Martin L. Phillips
- Molecular Biology top 5%
- Cell Biology top 2%
- Genetics top 5%
- Immunology top 10%
- Ecology top 5%
- Co-authors
- M.R. SawayaVerne N. SchumakerDuilio CascioDavid EisenbergShuishu WangMichael StrongEmil ReislerShiho Tanaka
- Topics
- Cellular Mechanics and Interactions (15 papers)Glycosylation and Glycoproteins Research (8 papers)Cardiomyopathy and Myosin Studies (8 papers)
- Partner nations
- United StatesCanadaIsrael
In The Last Decade
Martin L. Phillips
72 papers receiving 3.1k citations
Hit Papers
Peers
Comparison fields: 5 of 122
- Molecular Biology 1.9k
- Cell Biology 533
- Genetics 350
- Immunology 340
- Ecology 316
Countries citing papers authored by Martin L. Phillips
This map shows the geographic impact of Martin L. Phillips's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin L. Phillips with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin L. Phillips more than expected).
Fields of papers citing papers by Martin L. Phillips
This network shows the impact of papers produced by Martin L. Phillips. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin L. Phillips. The network helps show where Martin L. Phillips may publish in the future.
Co-authorship network of co-authors of Martin L. Phillips
This figure shows the co-authorship network connecting the top 25 collaborators of Martin L. Phillips. A scholar is included among the top collaborators of Martin L. Phillips based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin L. Phillips. Martin L. Phillips is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 22 | |
| 3 | 25 | |
| 4 | 68 | |
| 5 | 55 | |
| 6 | 86 | |
| 7 | 14 | |
| 8 | Toward the structural genomics of complexes: Crystal structure of a PE/PPE protein complex from Mycobacterium tuberculosisbreakdown → | 607 |
| 9 | 79 | |
| 10 | 98 | |
| 11 | 29 | |
| 12 | 59 | |
| 13 | 35 | |
| 14 | 44 | |
| 15 | 34 | |
| 16 | 5 | |
| 17 | 34 | |
| 18 | 113 | |
| 19 | 3 | |
| 20 | 13 |
About Martin L. Phillips
Martin L. Phillips is a scholar working on Cell Biology, Biophysics and Structural Biology, having authored 72 papers that have together received 3.2k indexed citations. Recurring topics across this work include Cellular Mechanics and Interactions (15 papers), Glycosylation and Glycoproteins Research (8 papers) and Cardiomyopathy and Myosin Studies (8 papers). The work is most often cited by research in Structural Biology (52 citations), Cell Biology (533 citations) and Molecular Biology (1.9k citations). Martin L. Phillips has collaborated with scholars based in United States, Canada and Israel. Frequent co-authors include M.R. Sawaya, Verne N. Schumaker, Duilio Cascio, David Eisenberg, Shuishu Wang, Michael Strong, Emil Reisler, Shiho Tanaka, Todd O. Yeates and Jon E. Chatterton. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.