Mark E. Massari

3.2k total citations · 1 hit paper
21 papers, 2.5k citations indexed

About

Mark E. Massari is a scholar working on Molecular Biology, Immunology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Mark E. Massari has authored 21 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 4 papers in Immunology and 3 papers in Cellular and Molecular Neuroscience. Recurrent topics in Mark E. Massari's work include Genomics and Chromatin Dynamics (5 papers), Nitric Oxide and Endothelin Effects (3 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Mark E. Massari is often cited by papers focused on Genomics and Chromatin Dynamics (5 papers), Nitric Oxide and Endothelin Effects (3 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Mark E. Massari collaborates with scholars based in United States, Netherlands and Australia. Mark E. Massari's co-authors include Cornelis Murre, Melanie W. Quong, Isaac Engel, Gretchen Bain, Maarten H. Stuiver, Beth Furnari, Marc A. van Dijk, Richard Rivera, Ronald Zwart and Patricia A. Jennings and has published in prestigious journals such as Blood, Molecular Cell and The Journal of Immunology.

In The Last Decade

Mark E. Massari

21 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Helix-Loop-Helix Proteins: Regulators of Transcription in Eucaryotic Organisms

2000 1.4k citations Mark E. Massari, Cornelis Murre Molecular and Cellular Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mark E. Massari United States	12	1.8k	423	390	388	217	21	2.5k
Bernard Jost France	27	1.6k 0.9×	434 1.0×	219 0.6×	235 0.6×	226 1.0×	48	2.3k
Bernward Klocke Germany	11	1.5k 0.8×	387 0.9×	138 0.4×	284 0.7×	214 1.0×	14	2.1k
Ramiro Ramírez‐Solis United Kingdom	23	2.4k 1.3×	1.1k 2.5×	268 0.7×	213 0.5×	285 1.3×	39	3.2k
Catherine Ucla Switzerland	22	1.7k 0.9×	644 1.5×	276 0.7×	382 1.0×	125 0.6×	26	2.5k
Tada-aki Hori Japan	27	1.5k 0.8×	574 1.4×	235 0.6×	334 0.9×	441 2.0×	89	2.3k
Eirik Frengen Norway	23	1.6k 0.9×	822 1.9×	329 0.8×	308 0.8×	354 1.6×	72	2.6k
Stéphanie Le Gras France	32	2.0k 1.1×	437 1.0×	347 0.9×	443 1.1×	290 1.3×	57	2.9k
Yvonne J. K. Edwards United States	26	1.7k 0.9×	566 1.3×	288 0.7×	226 0.6×	108 0.5×	67	2.5k
N. C. Tolga Emre United States	10	3.1k 1.7×	273 0.6×	364 0.9×	374 1.0×	347 1.6×	13	3.6k
N A Jenkins United States	32	2.1k 1.1×	788 1.9×	152 0.4×	698 1.8×	367 1.7×	37	3.4k

Countries citing papers authored by Mark E. Massari

Since Specialization

Citations

This map shows the geographic impact of Mark E. Massari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark E. Massari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark E. Massari more than expected).

Fields of papers citing papers by Mark E. Massari

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark E. Massari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark E. Massari. The network helps show where Mark E. Massari may publish in the future.

Co-authorship network of co-authors of Mark E. Massari

This figure shows the co-authorship network connecting the top 25 collaborators of Mark E. Massari. A scholar is included among the top collaborators of Mark E. Massari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark E. Massari. Mark E. Massari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

1.

Nguyen, Jasmine, Juping Liu, Paweł Stańczak, et al.. (2017). Method for rapid optimization of recombinant GPCR protein expression and stability using virus-like particles. Protein Expression and Purification. 133. 41–49. 10 indexed citations

2.

Nguyen, Jasmine, Juping Liu, Paweł Stańczak, et al.. (2017). Dataset on Galanin Receptor 3 mutants that improve recombinant receptor expression and stability in an agonist and antagonist bound form. Data in Brief. 12. 603–607. 2 indexed citations

3.

Rojas, Rafael J., et al.. (2015). Reversible and irreversible small molecule inhibitors of monoamine oxidase B (MAO-B) investigated by biophysical techniques. Bioorganic & Medicinal Chemistry. 23(4). 770–778. 30 indexed citations

4.

Gahman, Timothy C., Mark R. Herbert, Kent T. Symons, et al.. (2011). Identification and SAR of selective inducible nitric oxide synthase (iNOS) dimerization inhibitors. Bioorganic & Medicinal Chemistry Letters. 21(22). 6888–6894. 8 indexed citations

5.

