Mark K. Saville

3.9k citations
33 papers · 3.2k · 1 hit paper · h-index 23

Impact in

  • Oncology top 1%
    • Cancer-related Molecular Pathways
    • Peptidase Inhibition and Analysis
    • Ubiquitin and proteasome pathways
    • RNA modifications and cancer
    • Epigenetics and DNA Methylation

Papers in

    • Ubiquitin and proteasome pathways 16
    • Epigenetics and DNA Methylation 6
    • Cancer-related gene regulation 4
    • Genomics and Chromatin Dynamics 3
    • Cancer-related Molecular Pathways 20

Mark K. Saville

33 papers receiving 3.2k citations

Mark K. Saville's Hit Papers

p53 isoforms can regulate p53 transcriptional activity 2005 · 631 citations
6310+7+14Years since publication200400600

Peers

Mark K. Saville
Comparison fields: 5 of 91
  • Oncology 1.9k
  • Molecular Biology 2.7k
  • Cancer Research 490
  • Biotechnology 267
  • Cell Biology 362
Replace Carl G. Maki with:
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Rebecca Haffner Israel
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Mark K. Saville relative to Carl G. Maki United States Carl G. Maki's profile →
Citations per field
00.5×1.6×
Carl G. Maki · 1×
Citations per year

Countries citing papers authored by Mark K. Saville

Since Specialization
Citations

This map shows the geographic impact of Mark K. Saville's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark K. Saville with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark K. Saville more than expected).

Fields of papers citing papers by Mark K. Saville

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark K. Saville. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark K. Saville. The network helps show where Mark K. Saville may publish in the future.

Co-authors

The 25 scholars most cited alongside Mark K. Saville, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark K. Saville Line = papers co-authored together Mark K. Saville links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.

#Work
1
p53 isoforms can regulate p53 transcriptional activity
Hit paper breakdown →
2005631
2 2004442
3 2007238
4 2006227
5 2000212
6 2001183
7 2008162
8 2001145
9 2004124
10 200987
11 201280
12 200871
13 199871
14 200150
15 200448
16 201340
17 200940
18 199740
19 200740
20 199438

About Mark K. Saville

Mark K. Saville is a scholar working on Molecular Biology, Oncology, Cell Biology, Epidemiology and Cancer Research, having authored 33 papers that have together received 3.2k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (20 papers), Ubiquitin and proteasome pathways (16 papers), Epigenetics and DNA Methylation (6 papers), Cancer-related gene regulation (4 papers), Genomics and Chromatin Dynamics (3 papers), Microtubule and mitosis dynamics (3 papers), Cancer Research and Treatments (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). The work is most often cited by research in Oncology (1.9k citations), Molecular Biology (2.7k citations), Cancer Research (490 citations), Biotechnology (267 citations) and Cell Biology (362 citations). Mark K. Saville has collaborated with scholars based in United Kingdom, United States and Belgium. Frequent co-authors include David P. Lane, Dimitris P. Xirodimas, Jean‐Christophe Bourdon, Alison Sparks, Nerea Allende-Vega, Ronald T. Hay, Kenneth Fernandes, Alexandra Diot, Geng Liu and Lauren Stevenson. Their work appears in journals such as Oncogene, Journal of Biological Chemistry, Biochemical Journal, The EMBO Journal and Oncotarget.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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