Marc Wiedner

442 total citations
12 papers, 242 citations indexed

About

Marc Wiedner is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Marc Wiedner has authored 12 papers receiving a total of 242 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Immunology. Recurrent topics in Marc Wiedner's work include DNA Repair Mechanisms (7 papers), CRISPR and Genetic Engineering (4 papers) and Cancer-related Molecular Pathways (3 papers). Marc Wiedner is often cited by papers focused on DNA Repair Mechanisms (7 papers), CRISPR and Genetic Engineering (4 papers) and Cancer-related Molecular Pathways (3 papers). Marc Wiedner collaborates with scholars based in Austria, United Kingdom and Germany. Marc Wiedner's co-authors include Joanna I. Loizou, Joana Ferreira da Silva, Georgia Velimezi, Jana Procházková, Abdelghani Mazouzi, Christoph Bock, Marina A. Lynch, Lukas Kenner, Katarzyna Drela and Marcin Jurga and has published in prestigious journals such as Nature Communications, Molecular Cell and Cancer Research.

In The Last Decade

Marc Wiedner

11 papers receiving 241 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marc Wiedner Austria 9 179 41 38 32 27 12 242
Brenton R. Paolella United States 10 198 1.1× 45 1.1× 68 1.8× 18 0.6× 8 0.3× 12 269
M. Victoria Simón Argentina 10 362 2.0× 33 0.8× 26 0.7× 17 0.5× 29 1.1× 12 495
Xuerong Sun China 11 257 1.4× 23 0.6× 79 2.1× 12 0.4× 17 0.6× 20 356
Charles A.C. Williams United Kingdom 6 159 0.9× 28 0.7× 44 1.2× 38 1.2× 6 0.2× 10 226
Mana Alshehri Saudi Arabia 9 148 0.8× 25 0.6× 31 0.8× 19 0.6× 8 0.3× 15 215
Ying Xia China 9 285 1.6× 47 1.1× 82 2.2× 34 1.1× 31 1.1× 20 434
Hedi Liu United States 7 269 1.5× 64 1.6× 47 1.2× 21 0.7× 12 0.4× 14 324
Olatz Arrizabalaga Spain 6 176 1.0× 68 1.7× 117 3.1× 79 2.5× 16 0.6× 7 282
Angélica Horrillo Spain 6 211 1.2× 50 1.2× 35 0.9× 26 0.8× 5 0.2× 7 309

Countries citing papers authored by Marc Wiedner

Since Specialization
Citations

This map shows the geographic impact of Marc Wiedner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marc Wiedner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marc Wiedner more than expected).

Fields of papers citing papers by Marc Wiedner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marc Wiedner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marc Wiedner. The network helps show where Marc Wiedner may publish in the future.

Co-authorship network of co-authors of Marc Wiedner

This figure shows the co-authorship network connecting the top 25 collaborators of Marc Wiedner. A scholar is included among the top collaborators of Marc Wiedner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marc Wiedner. Marc Wiedner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Valieris, Renan, Rodrigo D. Drummond, João Paulo da Silveira Nogueira Lima, et al.. (2021). Mutational Signatures Driven by Epigenetic Determinants Enable the Stratification of Patients with Gastric Cancer for Therapeutic Intervention. Cancers. 13(3). 490–490. 4 indexed citations
2.
Silva, Joana Ferreira da, Marc Wiedner, Paul Datlinger, et al.. (2019). Genome-scale CRISPR screens are efficient in non-homologous end-joining deficient cells. Scientific Reports. 9(1). 15751–15751. 17 indexed citations
3.
Silva, Joana Ferreira da, Thanos P. Mourikis, Peter Májek, et al.. (2019). Mapping the Human Kinome in Response to DNA Damage. Cell Reports. 26(3). 555–563.e6. 16 indexed citations
4.
Velimezi, Georgia, Francisco Muñoz‐Martínez, Wouter W. Wiegant, et al.. (2018). Map of synthetic rescue interactions for the Fanconi anemia DNA repair pathway identifies USP48. Nature Communications. 9(1). 2280–2280. 28 indexed citations
5.
Mazouzi, Abdelghani, Federica Battistini, Sarah C. Moser, et al.. (2017). Repair of UV-Induced DNA Damage Independent of Nucleotide Excision Repair Is Masked by MUTYH. Molecular Cell. 68(4). 797–807.e7. 29 indexed citations
6.
Velimezi, Georgia, Abdelghani Mazouzi, Marc Wiedner, et al.. (2017). Parallel genome-wide screens identify synthetic viable interactions between the BLM helicase complex and Fanconi anemia. Nature Communications. 8(1). 1238–1238. 23 indexed citations
7.
Mazouzi, Abdelghani, Alexey Stukalov, André C. Müller, et al.. (2016). A Comprehensive Analysis of the Dynamic Response to Aphidicolin-Mediated Replication Stress Uncovers Targets for ATM and ATMIN. Cell Reports. 15(4). 893–908. 25 indexed citations
8.
Procházková, Jana, Shinya Sakaguchi, Abdelghani Mazouzi, et al.. (2015). DNA Repair Cofactors ATMIN and NBS1 Are Required to Suppress T Cell Activation. PLoS Genetics. 11(11). e1005645–e1005645. 12 indexed citations
9.
Wiedner, Marc, et al.. (2014). ATMIN is required for the ATM-mediated signaling and recruitment of 53BP1 to DNA damage sites upon replication stress. DNA repair. 24. 122–130. 23 indexed citations
10.
Javaheri, Tahereh, Harini Nivarthi, Eleni M. Tomazou, et al.. (2014). Abstract 61: A mouse model for small round cell tumors induced by the Ewing sarcoma oncogene EWS/FLI1. Cancer Research. 74(19_Supplement). 61–61.
11.
McGuckin, Colin, Marcin Jurga, Anna Sarnowska, et al.. (2013). Ischemic brain injury: A consortium analysis of key factors involved in mesenchymal stem cell-mediated inflammatory reduction. Archives of Biochemistry and Biophysics. 534(1-2). 88–97. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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