María‐Ángeles Navas

1.1k total citations
24 papers, 839 citations indexed

About

María‐Ángeles Navas is a scholar working on Surgery, Molecular Biology and Genetics. According to data from OpenAlex, María‐Ángeles Navas has authored 24 papers receiving a total of 839 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Surgery, 14 papers in Molecular Biology and 6 papers in Genetics. Recurrent topics in María‐Ángeles Navas's work include Pancreatic function and diabetes (15 papers), Metabolism, Diabetes, and Cancer (10 papers) and Autophagy in Disease and Therapy (4 papers). María‐Ángeles Navas is often cited by papers focused on Pancreatic function and diabetes (15 papers), Metabolism, Diabetes, and Cancer (10 papers) and Autophagy in Disease and Therapy (4 papers). María‐Ángeles Navas collaborates with scholars based in Spain, United States and Netherlands. María‐Ángeles Navas's co-authors include Markus Stoffel, Stephen A. Duncan, Janet Rossant, Daniel Dufort, Juana M. Gancedo, Satoru Kuwajima, David Q. Shih, Olivier Vincent, Sebastián Cerdán and Enrique Blázquez and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and PLoS ONE.

In The Last Decade

María‐Ángeles Navas

24 papers receiving 824 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
María‐Ángeles Navas Spain 14 557 468 279 154 65 24 839
Ayse G. Kayali United States 14 413 0.7× 284 0.6× 140 0.5× 120 0.8× 64 1.0× 26 821
Shun Lu United States 15 500 0.9× 322 0.7× 155 0.6× 183 1.2× 50 0.8× 28 898
Jason S. Seidman United States 11 246 0.4× 177 0.4× 107 0.4× 89 0.6× 166 2.6× 17 680
Yasaman Aghazadeh Canada 12 428 0.8× 261 0.6× 145 0.5× 172 1.1× 19 0.3× 16 751
Manasvi S. Shah United States 14 444 0.8× 198 0.4× 228 0.8× 173 1.1× 20 0.3× 21 807
Shannon O’Dwyer Canada 11 736 1.3× 1.2k 2.5× 576 2.1× 478 3.1× 36 0.6× 15 1.5k
P. M. Nishina United States 6 265 0.5× 145 0.3× 99 0.4× 61 0.4× 78 1.2× 8 566
R V Rajotte Canada 11 156 0.3× 571 1.2× 357 1.3× 167 1.1× 24 0.4× 19 768
Martine Cordier–Bussat France 19 383 0.7× 304 0.6× 128 0.5× 246 1.6× 212 3.3× 34 900
Sylvie Renouf France 13 200 0.4× 190 0.4× 62 0.2× 222 1.4× 35 0.5× 19 644

Countries citing papers authored by María‐Ángeles Navas

Since Specialization
Citations

This map shows the geographic impact of María‐Ángeles Navas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by María‐Ángeles Navas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites María‐Ángeles Navas more than expected).

Fields of papers citing papers by María‐Ángeles Navas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by María‐Ángeles Navas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by María‐Ángeles Navas. The network helps show where María‐Ángeles Navas may publish in the future.