Symons, Kent T., Mark E. Massari, John V. Anzola, et al.. (2010). Pharmacological Characterization of KLYP961, a Dual Inhibitor of Inducible and Neuronal Nitric-Oxide Synthases. Journal of Pharmacology and Experimental Therapeutics. 336(2). 468–478. 12 indexed citations

6.

Symons, Kent T., Mark E. Massari, Jeffrey Roppe, et al.. (2009). KLYP956 Is a Non-Imidazole-Based Orally Active Inhibitor of Nitric-Oxide Synthase Dimerization. Molecular Pharmacology. 76(1). 153–162. 11 indexed citations

7.

Bhalla, Savita, et al.. (2008). Differential Roles for the E2A Activation Domains in B Lymphocytes and Macrophages. The Journal of Immunology. 180(3). 1694–1703. 22 indexed citations

8.

Symons, Kent T., Mark E. Massari, David Jenkins, et al.. (2008). Inhibition of Inducible Nitric Oxide Synthase Expression by a Novel Small Molecule Activator of the Unfolded Protein Response. PubMed. 2. 1–9. 4 indexed citations

9.

Massari, Mark E. & Cornelis Murre. (2000). Helix-Loop-Helix Proteins: Regulators of Transcription in Eucaryotic Organisms. Molecular and Cellular Biology. 20(2). 429–440. 1450 indexed citations breakdown →

10.

Massari, Mark E., Patrick A. Grant, Marilyn G. Pray-Grant, et al.. (1999). A Conserved Motif Present in a Class of Helix-Loop-Helix Proteins Activates Transcription by Direct Recruitment of the SAGA Complex. Molecular Cell. 4(1). 63–73. 117 indexed citations

11.

Massari, Mark E., Richard Rivera, Joseph R. Voland, et al.. (1998). Characterization of ABF-1, a Novel Basic Helix-Loop-Helix Transcription Factor Expressed in Activated B Lymphocytes. Molecular and Cellular Biology. 18(6). 3130–3139. 58 indexed citations

12.

Massari, Mark E., Patricia A. Jennings, & Cornelis Murre. (1996). The AD1 Transactivation Domain of E2A Contains a Highly Conserved Helix Which Is Required for Its Activity in both Saccharomyces cerevisiae and Mammalian Cells. Molecular and Cellular Biology. 16(1). 121–129. 88 indexed citations

13.

Henderson, Eva, et al.. (1995). c-jun inhibits insulin control element-mediated transcription by affecting the transactivation potential of the E2A gene products. Molecular and Cellular Biology. 15(3). 1398–1404. 27 indexed citations

14.

Murre, Cornelis, Gretchen Bain, Marc A. van Dijk, et al.. (1994). Structure and function of helix-loop-helix proteins. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1218(2). 129–135. 407 indexed citations

15.

Ejima, Eri, et al.. (1993). Cell-type-specific transactivation of the parathyroid hormone-related protein gene promoter by the human T-cell leukemia virus type I (HTLV- I) tax and HTLV-II tax proteins. Blood. 81(4). 1017–1024. 41 indexed citations

16.

Quong, Melanie W., et al.. (1993). A new transcriptional-activation motif restricted to a class of helix-loop-helix proteins is functionally conserved in both yeast and mammalian cells.. Molecular and Cellular Biology. 13(2). 792–800. 68 indexed citations

17.

Quong, Melanie W., Mark E. Massari, Ronald Zwart, & Cornelis Murre. (1993). A New Transcriptional-Activation Motif Restricted to a Class of Helix-Loop-Helix Proteins Is Functionally Conserved in Both Yeast and Mammalian Cells. Molecular and Cellular Biology. 13(2). 792–800. 139 indexed citations

18.

Ejima, Eri, et al.. (1993). Cell-type-specific transactivation of the parathyroid hormone-related protein gene promoter by the human T-cell leukemia virus type I (HTLV- I) tax and HTLV-II tax proteins. Blood. 81(4). 1017–1024. 2 indexed citations

19.

Bennardini, Federico, Paola Failli, Rosanna Matucci, et al.. (1987). L‐ARGININE METHYLESTER REDUCES Ca²⁺/Cl⁻ ‐DEPENDENT L‐[³H]GLUTAMATE BINDING AND Ca²⁺‐ACTIVATED NEUTRAL PROTEASE ACTIVITY IN RAT HIPPOCAMPAL MEMBRANES. Fundamental and Clinical Pharmacology. 1(5). 297–306. 1 indexed citations

20.

Massari, Mark E., et al.. (1971). Tréhalase du sérum humain dans quelques affections hépatiques. Clinica Chimica Acta. 35(2). 335–343. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact