Mark J.S. Heath

3.3k total citations · 1 hit paper
14 papers, 1.8k citations indexed

About

Mark J.S. Heath is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Mark J.S. Heath has authored 14 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 6 papers in Physiology. Recurrent topics in Mark J.S. Heath's work include Ion channel regulation and function (5 papers), Pain Mechanisms and Treatments (4 papers) and Receptor Mechanisms and Signaling (4 papers). Mark J.S. Heath is often cited by papers focused on Ion channel regulation and function (5 papers), Pain Mechanisms and Treatments (4 papers) and Receptor Mechanisms and Signaling (4 papers). Mark J.S. Heath collaborates with scholars based in United States, Germany and United Kingdom. Mark J.S. Heath's co-authors include Marion B. E. Davis, Joseph J. LoTurco, David F. Owens, AR Kriegstein, René Hen, Mehmet C. Öz, Etienne Sibille, Luca Santarelli, Gabriella Gobbi and Pierre Blier and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Journal of Clinical Investigation.

In The Last Decade

Mark J.S. Heath

13 papers receiving 1.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

GABA and glutamate depolarize cortical progenitor cells and inhibit DNA synthesis

1995 886 citations Joseph J. LoTurco, David F. Owens et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mark J.S. Heath United States	11	993	712	407	172	166	14	1.8k
Takeshi Yagi Japan	19	525 0.5×	446 0.6×	139 0.3×	109 0.6×	226 1.4×	45	1.6k
Laurent Bezin France	25	439 0.4×	619 0.9×	189 0.5×	130 0.8×	183 1.1×	65	1.7k
Thomas M. Freiman Germany	24	932 0.9×	450 0.6×	304 0.7×	238 1.4×	152 0.9×	117	2.0k
Adam Chodobski United States	22	527 0.5×	740 1.0×	251 0.6×	42 0.2×	189 1.1×	48	2.1k
Kong Eric You-Ten Canada	14	509 0.5×	498 0.7×	109 0.3×	226 1.3×	167 1.0×	35	1.4k
David B. Kantor United States	13	678 0.7×	515 0.7×	237 0.6×	108 0.6×	150 0.9×	22	1.3k
William G. Dail United States	21	424 0.4×	504 0.7×	111 0.3×	57 0.3×	353 2.1×	39	1.8k
Guo‐Fang Tseng Taiwan	24	951 1.0×	360 0.5×	205 0.5×	384 2.2×	172 1.0×	81	1.8k
Yoshihiro Tsuruo Japan	37	1.2k 1.2×	781 1.1×	142 0.3×	154 0.9×	302 1.8×	125	3.6k

Countries citing papers authored by Mark J.S. Heath

Since Specialization

Citations

This map shows the geographic impact of Mark J.S. Heath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark J.S. Heath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark J.S. Heath more than expected).

Fields of papers citing papers by Mark J.S. Heath

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark J.S. Heath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark J.S. Heath. The network helps show where Mark J.S. Heath may publish in the future.

Co-authorship network of co-authors of Mark J.S. Heath

This figure shows the co-authorship network connecting the top 25 collaborators of Mark J.S. Heath. A scholar is included among the top collaborators of Mark J.S. Heath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark J.S. Heath. Mark J.S. Heath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

14 of 14 papers shown

Takayama, Hiroo, et al.. (2020). An Unrecognized Contraindication to Transesophageal Echocardiographic Probe Placement: The Aberrant Right Subclavian Artery. CASE. 4(5). 448–451. 1 indexed citations

Santarelli, Luca, Gabriella Gobbi, Etienne Sibille, et al.. (2001). Genetic and pharmacological disruption of neurokinin 1 receptor function decreases anxiety-related behaviors and increases serotonergic function. Proceedings of the National Academy of Sciences. 98(4). 1912–1917. 239 indexed citations

Heath, Mark J.S. & Marc L. Dickstein. (2000). Perioperative management of the left ventricular assist device recipient. Progress in Cardiovascular Diseases. 43(1). 47–54. 9 indexed citations

King, Tamara, et al.. (2000). The development of the nociceptive responses in neurokinin-1 receptor knockout mice. Neuroreport. 11(3). 587–591. 13 indexed citations

Wood, John N. & Mark J.S. Heath. (2000). Molecules that specify modality: Mechanisms of nociception. Journal of Pain. 1(3). 19–25. 3 indexed citations

Morales, David L.S., John D. Madigan, Suzanne Cullinane, et al.. (1999). Reversal by Vasopressin of Intractable Hypotension in the Late Phase of Hemorrhagic Shock. Circulation. 100(3). 226–229. 161 indexed citations

McGehee, Daniel S., et al.. (1997). Mechanism of extracellular Ca²⁺ receptor‐stimulated hormone release from sheep thyroid parafollicular cells. The Journal of Physiology. 502(1). 31–44. 86 indexed citations

Pinsky, David J., Yoshifumi Naka, Hui Liao, et al.. (1996). Hypoxia-induced exocytosis of endothelial cell Weibel-Palade bodies. A mechanism for rapid neutrophil recruitment after cardiac preservation.. Journal of Clinical Investigation. 97(2). 493–500. 238 indexed citations

Heath, Mark J.S. & René Hen. (1995). Serotonin Receptors: Genetic insights into serotonin function. Current Biology. 5(9). 997–999. 30 indexed citations

10.

LoTurco, Joseph J., David F. Owens, Mark J.S. Heath, Marion B. E. Davis, & AR Kriegstein. (1995). GABA and glutamate depolarize cortical progenitor cells and inhibit DNA synthesis. Neuron. 15(6). 1287–1298. 886 indexed citations breakdown →

11.

Kyrozis, Andreas, Peter A. Goldstein, Mark J.S. Heath, & Amy B. MacDermott. (1995). Calcium entry through a subpopulation of AMPA receptors desensitized neighbouring NMDA receptors in rat dorsal horn neurons.. The Journal of Physiology. 485(2). 373–381. 63 indexed citations

12.

Heath, Mark J.S., Thierry Lints, C. Justin Lee, & Jane Dodd. (1995). Functional expression of the tachykinin NK1 receptor by floor plate cells in the embryonic rat spinal cord and brainstem.. The Journal of Physiology. 486(1). 139–148. 12 indexed citations

13.

Heath, Mark J.S., Mary D. Womack, & Amy B. MacDermott. (1994). Substance P elevates intracellular calcium in both neurons and glial cells from the dorsal horn of the spinal cord. Journal of Neurophysiology. 72(3). 1192–1198. 47 indexed citations

14.

Robinson, John S., et al.. (1969). Physiological Responses to Intermittent Methohexitone for Conservative Dentistry. BMJ. 2(5656). 540–543. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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