Mark Carlton

10.2k total citations · 5 hit papers
46 papers, 7.6k citations indexed

About

Mark Carlton is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Mark Carlton has authored 46 papers receiving a total of 7.6k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 11 papers in Genetics. Recurrent topics in Mark Carlton's work include Receptor Mechanisms and Signaling (9 papers), Hypothalamic control of reproductive hormones (6 papers) and Animal Genetics and Reproduction (6 papers). Mark Carlton is often cited by papers focused on Receptor Mechanisms and Signaling (9 papers), Hypothalamic control of reproductive hormones (6 papers) and Animal Genetics and Reproduction (6 papers). Mark Carlton collaborates with scholars based in United Kingdom, United States and Japan. Mark Carlton's co-authors include William H Colledge, Martin Evans, Samuel Aparício, Dirk Zahn, Alan G. Hendrick, Rosemary R. Thresher, John Dixon, Sophie Messager, Emmanouella E. Chatzidaki and Stephen O’Rahilly and has published in prestigious journals such as Nature, New England Journal of Medicine and Cell.

In The Last Decade

Mark Carlton

43 papers receiving 7.4k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The GPR54 Gene as a Regulator of Puberty

2003 1.9k citations Stephanie B. Seminara, Sophie Messager et al. New England Journal of Medicine profile →
Kisspeptin directly stimulates gonadotropin-releasing hormone release via G protein-coupled receptor 54

2005 959 citations Sophie Messager, Emmanouella E. Chatzidaki et al. Proceedings of the National Academy of Sciences profile →
The Oncogenic Cysteine-rich LIM domain protein Rbtn2 is essential for erythroid development

1994 513 citations Alan J. Warren, William H Colledge et al. Cell profile →
Eomesodermin is required for mouse trophoblast development and mesoderm formation

2000 506 citations Andreas Russ, William H Colledge et al. Nature profile →
Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs

1994 401 citations William H Colledge, Mark Carlton et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mark Carlton United Kingdom	24	4.3k	3.6k	1.6k	1.1k	702	46	7.6k
Stephanie B. Seminara United States	49	5.3k 1.2×	8.9k 2.5×	3.2k 2.0×	1.6k 1.5×	616 0.9×	122	10.5k
Ana Cláudia Latronico Brazil	50	3.5k 0.8×	3.9k 1.1×	2.7k 1.7×	364 0.3×	843 1.2×	225	8.0k
Holly A. Ingraham United States	56	7.4k 1.7×	1.9k 0.5×	5.1k 3.3×	620 0.6×	1.5k 2.1×	101	12.9k
Susan Wray United States	47	1.7k 0.4×	3.2k 0.9×	1.4k 0.9×	810 0.7×	215 0.3×	124	6.0k
Nicolás de Roux France	32	2.5k 0.6×	3.4k 0.9×	1.7k 1.1×	655 0.6×	261 0.4×	112	5.3k
Vincent Goffin France	49	3.0k 0.7×	949 0.3×	1.9k 1.2×	474 0.4×	259 0.4×	167	8.5k
Yayoi Ikeda Japan	34	4.1k 1.0×	1.7k 0.5×	4.7k 3.0×	356 0.3×	916 1.3×	130	7.9k
Hamish M. Fraser United Kingdom	51	2.5k 0.6×	4.3k 1.2×	1.4k 0.9×	309 0.3×	2.3k 3.2×	191	8.3k
Masatsugu Ueda Japan	37	2.3k 0.5×	695 0.2×	837 0.5×	1.5k 1.4×	471 0.7×	169	6.0k
Christopher J. Ormandy Australia	51	3.5k 0.8×	597 0.2×	1.9k 1.2×	324 0.3×	326 0.5×	110	7.2k

Countries citing papers authored by Mark Carlton

Since Specialization

Citations

This map shows the geographic impact of Mark Carlton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Carlton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Carlton more than expected).

Fields of papers citing papers by Mark Carlton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Carlton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Carlton. The network helps show where Mark Carlton may publish in the future.

Co-authorship network of co-authors of Mark Carlton

This figure shows the co-authorship network connecting the top 25 collaborators of Mark Carlton. A scholar is included among the top collaborators of Mark Carlton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark Carlton. Mark Carlton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Kieburtz, Karl, et al.. (2024). CVN424, A novel GPR6 inverse agonist for the treatment of Parkinson disease (PD): Phase 2 clinical trial. Parkinsonism & Related Disorders. 122. 106124–106124.

