Martin R. Stämpfli

7.6k total citations · 1 hit paper
135 papers, 5.5k citations indexed

About

Martin R. Stämpfli is a scholar working on Immunology, Physiology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Martin R. Stämpfli has authored 135 papers receiving a total of 5.5k indexed citations (citations by other indexed papers that have themselves been cited), including 60 papers in Immunology, 59 papers in Physiology and 40 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Martin R. Stämpfli's work include Asthma and respiratory diseases (54 papers), Chronic Obstructive Pulmonary Disease (COPD) Research (27 papers) and Pediatric health and respiratory diseases (21 papers). Martin R. Stämpfli is often cited by papers focused on Asthma and respiratory diseases (54 papers), Chronic Obstructive Pulmonary Disease (COPD) Research (27 papers) and Pediatric health and respiratory diseases (21 papers). Martin R. Stämpfli collaborates with scholars based in Canada, United States and Sweden. Martin R. Stämpfli's co-authors include Gary P. Anderson, Manel Jordana, Clinton S. Robbins, Carla M. T. Bauer, Warren G. Foster, Beata Gajewska, Mathieu C. Morissette, Jake K. Nikota, Caleb C. J. Zavitz and Gordon J. Gaschler and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature reviews. Immunology.

In The Last Decade

Martin R. Stämpfli

135 papers receiving 5.5k citations

Hit Papers

How cigarette smoke skews immune responses to promote inf... 2009 2026 2014 2020 2009 100 200 300 400 500
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. How cigarette smoke skews immune responses to promote infection, lung disease and cancer
    2009 501 citations Martin R. Stämpfli et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Martin R. Stämpfli Canada 42 1.9k 1.8k 1.8k 1.2k 737 135 5.5k
Hong Wei Chu United States 43 2.0k 1.1× 2.8k 1.6× 3.0k 1.7× 1.3k 1.1× 1.2k 1.6× 167 6.9k
Sandra Hodge Australia 39 2.0k 1.0× 2.7k 1.5× 2.2k 1.2× 1.2k 1.1× 767 1.0× 139 5.8k
Joërg Mattes Australia 41 2.1k 1.1× 1.3k 0.7× 2.2k 1.2× 1.4k 1.2× 607 0.8× 154 5.6k
Patricia W. Finn United States 39 2.0k 1.0× 897 0.5× 1.5k 0.8× 1.1k 1.0× 440 0.6× 146 4.9k
Michael R. Edwards United Kingdom 44 2.9k 1.5× 1.9k 1.0× 2.9k 1.6× 1.3k 1.2× 2.1k 2.9× 143 7.5k
Ian Sabroe United Kingdom 50 3.5k 1.8× 1.8k 1.0× 1.4k 0.8× 1.5k 1.3× 898 1.2× 158 7.5k
Katherine J. Baines Australia 40 1.9k 1.0× 2.1k 1.1× 3.0k 1.7× 1.2k 1.1× 371 0.5× 110 5.0k
Sanjiv Sur United States 42 1.8k 1.0× 1.2k 0.6× 2.5k 1.4× 1.2k 1.0× 290 0.4× 99 5.0k
Nobuyuki Hizawa Japan 35 1.3k 0.7× 1.8k 1.0× 1.5k 0.8× 1.1k 1.0× 516 0.7× 247 5.0k
Charles McSharry United Kingdom 46 2.3k 1.2× 2.3k 1.3× 2.4k 1.4× 1.1k 0.9× 486 0.7× 128 7.2k

Countries citing papers authored by Martin R. Stämpfli

Since Specialization
Citations

This map shows the geographic impact of Martin R. Stämpfli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin R. Stämpfli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin R. Stämpfli more than expected).

Fields of papers citing papers by Martin R. Stämpfli

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin R. Stämpfli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin R. Stämpfli. The network helps show where Martin R. Stämpfli may publish in the future.

