Marco Venanzoni

435 total citations
12 papers, 378 citations indexed

About

Marco Venanzoni is a scholar working on Oncology, Molecular Biology and Epidemiology. According to data from OpenAlex, Marco Venanzoni has authored 12 papers receiving a total of 378 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Oncology, 6 papers in Molecular Biology and 3 papers in Epidemiology. Recurrent topics in Marco Venanzoni's work include Viral-associated cancers and disorders (4 papers), Cancer-related Molecular Pathways (3 papers) and TGF-β signaling in diseases (2 papers). Marco Venanzoni is often cited by papers focused on Viral-associated cancers and disorders (4 papers), Cancer-related Molecular Pathways (3 papers) and TGF-β signaling in diseases (2 papers). Marco Venanzoni collaborates with scholars based in Italy, United States and Canada. Marco Venanzoni's co-authors include Arun Seth, Takis S. Papas, Hua Li, A Seth, David R. Hodge, J.L. Ostrowski, Barbara Beatty, I. Kola, Ismail Kola and Sergio Giunta and has published in prestigious journals such as Oncogene, Cell Death and Differentiation and International Journal of Cancer.

In The Last Decade

Marco Venanzoni

11 papers receiving 372 citations

Peers

Marco Venanzoni
Comparison fields: 5 of 62
  • Molecular Biology 220
  • Oncology 135
  • Cancer Research 95
  • Endocrinology, Diabetes and Metabolism 56
  • Genetics 43
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Citations per field, relative to Marco Venanzoni
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Citations per year, relative to Marco Venanzoni
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Countries citing papers authored by Marco Venanzoni

Since Specialization
Citations

This map shows the geographic impact of Marco Venanzoni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marco Venanzoni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marco Venanzoni more than expected).

Fields of papers citing papers by Marco Venanzoni

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marco Venanzoni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marco Venanzoni. The network helps show where Marco Venanzoni may publish in the future.

Co-authorship network of co-authors of Marco Venanzoni

This figure shows the co-authorship network connecting the top 25 collaborators of Marco Venanzoni. A scholar is included among the top collaborators of Marco Venanzoni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marco Venanzoni. Marco Venanzoni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
# Work Indexed citations
1 43
2 45
3 18
4 114
5 27
6 0
7
ETS1 and ETS2 in p53 regulation: spatial separation of ETS binding sites (EBS) modulate protein: DNA interaction.
56
8
Interaction of ETS-1 and ERGB/FLI-1 proteins with DNA is modulated by spacing between multiple binding sites as well as phosphorylation.
24
9 8
10 22
11 19
12
Nasopharyngeal carcinoma: the diagnostic value of the antibody-dependent, cellular cytotoxicity test and of EBV serology.
2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026