary endothelial cells. Physiological Genomics. 44(10). 551–561. 10 indexed citations
10.
Liu, Ling, Mary Y. K. Lee, Cheng Xu, et al.. (2010). SIRT1 Promotes Proliferation and Prevents Senescence Through Targeting LKB1 in Primary Porcine Aortic Endothelial Cells. Circulation Research. 106(8). 1384–1393. 248 indexed citations
11.
Lee, Mary Y. K., Huiying Li, Yang Xiao, et al.. (2010). Chronic administration of BMS309403 improves endothelial function in apolipoprotein E‐deficient mice and in cultured human endothelial cells. British Journal of Pharmacology. 162(7). 1564–1576. 80 indexed citations
12.
Lee, Mary Y. K., Yu Wang, & Paul M. Vanhoutte. (2009). Senescence of Cultured Porcine Coronary Arterial Endothelial Cells Is Associated with Accelerated Oxidative Stress and Activation of NFĸB. Journal of Vascular Research. 47(4). 287–298. 67 indexed citations
13.
Lee, Mary Y. K., Grith Lykke Sørensen, Uffe Holmskov, & Paul M. Vanhoutte. (2008). The presence and activity of SP-D in porcine coronary endothelial cells depend on Akt/PI3K, Erk and nitric oxide and decrease after multiple passaging. Molecular Immunology. 46(6). 1050–1057. 15 indexed citations
14.
Lee, Mary Y. K., Hung‐Fat Tse, Chung‐Wah Siu, et al.. (2007). Genomic Changes in Regenerated Porcine Coronary Arterial Endothelial Cells. Arteriosclerosis Thrombosis and Vascular Biology. 27(11). 2443–2449. 51 indexed citations
15.
Lee, Mary Y. K., Susan W.S. Leung, Paul M. Vanhoutte, & Ricky Y.K. Man. (2004). Genistein reduces agonist-induced contractions of porcine coronary arterial smooth muscle in a cyclic AMP-dependent manner. European Journal of Pharmacology. 503(1-3). 165–172. 16 indexed citations
16.
Lee, Mary Y. K. & Ricky Y.K. Man. (2003). The phytoestrogen genistein enhances endothelium-independent relaxation in the porcine coronary artery. European Journal of Pharmacology. 481(2-3). 227–232. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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