Mark A. Plessinger

1.1k total citations
32 papers, 829 citations indexed

About

Mark A. Plessinger is a scholar working on Pediatrics, Perinatology and Child Health, Pharmacology and Epidemiology. According to data from OpenAlex, Mark A. Plessinger has authored 32 papers receiving a total of 829 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Pediatrics, Perinatology and Child Health, 7 papers in Pharmacology and 6 papers in Epidemiology. Recurrent topics in Mark A. Plessinger's work include Prenatal Substance Exposure Effects (10 papers), Cannabis and Cannabinoid Research (6 papers) and Neurotransmitter Receptor Influence on Behavior (4 papers). Mark A. Plessinger is often cited by papers focused on Prenatal Substance Exposure Effects (10 papers), Cannabis and Cannabinoid Research (6 papers) and Neurotransmitter Receptor Influence on Behavior (4 papers). Mark A. Plessinger collaborates with scholars based in United States. Mark A. Plessinger's co-authors include James R. Woods, Richard K. Miller, K. Clark, Karl B. Scheidweiler, Archana Sharma, Tai C. Kwong, David M. Sherer, Alan G. Fantel, Ronald W. Wood and Edward P. Norkus and has published in prestigious journals such as Annals of the New York Academy of Sciences, American Journal of Obstetrics and Gynecology and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Mark A. Plessinger

31 papers receiving 793 citations

Peers

Mark A. Plessinger
Rose Maxwell United States
Per Melin Sweden
Stephen A. Greene United States
Kim Dao Switzerland
Harlan F. Hill United States
Salha S. Daniel United States
Nicole Calder United Kingdom
Shaon Sengupta United States
Rose Maxwell United States
Mark A. Plessinger
Citations per year, relative to Mark A. Plessinger Mark A. Plessinger (= 1×) peers Rose Maxwell

Countries citing papers authored by Mark A. Plessinger

Since Specialization
Citations

This map shows the geographic impact of Mark A. Plessinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark A. Plessinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark A. Plessinger more than expected).

Fields of papers citing papers by Mark A. Plessinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark A. Plessinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark A. Plessinger. The network helps show where Mark A. Plessinger may publish in the future.

Co-authorship network of co-authors of Mark A. Plessinger

This figure shows the co-authorship network connecting the top 25 collaborators of Mark A. Plessinger. A scholar is included among the top collaborators of Mark A. Plessinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark A. Plessinger. Mark A. Plessinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Scheidweiler, Karl B., et al.. (2003). Pharmacokinetics and Pharmacodynamics of Methylecgonidine, a Crack Cocaine Pyrolyzate. Journal of Pharmacology and Experimental Therapeutics. 307(3). 1179–1187. 24 indexed citations
2.
Woods, James R., et al.. (2002). The effect of labor on maternal and fetal vitamins C and E. American Journal of Obstetrics and Gynecology. 187(5). 1179–1183. 31 indexed citations
3.
Woods, James R., Mark A. Plessinger, & Richard K. Miller. (2001). Vitamins C and E: Missing links in preventing preterm premature rupture of membranes?. American Journal of Obstetrics and Gynecology. 185(1). 5–10. 92 indexed citations
4.
Plessinger, Mark A., James R. Woods, & Richard K. Miller. (2000). Pretreatment of human amnion-chorion with vitamins C and E prevents hypochlorous acid–induced damage. American Journal of Obstetrics and Gynecology. 183(4). 979–985. 58 indexed citations
5.
Plessinger, Mark A. & Richard K. Miller. (1999). Effects of Zidovudine (AZT) and dideoxyinosine (ddI) on human trophoblast cells. Reproductive Toxicology. 13(6). 537–546. 4 indexed citations
6.
Plessinger, Mark A. & James R. Woods. (1998). COCAINE IN PREGNANCY. Obstetrics and Gynecology Clinics of North America. 25(1). 99–118. 29 indexed citations
7.
Woods, James R., Mark A. Plessinger, & Alan G. Fantel. (1998). AN INTRODUCTION TO REACTIVE OXYGEN SPECIES AND THEIR POSSIBLE ROLES IN SUBSTANCE ABUSE. Obstetrics and Gynecology Clinics of North America. 25(1). 219–236. 20 indexed citations
8.
Plessinger, Mark A., et al.. (1997). Pharmacokinetic and Toxicity Studies of AZT (Zidovudine) Following Perfusion of Human Term Placenta for 14 Hours. Toxicology and Applied Pharmacology. 143(1). 13–21. 24 indexed citations
9.
Plessinger, Mark A., et al.. (1997). Human Placenta Does Not Reduce AZT (Zidovudine) to 3'-Amino-3'-Deoxythymidine. Experimental Biology and Medicine. 215(3). 243–247. 5 indexed citations
10.
Sherer, David M., et al.. (1994). Fetal scapular length in the ultrasonographic assessment of gestational age.. Journal of Ultrasound in Medicine. 13(7). 523–528. 5 indexed citations
11.
Woods, James R., et al.. (1994). Pregnancy enhances cocaine's actions on the heart and within the peripheral circulation. American Journal of Obstetrics and Gynecology. 170(4). 1027–1035. 10 indexed citations
12.
Sharma, Archana, Mark A. Plessinger, Richard K. Miller, & James R. Woods. (1993). Progesterone Antagonist Mifepristone (RU 486) Decreases Cardiotoxicity of Cocaine. Experimental Biology and Medicine. 202(3). 279–287. 12 indexed citations
13.
Plessinger, Mark A. & James R. Woods. (1993). Maternal, Placental, and Fetal Pathophysiology of Cocaine Exposure During Pregnancy. Clinical Obstetrics & Gynecology. 36(2). 267–278. 44 indexed citations
14.
Sherer, David M., Jacques S. Abramowicz, Mark A. Plessinger, & James R. Woods. (1993). Fetal sacral length in the ultrasonographic assessment of gestational age. American Journal of Obstetrics and Gynecology. 168(2). 626–633. 20 indexed citations
15.
Sharma, Archana, et al.. (1992). Pregnancy enhances cardiotoxicity of cocaine: Role of progesterone. Toxicology and Applied Pharmacology. 113(1). 30–35. 22 indexed citations
16.
Plessinger, Mark A. & James R. Woods. (1991). The cardiovascular effects of cocaine use in pregnancy. Reproductive Toxicology. 5(2). 99–113. 23 indexed citations
17.
Woods, James R. & Mark A. Plessinger. (1991). Maternal-Fetal Cardiovascular System: A Target of Cocaine. PsycEXTRA Dataset. 3 indexed citations
18.
Plessinger, Mark A., et al.. (1990). Progesterone increases cardiovascular toxicity to cocaine in nonpregnant ewes. American Journal of Obstetrics and Gynecology. 163(5). 1659–1664. 29 indexed citations
19.
Woods, James R. & Mark A. Plessinger. (1990). Pregnancy increases cardiovascular toxicity to cocaine. American Journal of Obstetrics and Gynecology. 162(2). 529–533. 59 indexed citations
20.
Plessinger, Mark A. & James R. Woods. (1987). Fetal auditory brain stem response: Effect of increasing stimulus rate during functional auditory development. American Journal of Obstetrics and Gynecology. 157(6). 1382–1387. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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