Mark C. Wilkes

3.0k total citations
36 papers, 2.5k citations indexed

About

Mark C. Wilkes is a scholar working on Molecular Biology, Hematology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Mark C. Wilkes has authored 36 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 7 papers in Hematology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Mark C. Wilkes's work include RNA modifications and cancer (12 papers), TGF-β signaling in diseases (11 papers) and Cancer-related gene regulation (10 papers). Mark C. Wilkes is often cited by papers focused on RNA modifications and cancer (12 papers), TGF-β signaling in diseases (11 papers) and Cancer-related gene regulation (10 papers). Mark C. Wilkes collaborates with scholars based in United States, United Kingdom and Sweden. Mark C. Wilkes's co-authors include Edward B. Leof, Maryanne Edens, Raimund Hirschberg, Shinong Wang, Andrew H. Limper, Craig Daniels, Stephen J. Murphy, Stephen Murphy, Asif Ahmed and Stephen J. Murphy and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Blood.

In The Last Decade

Mark C. Wilkes

35 papers receiving 2.5k citations

Peers

Mark C. Wilkes
Evangelia Pardali Netherlands
Fangming Lin United States
Karin Dahan Belgium
Lee Ann Baldridge United States
Evangelia Pardali Netherlands
Mark C. Wilkes
Citations per year, relative to Mark C. Wilkes Mark C. Wilkes (= 1×) peers Evangelia Pardali

Countries citing papers authored by Mark C. Wilkes

Since Specialization
Citations

This map shows the geographic impact of Mark C. Wilkes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark C. Wilkes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark C. Wilkes more than expected).

Fields of papers citing papers by Mark C. Wilkes

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark C. Wilkes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark C. Wilkes. The network helps show where Mark C. Wilkes may publish in the future.

Co-authorship network of co-authors of Mark C. Wilkes

This figure shows the co-authorship network connecting the top 25 collaborators of Mark C. Wilkes. A scholar is included among the top collaborators of Mark C. Wilkes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark C. Wilkes. Mark C. Wilkes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wilkes, Mark C., Y. Lucy Liu, Hye Na Kim, et al.. (2024). Activation of nemo-like kinase in diamond blackfan anemia suppresses early erythropoiesis by preventing mitochondrial biogenesis. Journal of Biological Chemistry. 300(8). 107542–107542. 1 indexed citations
2.
Wilkes, Mark C., Hee‐Don Chae, Alma‐Martina Cepika, et al.. (2023). SATB1 Chromatin Loops Regulate Megakaryocyte/Erythroid Progenitor Expansion by Facilitating HSP70 and GATA1 Induction. Stem Cells. 41(6). 560–569. 1 indexed citations
4.
Wilkes, Mark C., Alma‐Martina Cepika, Ascia Eskin, et al.. (2022). Downregulation of SATB1 by miRNAs reduces megakaryocyte/erythroid progenitor expansion in preclinical models of Diamond–Blackfan anemia. Experimental Hematology. 111. 66–78. 5 indexed citations
5.
Liu, Y. Lucy, et al.. (2022). Animal models of Diamond-Blackfan anemia: updates and challenges. Haematologica. 108(5). 1222–1231. 6 indexed citations
6.
Wilkes, Mark C., et al.. (2021). The active component of ginseng, ginsenoside Rb1, improves erythropoiesis in models of Diamond–Blackfan anemia by targeting Nemo-like kinase. Journal of Biological Chemistry. 297(3). 100988–100988. 10 indexed citations
7.
Wilkes, Mark C., Kavitha Siva, Gianluca Varetti, et al.. (2020). Metformin-induced suppression of Nemo-like kinase improves erythropoiesis in preclinical models of Diamond–Blackfan anemia through induction of miR-26a. Experimental Hematology. 91. 65–77. 11 indexed citations
8.
Danilova, Nadia, et al.. (2018). Innate immune system activation in zebrafish and cellular models of Diamond Blackfan Anemia. Scientific Reports. 8(1). 5165–5165. 22 indexed citations
9.
Wilkes, Mark C., et al.. (2018). Chromatin Organization By SATB1 Regulates HSP70 Induction in Early Erythropoiesis and Lost in Diamond Blackfan Anemia. Blood. 132(Supplement 1). 2591–2591. 2 indexed citations
10.
Wilkes, Mark C., Claire E. Repellin, & Kathleen M. Sakamoto. (2017). Beyond mRNA: The role of non-coding RNAs in normal and aberrant hematopoiesis. Molecular Genetics and Metabolism. 122(3). 28–38. 17 indexed citations
11.
Hong, Min, Mark C. Wilkes, Sumedha G. Penheiter, et al.. (2011). Non-Smad Transforming Growth Factor-β Signaling Regulated by Focal Adhesion Kinase Binding the p85 Subunit of Phosphatidylinositol 3-Kinase. Journal of Biological Chemistry. 286(20). 17841–17850. 45 indexed citations
12.
Andrianifahanana, Mahefatiana, Mark C. Wilkes, Claire E. Repellin, et al.. (2010). ERBB Receptor Activation Is Required for Profibrotic Responses to Transforming Growth Factor β. Cancer Research. 70(19). 7421–7430. 19 indexed citations
13.
Penheiter, Sumedha G., Raman Deep Singh, Claire E. Repellin, et al.. (2010). Type II Transforming Growth Factor-β Receptor Recycling Is Dependent upon the Clathrin Adaptor Protein Dab2. Molecular Biology of the Cell. 21(22). 4009–4019. 53 indexed citations
14.
Wilkes, Mark C., Claire E. Repellin, Min Hong, et al.. (2009). Erbin and the NF2 Tumor Suppressor Merlin Cooperatively Regulate Cell-Type-Specific Activation of PAK2 by TGF-β. Developmental Cell. 16(3). 433–444. 37 indexed citations
15.
Bhattacharyya, Swati, Wataru Ishida, Minghua Wu, et al.. (2009). A non-Smad mechanism of fibroblast activation by transforming growth factor-β via c-Abl and Egr-1: selective modulation by imatinib mesylate. Oncogene. 28(10). 1285–1297. 67 indexed citations
16.
Wilkes, Mark C. & Edward B. Leof. (2006). Transforming Growth Factor β Activation of c-Abl Is Independent of Receptor Internalization and Regulated by Phosphatidylinositol 3-Kinase and PAK2 in Mesenchymal Cultures. Journal of Biological Chemistry. 281(38). 27846–27854. 59 indexed citations
17.
Daniels, Craig, Mark C. Wilkes, Maryanne Edens, et al.. (2004). Imatinib mesylate inhibits the profibrogenic activity of TGF-β and prevents bleomycin-mediated lung fibrosis. Journal of Clinical Investigation. 114(9). 1308–1316. 442 indexed citations
18.
Murphy, Stephen J., Jules J.E. Doré, Maryanne Edens, et al.. (2004). Differential Trafficking of Transforming Growth Factor-β Receptors and Ligand in Polarized Epithelial Cells. Molecular Biology of the Cell. 15(6). 2853–2862. 39 indexed citations
19.
Wilkes, Mark C., Stephen J. Murphy, Nandor Garamszegi, & Edward B. Leof. (2003). Cell-Type-Specific Activation of PAK2 by Transforming Growth Factor β Independent of Smad2 and Smad3. Molecular and Cellular Biology. 23(23). 8878–8889. 115 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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