Co-authorship network of co-authors of María‐Ángeles Navas

This figure shows the co-authorship network connecting the top 25 collaborators of María‐Ángeles Navas. A scholar is included among the top collaborators of María‐Ángeles Navas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with María‐Ángeles Navas. María‐Ángeles Navas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Navas, María‐Ángeles, et al.. (2025). The PKA Signaling Pathway Regulates the Association of the Autophagy Initiation Complex With the Lipidation Machinery. Journal of Molecular Biology. 437(5). 168954–168954. 2 indexed citations
2.
Navas, María‐Ángeles, et al.. (2023). Coiled-coil-mediated dimerization of Atg16 is required for binding to the PROPPIN Atg21. Open Biology. 13(11). 230192–230192. 3 indexed citations
3.
Navas, María‐Ángeles, et al.. (2023). Effect of rapamycin on lysosomal accumulation in a CRISPR/Cas9‐based cellular model of VPS13A deficiency. Journal of Cellular and Molecular Medicine. 27(11). 1557–1564. 4 indexed citations
4.
Vincent, Olivier, et al.. (2021). The WIPI Gene Family and Neurodegenerative Diseases: Insights From Yeast and Dictyostelium Models. Frontiers in Cell and Developmental Biology. 9. 737071–737071. 13 indexed citations
5.
Escalante, Ricardo, et al.. (2021). A conserved ATG2 binding site in WIPI4 and yeast Hsv2 is disrupted by mutations causing β-propeller protein-associated neurodegeneration. Human Molecular Genetics. 31(1). 111–121. 12 indexed citations
6.
Vincent, Olivier, et al.. (2020). A novel reverse two-hybrid method for the identification of missense mutations that disrupt protein–protein binding. Scientific Reports. 10(1). 21043–21043. 7 indexed citations
7.
Oriola, Josep, et al.. (2018). Functional characterization of MODY2 mutations in the nuclear export signal of glucokinase. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(7). 2385–2394. 4 indexed citations
8.
Martı́nez, Rosa, et al.. (2017). Heterogeneity in phenotype of hyperinsulinism caused by activating glucokinase mutations: a novel mutation and its functional characterization. Clinical Endocrinology. 86(6). 778–783. 16 indexed citations
9.
Oriola, Josep, et al.. (2014). Lack of Glibenclamide Response in a Case of Permanent Neonatal Diabetes Caused by Incomplete Inactivation of Glucokinase. JIMD Reports. 20. 21–26. 4 indexed citations
10.
Rubio‐Cabezas, Oscar, Sharona Azriel, Olivier Vincent, et al.. (2012). Functional Characterization of MODY2 Mutations Highlights the Importance of the Fine-Tuning of Glucokinase and Its Role in Glucose Sensing. PLoS ONE. 7(1). e30518–e30518. 18 indexed citations
11.
Tinto, Nadia, Valentina Capobianco, Iolanda Coto, et al.. (2012). Glucokinase (GCK) Mutations and Their Characterization in MODY2 Children of Southern Italy. PLoS ONE. 7(6). e38906–e38906. 36 indexed citations
12.
Galán, María, et al.. (2010). Differential Effects of HNF-1α Mutations Associated with Familial Young-Onset Diabetes on Target Gene Regulation. Molecular Medicine. 17(3-4). 256–265. 31 indexed citations
13.
Sanz, Carmen, Patricia Vázquez, María‐Ángeles Navas, Elvira Álvarez, & Enrique Blázquez. (2007). Leptin but not neuropeptide Y up-regulated glucagon-like peptide 1 receptor expression in GT1-7 cells and rat hypothalamic slices. Metabolism. 57(1). 40–48. 13 indexed citations
14.
Sanz, Carmen, M. Isabel G. Roncero, Patricia Vázquez, María‐Ángeles Navas, & Enrique Blázquez. (2007). Effects of glucose and insulin on glucokinase activity in rat hypothalamus. Journal of Endocrinology. 193(2). 259–267. 18 indexed citations
15.
Galán, María, et al.. (2006). Functional analysis of human glucokinase gene mutations causing MODY2: exploring the regulatory mechanisms of glucokinase activity. Diabetologia. 50(2). 325–333. 48 indexed citations
16.
Navas, María‐Ángeles, Olivier Vincent, M. Isabel G. Roncero, et al.. (2006). Gene symbol: GCK. Disease: diabetes, MODY.. PubMed. 119(3). 363–363. 1 indexed citations
17.
Galán, María, Olivier Vincent, M. Isabel G. Roncero, et al.. (2005). Effects of novel maturity-onset diabetes of the young (MODY)-associated mutations on glucokinase activity and protein stability. Biochemical Journal. 393(1). 389–396. 45 indexed citations
18.
Navas, María‐Ángeles, et al.. (2000). The Human HNF-3 Genes: Cloning, Partial Sequence and Mutation Screening in Patients with Impaired Glucose Homeostasis. Human Heredity. 50(6). 370–381. 21 indexed citations
19.
Navas, María‐Ángeles, et al.. (1999). Functional characterization of the MODY1 gene mutations HNF4(R127W), HNF4(V255M), and HNF4(E276Q).. Diabetes. 48(7). 1459–1465. 49 indexed citations
20.
Gamo, Francisco‐Javier, María‐Ángeles Navas, Miguel Á. Blázquez, Carlos Gancedo, & Juana M. Gancedo. (1994). Catabolite inactivation of heterologous fructose‐1,6‐bisphosphatases and fructose‐1,6‐bisphosphatase‐β‐galactosidase fusion proteins inSaccharomyces cerevisiae. European Journal of Biochemistry. 222(3). 879–884. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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