Mark Boldin

21.3k total citations · 10 hit papers
52 papers, 16.5k citations indexed

About

Mark Boldin is a scholar working on Cancer Research, Molecular Biology and Immunology. According to data from OpenAlex, Mark Boldin has authored 52 papers receiving a total of 16.5k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Cancer Research, 32 papers in Molecular Biology and 22 papers in Immunology. Recurrent topics in Mark Boldin's work include MicroRNA in disease regulation (28 papers), Cancer-related molecular mechanisms research (12 papers) and Circular RNAs in diseases (11 papers). Mark Boldin is often cited by papers focused on MicroRNA in disease regulation (28 papers), Cancer-related molecular mechanisms research (12 papers) and Circular RNAs in diseases (11 papers). Mark Boldin collaborates with scholars based in United States, Israel and Japan. Mark Boldin's co-authors include David Baltimore, Konstantin D. Taganov, David Wallach, Ryan M. O’Connell, Tanya Goncharov, Nikolay Malinin, Eugene Varfolomeev, Dinesh S. Rao, Genhong Cheng and Igor Mett and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Mark Boldin

52 papers receiving 16.3k citations

Hit Papers

NF-κB-dependent induction of microRNA miR-146, an inhibi... 1995 2026 2005 2015 2006 1996 2007 1997 1999 1000 2.0k 3.0k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. NF-κB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses
    2006 3.6k citations Konstantin D. Taganov, Mark Boldin et al. Proceedings of the National Academy of Sciences profile →
  2. Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death
    1996 2.0k citations Mark Boldin, Tanya Goncharov et al. Cell profile →
  3. MicroRNA-155 is induced during the macrophage inflammatory response
    2007 1.6k citations Ryan M. O’Connell, Konstantin D. Taganov et al. Proceedings of the National Academy of Sciences profile →
  4. MAP3K-related kinase involved in NF-KB induction by TNF, CD95 and IL-1
    1997 1.2k citations Nikolay Malinin, Mark Boldin et al. Nature profile →
  5. TUMOR NECROSIS FACTOR RECEPTOR AND Fas SIGNALING MECHANISMS
    1999 1.1k citations David Wallach, Eugene Varfolomeev et al. profile →
  6. MicroRNAs: new regulators of immune cell development and function
    2008 923 citations David Baltimore, Mark Boldin et al. Nature Immunology profile →
  7. A Novel Protein That Interacts with the Death Domain of Fas/APO1 Contains a Sequence Motif Related to the Death Domain
    1995 850 citations Mark Boldin, Eugene Varfolomeev et al. Journal of Biological Chemistry profile →
  8. Function of miR-146a in Controlling Treg Cell-Mediated Regulation of Th1 Responses
    2010 819 citations Mark Boldin, Konstantin D. Taganov et al. Cell profile →
  9. miR-146a is a significant brake on autoimmunity, myeloproliferation, and cancer in mice
    2011 704 citations Mark Boldin, Konstantin D. Taganov et al. The Journal of Experimental Medicine profile →
  10. Sustained expression of microRNA-155 in hematopoietic stem cells causes a myeloproliferative disorder
    2008 557 citations Ryan M. O’Connell, Dinesh S. Rao et al. The Journal of Experimental Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark Boldin United States 32 10.5k 8.7k 6.3k 1.8k 1.2k 52 16.5k
Guido Franzoso United States 49 6.5k 0.6× 5.5k 0.6× 5.3k 0.8× 2.7k 1.5× 1.2k 0.9× 96 13.2k
Amer A. Beg United States 51 8.0k 0.8× 7.2k 0.8× 8.2k 1.3× 3.5k 2.0× 1.4k 1.1× 95 16.4k
Harald Wajant Germany 65 8.8k 0.8× 3.8k 0.4× 6.8k 1.1× 2.9k 1.6× 1.3k 1.0× 222 15.6k
Ke Zen China 78 12.6k 1.2× 10.1k 1.2× 3.5k 0.5× 1.3k 0.8× 1.5k 1.2× 254 19.3k
Michael J. May United States 48 5.1k 0.5× 4.9k 0.6× 5.3k 0.8× 2.1k 1.2× 1.2k 0.9× 84 12.2k
Xin Lin United States 70 7.2k 0.7× 3.8k 0.4× 5.5k 0.9× 2.6k 1.5× 1.7k 1.4× 252 15.0k
Cun-Yu Wang United States 40 11.5k 1.1× 5.4k 0.6× 3.5k 0.6× 3.7k 2.1× 1.1k 0.9× 48 18.6k
Hiroyasu Nakano Japan 56 5.9k 0.6× 3.7k 0.4× 4.8k 0.8× 2.1k 1.2× 1.3k 1.0× 158 11.3k
Claus Scheidereit Germany 66 7.6k 0.7× 5.4k 0.6× 5.3k 0.8× 2.9k 1.7× 801 0.6× 103 14.3k
Karen O’Rourke United States 39 15.5k 1.5× 4.3k 0.5× 7.8k 1.2× 3.5k 2.0× 2.1k 1.6× 44 20.2k

Countries citing papers authored by Mark Boldin

Since Specialization
Citations

This map shows the geographic impact of Mark Boldin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Boldin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Boldin more than expected).

Fields of papers citing papers by Mark Boldin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Boldin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Boldin. The network helps show where Mark Boldin may publish in the future.

