Martin Piskáček

768 total citations
26 papers, 526 citations indexed

About

Martin Piskáček is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Martin Piskáček has authored 26 papers receiving a total of 526 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 9 papers in Oncology and 8 papers in Immunology. Recurrent topics in Martin Piskáček's work include Genomics and Chromatin Dynamics (8 papers), Immunotherapy and Immune Responses (4 papers) and Peroxisome Proliferator-Activated Receptors (4 papers). Martin Piskáček is often cited by papers focused on Genomics and Chromatin Dynamics (8 papers), Immunotherapy and Immune Responses (4 papers) and Peroxisome Proliferator-Activated Receptors (4 papers). Martin Piskáček collaborates with scholars based in Czechia, Austria and United States. Martin Piskáček's co-authors include Andrea Knight, Martin Grábner, Maria Némethová, Pavel Kovarik, Martin Gregor, Helmut Ruis, Barbara Hamilton, Rudolf J. Schweyen, Martina Řezáčová and Gábor Zsurka and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Biochemical Journal.

In The Last Decade

Martin Piskáček

26 papers receiving 522 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Martin Piskáček Czechia 11 363 93 80 63 53 26 526
Salvatore Valvo United Kingdom 9 251 0.7× 72 0.8× 209 2.6× 29 0.5× 22 0.4× 15 546
Lingjun Meng China 11 424 1.2× 43 0.5× 122 1.5× 45 0.7× 32 0.6× 16 541
Alan A. Finegold United States 8 343 0.9× 40 0.4× 69 0.9× 66 1.0× 48 0.9× 9 549
Isabelle Tratner France 12 342 0.9× 75 0.8× 30 0.4× 31 0.5× 88 1.7× 15 468
Jeongbin Yim South Korea 9 376 1.0× 72 0.8× 91 1.1× 25 0.4× 84 1.6× 14 558
Clark C. Fjeld United States 6 455 1.3× 63 0.7× 35 0.4× 24 0.4× 104 2.0× 6 575
C Billardon France 14 243 0.7× 58 0.6× 108 1.4× 40 0.6× 35 0.7× 26 524
Ramona Weber Germany 13 547 1.5× 21 0.2× 61 0.8× 32 0.5× 32 0.6× 17 621
Neil Winegarden Canada 9 411 1.1× 20 0.2× 45 0.6× 40 0.6× 44 0.8× 13 585
Ram Bandyopadhyay United States 9 310 0.9× 50 0.5× 76 0.9× 38 0.6× 27 0.5× 16 504

Countries citing papers authored by Martin Piskáček

Since Specialization
Citations

This map shows the geographic impact of Martin Piskáček's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Piskáček with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Piskáček more than expected).

Fields of papers citing papers by Martin Piskáček

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Piskáček. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Piskáček. The network helps show where Martin Piskáček may publish in the future.

Co-authorship network of co-authors of Martin Piskáček

This figure shows the co-authorship network connecting the top 25 collaborators of Martin Piskáček. A scholar is included among the top collaborators of Martin Piskáček based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Piskáček. Martin Piskáček is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Knight, Andrea, et al.. (2024). Myc 9aaTAD activation domain binds to mediator of transcription with superior high affinity. Molecular Medicine. 30(1). 211–211. 1 indexed citations
2.
Houser, Josef, et al.. (2023). The NFkB activation domain is 14-amino-acid-long variant of the 9aaTAD. Biochemical Journal. 480(5). 297–306. 3 indexed citations
3.
Knight, Andrea, Martin Piskáček, Michal Jurajda, et al.. (2022). Expansions of tumor-reactive Vdelta1 gamma-delta T cells in newly diagnosed patients with chronic myeloid leukemia. Cancer Immunology Immunotherapy. 72(5). 1209–1224. 3 indexed citations
4.
Knight, Andrea & Martin Piskáček. (2022). Cryptic inhibitory regions nearby activation domains. Biochimie. 200. 19–26. 3 indexed citations
5.
Piskáček, Martin, Marie Tomandlová, Martin Smrčka, et al.. (2022). CNSC-13. TUMOR INFILTRATION OF GAMMA-DELTA T CELLS IN GLIOBLASTOMA. Neuro-Oncology. 24(Supplement_7). vii24–vii24. 1 indexed citations
6.
Hříbková, Hana, Jana Hořínková, Ľubica Lacinová, et al.. (2022). Clozapine Reverses Dysfunction of Glutamatergic Neurons Derived From Clozapine-Responsive Schizophrenia Patients. Frontiers in Cellular Neuroscience. 16. 830757–830757. 20 indexed citations
7.
Hritz, Jozef, et al.. (2021). Universal two‐point interaction of mediator KIX with 9aaTAD activation domains. Journal of Cellular Biochemistry. 122(10). 1544–1555. 3 indexed citations
8.
Knight, Andrea, Lucie Říhová, Miroslav Penka, et al.. (2021). Plasmacytoid Dendritic Cells in Patients with MGUS and Multiple Myeloma. Journal of Clinical Medicine. 10(16). 3717–3717. 9 indexed citations
9.
Hudeček, Robert, et al.. (2021). Blocking of EphA2 on Endometrial Tumor Cells Reduces Susceptibility to Vδ1 Gamma-Delta T-Cell-Mediated Killing. Frontiers in Immunology. 12. 752646–752646. 10 indexed citations
10.
Piskáček, Martin, et al.. (2019). The evolution of the 9aaTAD domain in Sp2 proteins: inactivation with valines and intron reservoirs. Cellular and Molecular Life Sciences. 77(9). 1793–1810. 9 indexed citations
11.
Piskáček, Martin, et al.. (2018). Nuclear hormone receptors: Ancient 9aaTAD and evolutionally gained NCoA activation pathways. The Journal of Steroid Biochemistry and Molecular Biology. 187. 118–123. 7 indexed citations
12.
Piskáček, Martin, et al.. (2017). The 9aaTAD Is Exclusive Activation Domain in Gal4. PLoS ONE. 12(1). e0169261–e0169261. 15 indexed citations
13.
Piskáček, Martin, et al.. (2016). The 9aaTAD Transactivation Domains: From Gal4 to p53. PLoS ONE. 11(9). e0162842–e0162842. 48 indexed citations
14.
Piskáček, Martin, Anna Vašků, Roman Hájek, & Andrea Knight. (2015). Shared structural features of the 9aaTAD family in complex with CBP. Molecular BioSystems. 11(3). 844–851. 17 indexed citations
15.
Raja, Karthick Raja Muthu, Lenka Kubiczková, Lucie Říhová, et al.. (2012). Functionally Suppressive CD8 T Regulatory Cells Are Increased in Patients with Multiple Myeloma: A Cause for Immune Impairment. PLoS ONE. 7(11). e49446–e49446. 38 indexed citations
16.
Piskáček, Martin, et al.. (2008). Conditional knockdown of hMRS2 results in loss of mitochondrial Mg + uptake and cell death. Journal of Cellular and Molecular Medicine. 13(4). 693–700. 64 indexed citations
17.
Gregor, Martin, et al.. (2007). Nine-amino-acid transactivation domain: Establishment and prediction utilities. Genomics. 89(6). 756–768. 111 indexed citations
18.
Piskáček, Martin, et al.. (2007). A novel 9-amino-acid transactivation domain in the C-terminal part of Sox18. Biochemical and Biophysical Research Communications. 360(2). 370–374. 29 indexed citations
19.
20.
Hamilton, Barbara, et al.. (1999). Functional Analysis of the Zn2Cys6Transcription Factors Oaf1p and Pip2p. Journal of Biological Chemistry. 274(32). 22208–22216. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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