Martine E. Lomax

1.9k total citations · 1 hit paper
23 papers, 1.6k citations indexed

About

Martine E. Lomax is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Martine E. Lomax has authored 23 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 12 papers in Oncology and 9 papers in Cancer Research. Recurrent topics in Martine E. Lomax's work include DNA Repair Mechanisms (16 papers), Carcinogens and Genotoxicity Assessment (9 papers) and Cancer-related Molecular Pathways (8 papers). Martine E. Lomax is often cited by papers focused on DNA Repair Mechanisms (16 papers), Carcinogens and Genotoxicity Assessment (9 papers) and Cancer-related Molecular Pathways (8 papers). Martine E. Lomax collaborates with scholars based in United Kingdom, United States and France. Martine E. Lomax's co-authors include Peter O’Neill, P. O’Neill, Lisa K. Folkes, Siobhan Cunniffe, Laura J. Eccles, D M Barnes, Steven M. Picksley, RS Camplejohn, Ted R. Hupp and Pamela Reynolds and has published in prestigious journals such as Nucleic Acids Research, Biochemistry and Oncogene.

In The Last Decade

Martine E. Lomax

23 papers receiving 1.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Biological Consequences of Radiation-induced DNA Damage: Relevance to Radiotherapy

2013 538 citations Martine E. Lomax, Lisa K. Folkes et al. Clinical Oncology profile →

Peers

Countries citing papers authored by Martine E. Lomax

Since Specialization

Citations

This map shows the geographic impact of Martine E. Lomax's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martine E. Lomax with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martine E. Lomax more than expected).

Fields of papers citing papers by Martine E. Lomax

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martine E. Lomax. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martine E. Lomax. The network helps show where Martine E. Lomax may publish in the future.

Co-authorship network of co-authors of Martine E. Lomax

This figure shows the co-authorship network connecting the top 25 collaborators of Martine E. Lomax. A scholar is included among the top collaborators of Martine E. Lomax based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martine E. Lomax. Martine E. Lomax is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

#	Work	Indexed citations
1	Antigen Selection for Enhanced Affinity T-Cell Receptor–Based Cancer Therapies 2016 ·SLAS DISCOVERY ·Emma Hickman, Martine E. Lomax, Bent K. Jakobsen	12
2	Efficient cleavage of single and clustered AP site lesions within mono-nucleosome templates by CHO-K1 nuclear extract contrasts with retardation of incision by purified APE1 2015 ·DNA repair ·Laura J. Eccles, Hervé Menoni, Dimitar Angelov, Martine E. Lomax, Peter O’Neill	19
3	Disruption of PARP1 function inhibits base excision repair of a sub-set of DNA lesions 2015 ·Nucleic Acids Research ·Pamela Reynolds, (unknown), Martine E. Lomax, Peter O’Neill	55
4	Increased mutability and decreased repairability of a three-lesion clustered DNA-damaged site comprised of an AP site and bi-stranded 8-oxoG lesions 2014 ·International Journal of Radiation Biology ·Siobhan Cunniffe, Alexandra K. Walker, (unknown), Peter O’Neill, Martine E. Lomax	17
5	Reduced repair capacity of a DNA clustered damage site comprised of 8-oxo-7,8-dihydro-2′-deoxyguanosine and 2-deoxyribonolactone results in an increased mutagenic potential of these lesions 2014 ·Mutation research. Fundamental and molecular mechanisms of mutagenesis ·Siobhan Cunniffe, Peter O’Neill, Marc M. Greenberg, Martine E. Lomax	23
6	Biological Consequences of Radiation-induced DNA Damage: Relevance to Radiotherapy breakdown → 2013 ·Clinical Oncology ·Martine E. Lomax, Lisa K. Folkes, P. O’Neill	538
7	Delayed repair of radiation induced clustered DNA damage: Friend or foe? 2010 ·Mutation research. Fundamental and molecular mechanisms of mutagenesis ·Laura J. Eccles, Peter O’Neill, Martine E. Lomax	195
8	Hierarchy of lesion processing governs the repair, double-strand break formation and mutability of three-lesion clustered DNA damage 2009 ·Nucleic Acids Research ·Laura J. Eccles, Martine E. Lomax, Peter O’Neill	58
9	Processing of thymine glycol in a clustered DNA damage site: mutagenic or cytotoxic 2009 ·Nucleic Acids Research ·Sophie Bellon, Naoya Shikazono, Siobhan Cunniffe, Martine E. Lomax, Peter O’Neill	49
10	5,6-Dihydrothymine Impairs the Base Excision Repair Pathway of a Closely Opposed AP Site or Single-Strand Break 2009 ·Radiation Research ·(unknown), Siobhan Cunniffe, Peter O’Neill, Martine E. Lomax	11
11	Interplay of two major repair pathways in the processing of complex double-strand DNA breaks 2008 ·DNA repair ·(unknown), P. E. S. Palmer, Zoitsa Maniou, Martine E. Lomax, Peter O’Neill	54
12	An AP site can protect against the mutagenic potential of 8-oxoG when present within a tandem clustered site in E. coli 2007 ·DNA repair ·Siobhan Cunniffe, Martine E. Lomax, Peter O’Neill	36
13	Base excision repair processing of abasic site/single-strand break lesions within clustered damage sites associated with XRCC1 deficiency 2007 ·Nucleic Acids Research ·(unknown), Martine E. Lomax, Peter O’Neill	41
14	8-OxoA Inhibits the Incision of an AP Site by the DNA Glycosylases Fpg, Nth and the AP Endonuclease HAP1 2005 ·Radiation Research ·Martine E. Lomax, Henrik Salje, Siobhan Cunniffe, Peter O’Neill	26
15	8-OxoG retards the activity of the ligase III/XRCC1 complex during the repair of a single-strand break, when present within a clustered DNA damage site 2003 ·DNA repair ·Martine E. Lomax, Siobhan Cunniffe, Peter O’Neill	71
16	Chemical Aspects of Clustered DNA Damage Induction by Ionising Radiation 2002 ·Radiation Protection Dosimetry ·Martine E. Lomax, (unknown), P. O’Neill	67
17	Polyoma virus disrupts ARF signaling to p53 2001 ·Oncogene ·Martine E. Lomax, Mike Fried	24
18	The value of rapid functional assays of germline p53 status in LFS and LFL families 2000 ·British Journal of Cancer ·Richard S. Camplejohn, Nayanta Sodha, R Gilchrist, Martine E. Lomax, (unknown), Cristina Miner, (unknown), D M Barnes, Rosalind A. Eeles	13
19	Characterization of p53 oligomerization domain mutations isolated from Li–Fraumeni and Li–Fraumeni like family members 1998 ·Oncogene ·Martine E. Lomax, D M Barnes, Ted R. Hupp, Steven M. Picksley, RS Camplejohn	88
20	Two functional assays employed to detect an unusual mutation in the oligomerisation domain of p53 in a Li-Fraumeni like family 1997 ·Oncogene ·Martine E. Lomax, D M Barnes, R Gilchrist, Steven M. Picksley, J M Varley, RS Camplejohn	54

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact