Martin Beinborn

3.2k total citations
68 papers, 2.6k citations indexed

About

Martin Beinborn is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Martin Beinborn has authored 68 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Cellular and Molecular Neuroscience, 41 papers in Molecular Biology and 19 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Martin Beinborn's work include Neuropeptides and Animal Physiology (44 papers), Receptor Mechanisms and Signaling (34 papers) and Diabetes Treatment and Management (18 papers). Martin Beinborn is often cited by papers focused on Neuropeptides and Animal Physiology (44 papers), Receptor Mechanisms and Signaling (34 papers) and Diabetes Treatment and Management (18 papers). Martin Beinborn collaborates with scholars based in United States, Germany and Italy. Martin Beinborn's co-authors include Alan S. Kopin, Edward W. McBride, Lee F. Kolakowski, Ming Lu, Laurence J. Miller, Herbert Y. Lin, Yong Ren, Jean‐Philippe Fortin, Chun‐I Chen and Youngmee Lee and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of the American Chemical Society.

In The Last Decade

Martin Beinborn

68 papers receiving 2.5k citations

Peers

Martin Beinborn

CMN

SURGE

EDM

ONCOL

Edward W. McBride United States

Y. Peng Loh United States

André Vandermeers Belgium

Laurent Taupenot United States

Lidia Demchyshyn Canada

M. Marcinkiewicz Canada

Philippe Brabet France

Roberta Possenti Italy

Thomas M. Laz United States

Remko A. Bakker Netherlands

Edward W. McBride United States

Martin Beinborn

1.0k ×0.8

CMN

758 ×0.6

452 ×0.6

SURGE

223 ×0.5

EDM

206 ×0.6

ONCOL

Citations per year, relative to Martin Beinborn Martin Beinborn (= 1×) peers Edward W. McBride

Countries citing papers authored by Martin Beinborn

Since Specialization

Citations

This map shows the geographic impact of Martin Beinborn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Beinborn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Beinborn more than expected).

Fields of papers citing papers by Martin Beinborn

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Beinborn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Beinborn. The network helps show where Martin Beinborn may publish in the future.

Co-authorship network of co-authors of Martin Beinborn

This figure shows the co-authorship network connecting the top 25 collaborators of Martin Beinborn. A scholar is included among the top collaborators of Martin Beinborn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Beinborn. Martin Beinborn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Montanari, Vittorio, et al.. (2024). Prolonged Activation of the GLP-1 Receptor via Covalent Capture. ACS Chemical Biology. 19(7). 1453–1465. 1 indexed citations

Julian, Bina, et al.. (2016). Mutation-Induced Functional Alterations of CCR6. Journal of Pharmacology and Experimental Therapeutics. 360(1). 106–116. 11 indexed citations

Fortin, Jean‐Philippe, et al.. (2013). Membrane Tethered Bursicon Constructs as Heterodimeric Modulators of the Drosophila G Protein–Coupled Receptor Rickets. Molecular Pharmacology. 83(4). 814–821. 11 indexed citations

Doyle, Jamie R., Jacqueline M. Lane, Martin Beinborn, & Alan S. Kopin. (2012). Naturally occurring HCA1 missense mutations result in loss of function: potential impact on lipid deposition. Journal of Lipid Research. 54(3). 823–830. 6 indexed citations

Beinborn, Martin, et al.. (2010). TGF-β regulates T-cell neurokinin-1 receptor internalization and function. Proceedings of the National Academy of Sciences. 107(9). 4293–4298. 37 indexed citations

Fortin, Jean‐Philippe, Inga Peter, Steven E. Reís, et al.. (2010). The μ-Opioid Receptor Variant N190K Is Unresponsive to Peptide Agonists yet Can be Rescued by Small-Molecule Drugs. Molecular Pharmacology. 78(5). 837–845. 19 indexed citations

Fortin, Jean‐Philippe, et al.. (2009). Membrane-tethered ligands are effective probes for exploring class B1 G protein-coupled receptor function. Proceedings of the National Academy of Sciences. 106(19). 8049–8054. 46 indexed citations

Ahn, Jung‐Mo, Sun-Young Han, Eunice Murage, & Martin Beinborn. (2009). Rational Design of Peptidomimetics for Class B GPCRs: Potent Non-Peptide GLP-1 Receptor Agonists. Advances in experimental medicine and biology. 611. 125–126. 2 indexed citations

Fortin, Jean‐Philippe, et al.. (2009). Pharmacological Characterization of Human Incretin Receptor Missense Variants. Journal of Pharmacology and Experimental Therapeutics. 332(1). 274–280. 70 indexed citations

10.

Murage, Eunice, et al.. (2008). Search for α-helical propensity in the receptor-bound conformation of glucagon-like peptide-1. Bioorganic & Medicinal Chemistry. 16(23). 10106–10112. 29 indexed citations

11.

Ren, Yong, et al.. (2008). Pharmacological Analysis of Human D1 and D2 Dopamine Receptor Missense Variants. Journal of Molecular Neuroscience. 34(3). 211–223. 23 indexed citations

12.

Watanabe, Kazuhiro, Martin Beinborn, Shinya Nagamatsu, Hitoshi Ishida, & Shinichi Takahashi. (2005). Ménétrier's disease in a patient with Helicobacter pylori infection is linked to elevated glucagon-like peptide-2 activity. Scandinavian Journal of Gastroenterology. 40(4). 477–481. 9 indexed citations

13.

Beinborn, Martin, et al.. (2005). A human glucagon-like peptide-1 receptor polymorphism results in reduced agonist responsiveness. Regulatory Peptides. 130(1-2). 1–6. 79 indexed citations

14.

Kopin, Alan S., et al.. (1995). The Role of the Cholecystokinin-B/Gastrin Receptor Transmembrane Domains in Determining Affinity for Subtype-selective Ligands(∗). Journal of Biological Chemistry. 270(10). 5019–5023. 77 indexed citations

15.

Bernhard, Wolfgang, et al.. (1994). Phospholipid Synthesis in Isolated Porcine Gastric Mucous Cells. Pharmacology. 48(3). 176–186. 4 indexed citations

16.

Kopin, Alan S., et al.. (1994). The CCK‐B/Gastrin Receptor. Annals of the New York Academy of Sciences. 713(1). 67–78. 13 indexed citations

17.

Beinborn, Martin, et al.. (1993). A single amino acid of the cholecystokinin-B/gastrin receptor determines specificity for non-peptide antagonists. Nature. 362(6418). 348–350. 184 indexed citations

18.

Beinborn, Martin, et al.. (1993). Isolation, identification and quantitative evaluation of specific cell types from the mammalian gastric mucosa. Cell and Tissue Research. 274(2). 229–240. 20 indexed citations

19.

Krömer, W., Eli M. Baron, Martin Beinborn, R. Boer, & Manfrid Eltze. (1990). Characterization of the muscarine receptor type on paracrine cells activated by McN-A-343 in the mouse isolated stomach. Naunyn-Schmiedeberg s Archives of Pharmacology. 341(3). 165–70. 11 indexed citations

20.

Seidler, Ursula, Martin Beinborn, & K.‐Fr. Sewing. (1989). Inhibition of Acid Formation in Rabbit Parietal Cells by Prostaglandins Is Mediated by the Prostaglandin E2 Receptor. Gastroenterology. 96(2). 314–320. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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