Bürli, Roland W., David G. Livermore, Robert G. Newman, et al.. (2024). Discovery and first-time disclosure of CVN766, an exquisitely selective orexin 1 receptor antagonist. Bioorganic & Medicinal Chemistry Letters. 100. 129629–129629. 7 indexed citations

Chadwick, Wayne, Stuart Maudsley, William M. Hull, et al.. (2023). The oDGal Mouse: A Novel, Physiologically Relevant Rodent Model of Sporadic Alzheimer’s Disease. International Journal of Molecular Sciences. 24(8). 6953–6953. 6 indexed citations

Margolin, David, et al.. (2022). A Phase I, First-in-Human, Healthy Volunteer Study to Investigate the Safety, Tolerability, and Pharmacokinetics of CVN424, a Novel G Protein-Coupled Receptor 6 Inverse Agonist for Parkinson’s Disease. Journal of Pharmacology and Experimental Therapeutics. 381(1). 33–41. 8 indexed citations

Soutar, Marc P. M., Shuichi Miyakawa, Jasmine Harley, et al.. (2018). AKT signalling selectively regulates PINK1 mitophagy in SHSY5Y cells and human iPSC-derived neurons. Scientific Reports. 8(1). 8855–8855. 47 indexed citations

Waller‐Evans, Helen, Simone Prömel, Tobias Langenhan, et al.. (2010). The Orphan Adhesion-GPCR GPR126 Is Required for Embryonic Development in the Mouse. PLoS ONE. 5(11). e14047–e14047. 66 indexed citations

Cash, Jenna L., Andreas Russ, John Dixon, et al.. (2008). Synthetic chemerin-derived peptides suppress inflammation through ChemR23. The Journal of Experimental Medicine. 205(4). 767–775. 313 indexed citations

Tassigny, Xavier d’Anglemont de, et al.. (2008). Kisspeptin Can Stimulate Gonadotropin-Releasing Hormone (GnRH) Release by a Direct Action at GnRH Nerve Terminals. Endocrinology. 149(8). 3926–3932. 229 indexed citations

Tassigny, Xavier d’Anglemont de, John Dixon, Kate Day, et al.. (2007). Hypogonadotropic hypogonadism in mice lacking a functional Kiss1 gene. Proceedings of the National Academy of Sciences. 104(25). 10714–10719. 478 indexed citations

10.

Carlton, Mark, et al.. (2007). Hypogonadotrophic hypogonadism in mice lacking a functional Kiss-1 gene. 14. 10 indexed citations

11.

Seminara, Stephanie B., Sophie Messager, Emmanouella E. Chatzidaki, et al.. (2004). The GPR54 Gene as a Regulator of Puberty. Obstetrical & Gynecological Survey. 59(5). 351–353. 173 indexed citations

12.

Tse, Eric, Andrew J.H. Smith, Stephen P. Hunt, et al.. (2004). Null Mutation of the Lmo4 Gene or a Combined Null Mutation of the Lmo1 / Lmo3 Genes Causes Perinatal Lethality, and Lmo4 Controls Neural Tube Development in Mice. Molecular and Cellular Biology. 24(5). 2063–2073. 72 indexed citations

13.

Payer, Bernhard, Mitinori Saitou, Sheila C. Barton, et al.. (2003). stella Is a Maternal Effect Gene Required for Normal Early Development in Mice. Current Biology. 13(23). 2110–2117. 299 indexed citations

14.

Groombridge, Jim J., Carl G. Jones, Richard A. Nichols, Mark Carlton, & Michael W. Bruford. (2003). Molecular phylogeny and morphological change in the Psittacula parakeets. Molecular Phylogenetics and Evolution. 31(1). 96–108. 39 indexed citations

15.

Seminara, Stephanie B., Sophie Messager, Emmanouella E. Chatzidaki, et al.. (2003). The GPR54 Gene as a Regulator of Puberty. New England Journal of Medicine. 349(17). 1614–1627. 1950 indexed citations breakdown →

16.

Gilmour, Darren, Gholson J. Lyon, Mark Carlton, et al.. (1998). Mice deficient for the secreted glycoprotein SPARC/osteonectin/BM40 develop normally but show severe age-onset cataract formation and disruption of the lens. The EMBO Journal. 17(7). 1860–1870. 199 indexed citations

17.

Couldrey, Christine, Mark Carlton, Jack Ferrier, William H Colledge, & Martin Evans. (1998). Disruption of murine α-enolase by a retroviral gene trap results in early embryonic lethality. Developmental Dynamics. 212(2). 284–292. 10 indexed citations

18.

Evans, Martin, Mark Carlton, & Andreas Russ. (1997). Gene trapping and functional genomics. Trends in Genetics. 13(9). 370–374. 60 indexed citations

19.

Colledge, William H, Mark Carlton, Garry B. Udy, & Martin Evans. (1994). Disruption of c-mos causes parthenogenetic development of unfertilized mouse eggs. Nature. 370(6484). 65–68. 401 indexed citations breakdown →

20.

Warren, Alan J., William H Colledge, Mark Carlton, et al.. (1994). The Oncogenic Cysteine-rich LIM domain protein Rbtn2 is essential for erythroid development. Cell. 78(1). 45–57. 513 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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