Marcos Rivas

1.3k total citations
20 papers, 1.1k citations indexed

About

Marcos Rivas is a scholar working on Molecular Biology, Cell Biology and Ophthalmology. According to data from OpenAlex, Marcos Rivas has authored 20 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 7 papers in Cell Biology and 3 papers in Ophthalmology. Recurrent topics in Marcos Rivas's work include Cellular transport and secretion (6 papers), Endoplasmic Reticulum Stress and Disease (5 papers) and Glaucoma and retinal disorders (3 papers). Marcos Rivas is often cited by papers focused on Cellular transport and secretion (6 papers), Endoplasmic Reticulum Stress and Disease (5 papers) and Glaucoma and retinal disorders (3 papers). Marcos Rivas collaborates with scholars based in Spain, United States and Japan. Marcos Rivas's co-authors include Vytas A. Bankaitis, José R. Naranjo, Min Fang, Pilar Santisteban, Bradford S. Powell, Donald L. Court, Charles L. Turnbough, Zhigang Xie, Glenn D. Prestwich and Britt Mellström and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Marcos Rivas

19 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marcos Rivas Spain 14 780 452 192 130 105 20 1.1k
Amelia Chang United States 12 1.1k 1.4× 123 0.3× 156 0.8× 99 0.8× 56 0.5× 19 1.3k
J LaMendola United States 11 510 0.7× 355 0.8× 93 0.5× 93 0.7× 185 1.8× 16 987
R W Mercer United States 16 1.1k 1.4× 153 0.3× 137 0.7× 65 0.5× 185 1.8× 21 1.4k
Marjorie A. Phillips United States 22 699 0.9× 318 0.7× 79 0.4× 127 1.0× 67 0.6× 45 1.3k
Edgar F. da Cruz e Silva Portugal 23 1.1k 1.4× 443 1.0× 212 1.1× 47 0.4× 49 0.5× 46 1.6k
Jose M. Orozco United States 7 965 1.2× 269 0.6× 91 0.5× 45 0.3× 111 1.1× 8 1.3k
Ángel Román Spain 20 950 1.2× 201 0.4× 130 0.7× 25 0.2× 59 0.6× 51 1.4k
Susan E. Sadler United States 15 431 0.6× 145 0.3× 128 0.7× 86 0.7× 50 0.5× 23 911
Bob Monks United States 10 547 0.7× 46 0.1× 144 0.8× 108 0.8× 55 0.5× 10 825

Countries citing papers authored by Marcos Rivas

Since Specialization
Citations

This map shows the geographic impact of Marcos Rivas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marcos Rivas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marcos Rivas more than expected).

Fields of papers citing papers by Marcos Rivas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marcos Rivas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marcos Rivas. The network helps show where Marcos Rivas may publish in the future.

Co-authorship network of co-authors of Marcos Rivas

This figure shows the co-authorship network connecting the top 25 collaborators of Marcos Rivas. A scholar is included among the top collaborators of Marcos Rivas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marcos Rivas. Marcos Rivas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mann, Eric, Jeffrey A. Kammer, Jella An, et al.. (2025). Prospective 18-Month Study of Bimatoprost Intracameral Implant in Patients with Open-Angle Glaucoma or Ocular Hypertension in US Clinical Practice. Drugs. 85(3). 397–414. 1 indexed citations
2.
Cordeiro, M. Francesca, et al.. (2024). Prevalence of comorbidities with the potential to increase the risk of nonadherence to topical ocular hypotensive medication in patients with open-angle glaucoma. Current Medical Research and Opinion. 40(4). 647–655. 4 indexed citations
3.
Medeiros, Felipe A., Arsham Sheybani, Manjool Shah, et al.. (2022). Single Administration of Intracameral Bimatoprost Implant 10 µg in Patients with Open-Angle Glaucoma or Ocular Hypertension. Ophthalmology and Therapy. 11(4). 1517–1537. 19 indexed citations
4.
5.
Baczyk, Dora, Mark Kibschull, Britt Mellström, et al.. (2013). DREAM Mediated Regulation of GCM1 in the Human Placental Trophoblast. PLoS ONE. 8(1). e51837–e51837. 22 indexed citations
6.
Rivas, Marcos, et al.. (2011). Building the DREAM interactome. Science China Life Sciences. 54(8). 786–792. 16 indexed citations
7.
Rivas, Marcos, et al.. (2010). Redox Signaling Regulates Transcriptional Activity of the Ca 2+ -Dependent Repressor DREAM. Antioxidants and Redox Signaling. 14(7). 1237–1243. 14 indexed citations
8.
Rivas, Marcos, Britt Mellström, Begoña Torres, et al.. (2009). The DREAM Protein Is Associated with Thyroid Enlargement and Nodular Development. Molecular Endocrinology. 23(6). 862–870. 22 indexed citations
9.
Rivas, Marcos, Britt Mellström, Begoña Torres, et al.. (2009). The DREAM Protein Is Associated to Thyroid Enlargement and Nodular Development. The Journal of Clinical Endocrinology & Metabolism. 94(5). 1838–1838. 2 indexed citations
10.
Rivas, Marcos & José R. Naranjo. (2007). Thyroid hormones, learning and memory. Genes Brain & Behavior. 6(s1). 40–44. 91 indexed citations
11.
12.
Rivas, Marcos, Britt Mellström, José R. Naranjo, & Pilar Santisteban. (2004). Transcriptional Repressor DREAM Interacts with Thyroid Transcription Factor-1 and Regulates Thyroglobulin Gene Expression. Journal of Biological Chemistry. 279(32). 33114–33122. 61 indexed citations
13.
Rivas, Marcos & Pilar Santisteban. (2003). TSH-activated signaling pathways in thyroid tumorigenesis. Molecular and Cellular Endocrinology. 213(1). 31–45. 81 indexed citations
14.
Medina, Diego L., Marcos Rivas, Patricia de la Cruz‐Ojeda, et al.. (2002). RhoA Activation Promotes Transformation and Loss of Thyroid Cell Differentiation Interfering with Thyroid Transcription Factor-1 Activity. Molecular Endocrinology. 16(1). 33–44. 7 indexed citations
15.
Li, Xinmin, Marcos Rivas, Min Fang, et al.. (2002). Analysis of oxysterol binding protein homologue Kes1p function in regulation of Sec14p-dependent protein transport from the yeast Golgi complex. The Journal of Cell Biology. 157(1). 63–78. 207 indexed citations
16.
Rivas, Marcos, Brian G. Kearns, Zhigang Xie, et al.. (1999). Pleiotropic Alterations in Lipid Metabolism in Yeastsac1Mutants: Relationship to “Bypass Sec14p” and Inositol Auxotrophy. Molecular Biology of the Cell. 10(7). 2235–2250. 126 indexed citations
17.
Fang, Min, Marcos Rivas, & Vytas A. Bankaitis. (1998). The contribution of lipids and lipid metabolism to cellular functions of the Golgi complex. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1404(1-2). 85–100. 30 indexed citations
18.
19.
Xie, Zhigang, Min Fang, Marcos Rivas, et al.. (1998). Phospholipase D activity is required for suppression of yeast phosphatidylinositol transfer protein defects. Proceedings of the National Academy of Sciences. 95(21). 12346–12351. 154 indexed citations
20.
Powell, Bradford S., et al.. (1994). Rapid confirmation of single copy lambda prophage integration by PCR. Nucleic Acids Research. 22(25). 5765–5766. 202 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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