Co-authorship network of co-authors of Martin R. Stämpfli

This figure shows the co-authorship network connecting the top 25 collaborators of Martin R. Stämpfli. A scholar is included among the top collaborators of Martin R. Stämpfli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin R. Stämpfli. Martin R. Stämpfli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Milad, Nadia, Peiyao Wang, Sam Afkhami, et al.. (2023). Cannabis smoke suppresses antiviral immune responses to influenza A in mice. ERJ Open Research. 9(6). 219–2023. 4 indexed citations
2.
Wang, Zhang, Yuqiong Yang, Zhengzheng Yan, et al.. (2020). Multi-omic meta-analysis identifies functional signatures of airway microbiome in chronic obstructive pulmonary disease. The ISME Journal. 14(11). 2748–2765. 52 indexed citations
3.
Roos, Abraham B., Sanjay Sethi, Jake K. Nikota, et al.. (2015). IL-17A and the Promotion of Neutrophilia in Acute Exacerbation of Chronic Obstructive Pulmonary Disease. American Journal of Respiratory and Critical Care Medicine. 192(4). 428–437. 84 indexed citations
4.
Roos, Abraham B., Caroline Sandén, Michiko Mori, et al.. (2015). IL-17A Is Elevated in End-Stage Chronic Obstructive Pulmonary Disease and Contributes to Cigarette Smoke–induced Lymphoid Neogenesis. American Journal of Respiratory and Critical Care Medicine. 191(11). 1232–1241. 96 indexed citations
5.
Roos, Abraham B., Leif Bjermer, Martin R. Stämpfli, & Jonas S. Erjefält. (2014). Elevated IL-17A in very severe COPD is localized to mast cells and correlates with lung function decline. European Respiratory Journal. 44(Suppl 58). 1722–1722. 1 indexed citations
6.
Bauer, Carla M. T., Donavan T. Cheng, John Allard, et al.. (2013). Precision cut lung slices: A novel method for examining mechanisms underlying respiratory diseases. European Respiratory Journal. 42(Suppl 57). P669–P669. 1 indexed citations
7.
Didon, Lukas, Jenny L. Barton, Abraham B. Roos, et al.. (2011). Lung Epithelial CCAAT/Enhancer-binding Protein-β Is Necessary for the Integrity of Inflammatory Responses to Cigarette Smoke. American Journal of Respiratory and Critical Care Medicine. 184(2). 233–242. 30 indexed citations
8.
Nikota, Jake K., Fernando Botelho, Carla M. T. Bauer, et al.. (2011). Differential expression and function of breast regression protein 39 (BRP-39) in murine models of subacute cigarette smoke exposure and allergic airway inflammation. Respiratory Research. 12(1). 39–39. 22 indexed citations
9.
Llop‐Guevara, Alba, Jake K. Nikota, Carla M. T. Bauer, et al.. (2011). Cigarette Smoke Differentially Affects Eosinophilia and Remodeling in a Model of House Dust Mite Asthma. American Journal of Respiratory Cell and Molecular Biology. 45(4). 753–760. 36 indexed citations
10.
Zavitz, Caleb C. J., Carla M. T. Bauer, Gordon J. Gaschler, et al.. (2010). Dysregulated Macrophage-Inflammatory Protein-2 Expression Drives Illness in Bacterial Superinfection of Influenza. The Journal of Immunology. 184(4). 2001–2013. 19 indexed citations
11.
Gaschler, Gordon J., Marko Škrtić, Caleb C. J. Zavitz, et al.. (2009). Bacteria Challenge in Smoke-exposed Mice Exacerbates Inflammation and Skews the Inflammatory Profile. American Journal of Respiratory and Critical Care Medicine. 179(8). 666–675. 87 indexed citations
12.
Bauer, Carla M. T., Stephanie J. DeWitte‐Orr, Caleb C. J. Zavitz, et al.. (2008). Cigarette Smoke Suppresses Type I Interferon-Mediated Antiviral Immunity in Lung Fibroblast and Epithelial Cells. Journal of Interferon & Cytokine Research. 28(3). 167–179. 47 indexed citations
13.
Robbins, Clinton S., Carla M. T. Bauer, Gordon J. Gaschler, et al.. (2006). Cigarette Smoke Impacts Immune Inflammatory Responses to Influenza in Mice. American Journal of Respiratory and Critical Care Medicine. 174(12). 1342–1351. 88 indexed citations
14.
Álvarez, David, Filip K. Świrski, Ramzi Fattouh, et al.. (2006). Inhalation Tolerance Is Induced Selectively in Thoracic Lymph Nodes but Executed Pervasively at Distant Mucosal and Nonmucosal Tissues. The Journal of Immunology. 176(4). 2568–2580. 16 indexed citations
15.
Álvarez, David, Ramzi Fattouh, Jiangfeng Sun, et al.. (2005). Cutaneous Antigen Priming via Gene Gun Leads to Skin-Selective Th2 Immune-Inflammatory Responses. The Journal of Immunology. 174(3). 1664–1674. 29 indexed citations
16.
Gajewska, Beata, Agostino Tafuri, Filip K. Świrski, et al.. (2005). B7RP-1 Is Not Required for the Generation of Th2 Responses in a Model of Allergic Airway Inflammation but Is Essential for the Induction of Inhalation Tolerance. The Journal of Immunology. 174(5). 3000–3005. 19 indexed citations
17.
Drannik, Anna G., Mahmoud A. Pouladi, Clinton S. Robbins, et al.. (2004). Impact of Cigarette Smoke on Clearance and Inflammation after Pseudomonas aeruginosa Infection. American Journal of Respiratory and Critical Care Medicine. 170(11). 1164–1171. 128 indexed citations
18.
Bonniaud, Philippe, Martin Kolb, Jennifer Robertson, et al.. (2004). Smad3 Null Mice Develop Airspace Enlargement and Are Resistant to TGF-β-Mediated Pulmonary Fibrosis. The Journal of Immunology. 173(3). 2099–2108. 313 indexed citations
19.
Świrski, Filip K., Dusan Sajic, Clinton S. Robbins, et al.. (2002). Chronic Exposure to Innocuous Antigen in Sensitized Mice Leads to Suppressed Airway Eosinophilia That Is Reversed by Granulocyte Macrophage Colony-Stimulating Factor. The Journal of Immunology. 169(7). 3499–3506. 72 indexed citations
20.
Wiley, Ryan E., Kay Palmer, Beata Gajewska, et al.. (2001). Expression of the Th1 Chemokine IFN-γ-Inducible Protein 10 in the Airway Alters Mucosal Allergic Sensitization in Mice. The Journal of Immunology. 166(4). 2750–2759. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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