Mary E. Brandes

855 total citations
11 papers, 750 citations indexed

About

Mary E. Brandes is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Immunology. According to data from OpenAlex, Mary E. Brandes has authored 11 papers receiving a total of 750 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Pulmonary and Respiratory Medicine and 3 papers in Immunology. Recurrent topics in Mary E. Brandes's work include TGF-β signaling in diseases (3 papers), S100 Proteins and Annexins (3 papers) and Bone Metabolism and Diseases (2 papers). Mary E. Brandes is often cited by papers focused on TGF-β signaling in diseases (3 papers), S100 Proteins and Annexins (3 papers) and Bone Metabolism and Diseases (2 papers). Mary E. Brandes collaborates with scholars based in United States. Mary E. Brandes's co-authors include Sharon M. Wahl, J B Allen, Yasushi Ogawa, Carl L. Manthey, Pin‐Yu Perera, S N Vogel, Jacob N. Finkelstein, Kiyoshi Ohura, Lalage M. Wakefield and Maria Cristina Morganti-Kossmann and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The Journal of Immunology.

In The Last Decade

Mary E. Brandes

10 papers receiving 718 citations

Peers

Mary E. Brandes

MB

IMMUN

ONCOL

IA

CR

Bei-Chang Yang Taiwan

Frank Wempe Germany

Lisa Larson United States

Harukiyo Kawamura Japan

Laura Brogelli Italy

T Ikeuchi Japan

S. M. Krane United States

Catharine J. Andresen United States

Svante Stenman Finland

Christopher Benyon United Kingdom

Bei-Chang Yang Taiwan

Mary E. Brandes

230 ×0.7

MB

332 ×1.4

IMMUN

123 ×0.6

ONCOL

80 ×0.7

IA

69 ×0.7

CR

Citations per year, relative to Mary E. Brandes Mary E. Brandes (= 1×) peers Bei-Chang Yang

Countries citing papers authored by Mary E. Brandes

Since Specialization

Citations

This map shows the geographic impact of Mary E. Brandes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary E. Brandes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary E. Brandes more than expected).

Fields of papers citing papers by Mary E. Brandes

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary E. Brandes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary E. Brandes. The network helps show where Mary E. Brandes may publish in the future.

Co-authorship network of co-authors of Mary E. Brandes

This figure shows the co-authorship network connecting the top 25 collaborators of Mary E. Brandes. A scholar is included among the top collaborators of Mary E. Brandes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mary E. Brandes. Mary E. Brandes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Kossmann, Thomas, Carl L. Manthey, Mary E. Brandes, et al.. (1992). Kupffer Cells Express Type I TGF-βReceptors, Migrate to TGF-βand Participate in Streptococcal Cell Wall Induced Hepatic Granuloma Formation. Growth Factors. 7(1). 73–83. 13 indexed citations

2.

Morganti-Kossmann, Maria Cristina, Thomas Kossmann, Mary E. Brandes, Stephan E. Mergenhagen, & Sharon M. Wahl. (1992). Autocrine and paracrine regulation of astrocyte function by transforming growth factor-β. Journal of Neuroimmunology. 39(1-2). 163–173. 86 indexed citations

3.

Manthey, Carl L., Mary E. Brandes, Pin‐Yu Perera, & S N Vogel. (1992). Taxol increases steady-state levels of lipopolysaccharide-inducible genes and protein-tyrosine phosphorylation in murine macrophages. The Journal of Immunology. 149(7). 2459–2465. 163 indexed citations

4.

Brandes, Mary E., J B Allen, Yasushi Ogawa, & Sharon M. Wahl. (1991). Transforming growth factor beta 1 suppresses acute and chronic arthritis in experimental animals.. Journal of Clinical Investigation. 87(3). 1108–1113. 190 indexed citations

5.

Wong, H.L.S., et al.. (1991). IL-4 antagonizes induction of Fc gamma RIII (CD16) expression by transforming growth factor-beta on human monocytes. The Journal of Immunology. 147(6). 1843–1848. 31 indexed citations

6.

Brandes, Mary E., Lalage M. Wakefield, & Sharon M. Wahl. (1991). Modulation of monocyte type I transforming growth factor-beta receptors by inflammatory stimuli.. Journal of Biological Chemistry. 266(29). 19697–19703. 62 indexed citations

7.

Brandes, Mary E., et al.. (1991). Type I transforming growth factor-beta receptors on neutrophils mediate chemotaxis to transforming growth factor-beta. The Journal of Immunology. 147(5). 1600–1606. 132 indexed citations

8.

Brandes, Mary E. & Jacob N. Finkelstein. (1990). The production of alveolar macrophage-derived growth-regulating proteins in response to lung injury. Toxicology Letters. 54(1). 3–22. 21 indexed citations

9.

Brandes, Mary E. & Jacob N. Finkelstein. (1989). Induction of the Stress Response by Isolation of Rabbit Type II Pneumocytes. Experimental Lung Research. 15(1). 93–111. 13 indexed citations

10.

Brandes, Mary E. & Jacob N. Finkelstein. (1989). Stimulated Rabbit Alveolar Macrophages Secrete a Growth Factor for Type II Pneumocytes. American Journal of Respiratory Cell and Molecular Biology. 1(2). 101–109. 38 indexed citations

11.

Hogben, C. Adrian M., et al.. (1972). The electrophysiological response of the dogfish gastric mucosa to extracellular potassium. Comparative Biochemistry and Physiology Part A Physiology. 42(1). 153–168. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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