Co-authorship network of co-authors of Mark Boldin

This figure shows the co-authorship network connecting the top 25 collaborators of Mark Boldin. A scholar is included among the top collaborators of Mark Boldin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark Boldin. Mark Boldin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xiong, Min, et al.. (2024). The epithelial C15ORF48/miR-147-NDUFA4 axis is an essential regulator of gut inflammation, energy metabolism, and the microbiome. Proceedings of the National Academy of Sciences. 121(27). e2315944121–e2315944121. 4 indexed citations
2.
Wang, Wei-Le, et al.. (2022). The role of microRNA-142 in B cell activation and effector functions. The Journal of Immunology. 208(Supplement_1). 168.02–168.02. 1 indexed citations
3.
Saferding, Victoria, Melanie Hofmann, Julia S. Brunner, et al.. (2020). microRNA‐146a controls age‐related bone loss. Aging Cell. 19(11). e13244–e13244. 28 indexed citations
4.
Węgrzyn, Joanna, Kieran O’Neill, David J. H. F. Knapp, et al.. (2020). Altered microRNA expression links IL6 and TNF-induced inflammaging with myeloid malignancy in humans and mice. Blood. 135(25). 2235–2251. 41 indexed citations
5.
Su, Yu‐Lin, Xiuli Wang, Mati Mann, et al.. (2019). Myeloid cell–targeted miR-146a mimic inhibits NF-κB–driven inflammation and leukemia progression in vivo. Blood. 135(3). 167–180. 97 indexed citations
6.
Li, Bo, Xi Wang, In Young Choi, et al.. (2017). miR-146a modulates autoreactive Th17 cell differentiation and regulates organ-specific autoimmunity. Journal of Clinical Investigation. 127(10). 3702–3716. 115 indexed citations
7.
Butcher, Matthew J., Tayab Waseem, Nathaniel Magilnick, et al.. (2016). Atherosclerosis-Driven Treg Plasticity Results in Formation of a Dysfunctional Subset of Plastic IFNγ + Th1/Tregs. Circulation Research. 119(11). 1190–1203. 145 indexed citations
8.
Ito, Yoshiaki, Atsushi Inoue, Arisa Igarashi, et al.. (2016). Identification of targets of tumor suppressor microRNA-34a using a reporter library system. 2 indexed citations
9.
Rebane, Ana, Toomas Runnel, Alar Aab, et al.. (2014). MicroRNA-146a alleviates chronic skin inflammation in atopic dermatitis through suppression of innate immune responses in keratinocytes. Journal of Allergy and Clinical Immunology. 134(4). 836–847.e11. 161 indexed citations
10.
Boldin, Mark, Konstantin D. Taganov, Dinesh S. Rao, et al.. (2011). miR-146a is a significant brake on autoimmunity, myeloproliferation, and cancer in mice. The Journal of Experimental Medicine. 208(6). 1189–1201. 704 indexed citations breakdown →
11.
Boldin, Mark, Konstantin D. Taganov, Dinesh S. Rao, et al.. (2011). miR-146ais a significant brake on autoimmunity, myeloproliferation, and cancer in mice. The Journal of Cell Biology. 193(4). i10–i10. 1 indexed citations
12.
O’Connell, Ryan M., Dinesh S. Rao, Aadel A. Chaudhuri, et al.. (2008). Sustained expression of microRNA-155 in hematopoietic stem cells causes a myeloproliferative disorder. The Journal of Experimental Medicine. 205(3). 585–594. 557 indexed citations breakdown →
13.
Baltimore, David, Mark Boldin, Ryan M. O’Connell, Dinesh S. Rao, & Konstantin D. Taganov. (2008). MicroRNAs: new regulators of immune cell development and function. Nature Immunology. 9(8). 839–845. 923 indexed citations breakdown →
14.
Werner, Shannon L., Jeffrey D. Kearns, Candace Lynch, et al.. (2008). Encoding NF-κB temporal control in response to TNF: distinct roles for the negative regulators IκBα and A20. Genes & Development. 22(15). 2093–2101. 173 indexed citations
15.
O’Connell, Ryan M., Konstantin D. Taganov, Mark Boldin, Genhong Cheng, & David Baltimore. (2007). MicroRNA-155 is induced during the macrophage inflammatory response. Proceedings of the National Academy of Sciences. 104(5). 1604–1609. 1552 indexed citations breakdown →
16.
Malinin, Nikolay, et al.. (1997). MAP3K-related kinase involved in NF-KB induction by TNF, CD95 and IL-1. Nature. 385(6616). 540–544. 1153 indexed citations breakdown →
17.
Boldin, Mark, et al.. (1996). Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death. Cell. 85(6). 803–815. 1991 indexed citations breakdown →
18.
Varfolomeev, Eugene, Mark Boldin, Tanya Goncharov, & David Wallach. (1996). A potential mechanism of "cross-talk" between the p55 tumor necrosis factor receptor and Fas/APO1: proteins binding to the death domains of the two receptors also bind to each other.. The Journal of Experimental Medicine. 183(3). 1271–1275. 109 indexed citations
19.
Boldin, Mark, Eugene Varfolomeev, Zeev Pancer, et al.. (1995). A Novel Protein That Interacts with the Death Domain of Fas/APO1 Contains a Sequence Motif Related to the Death Domain. Journal of Biological Chemistry. 270(14). 7795–7798. 850 indexed citations breakdown →
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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