Charles A.C. Williams

459 total citations
10 papers, 226 citations indexed

About

Charles A.C. Williams is a scholar working on Molecular Biology, Cell Biology and Immunology. According to data from OpenAlex, Charles A.C. Williams has authored 10 papers receiving a total of 226 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Cell Biology and 2 papers in Immunology. Recurrent topics in Charles A.C. Williams's work include Pluripotent Stem Cells Research (4 papers), CRISPR and Genetic Engineering (3 papers) and Immunotherapy and Immune Responses (2 papers). Charles A.C. Williams is often cited by papers focused on Pluripotent Stem Cells Research (4 papers), CRISPR and Genetic Engineering (3 papers) and Immunotherapy and Immune Responses (2 papers). Charles A.C. Williams collaborates with scholars based in United Kingdom, United States and Denmark. Charles A.C. Williams's co-authors include Steven M. Pollard, Abdenour Soufi, Greg M. Findlay, Neza Alfazema, Carla Blin, Benjamin Southgate, María Ángeles Marqués‐Torrejón, Ryan J. H. West, Christopher Elliott and Rachel Toth and has published in prestigious journals such as Nature Communications, Genes & Development and Journal of Molecular Biology.

In The Last Decade

Charles A.C. Williams

9 papers receiving 225 citations

Peers

Charles A.C. Williams

MB

CR

GENET

DN

ONCOL

Giuseppe Aiello Italy

Diana Aguilar‐Morante Spain

Lina Dahl Sweden

Brenton R. Paolella United States

Vilma Rraklli Sweden

Samuel O. Lawn Canada

Claudio Ballabio Italy

Marc Wiedner Austria

Sara J. Fenstermacher United States

John DeSisto United States

Giuseppe Aiello Italy

Charles A.C. Williams

135 ×0.8

MB

34 ×0.8

CR

65 ×1.7

GENET

29 ×0.8

DN

56 ×2.0

ONCOL

Citations per year, relative to Charles A.C. Williams Charles A.C. Williams (= 1×) peers Giuseppe Aiello

Countries citing papers authored by Charles A.C. Williams

Since Specialization

Citations

This map shows the geographic impact of Charles A.C. Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles A.C. Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles A.C. Williams more than expected).

Fields of papers citing papers by Charles A.C. Williams

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Charles A.C. Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles A.C. Williams. The network helps show where Charles A.C. Williams may publish in the future.

Co-authorship network of co-authors of Charles A.C. Williams

This figure shows the co-authorship network connecting the top 25 collaborators of Charles A.C. Williams. A scholar is included among the top collaborators of Charles A.C. Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Charles A.C. Williams. Charles A.C. Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

10 of 10 papers shown

1.

Williams, Charles A.C., Dounia Djeghloul, Nicolás Veland, et al.. (2025). SOX2 phosphorylation during mitosis limits genomic damage. Genes & Development. 40(3-4). 185–198.

2.

Williams, Charles A.C., et al.. (2023). Identification of the novel HLA‐C*03:648 allele by next generation sequencing. HLA. 103(1). e15324–e15324. 2 indexed citations

3.

Williams, Charles A.C., et al.. (2023). Identification of the novel HLA‐B*57:168 allele by next generation sequencing. HLA. 103(1). e15327–e15327. 1 indexed citations

4.

Marqués‐Torrejón, María Ángeles, Charles A.C. Williams, Benjamin Southgate, et al.. (2021). LRIG1 is a gatekeeper to exit from quiescence in adult neural stem cells. Nature Communications. 12(1). 2594–2594. 59 indexed citations

5.

Bressan, Raul Bardini, Benjamin Southgate, Kirsty M. Ferguson, et al.. (2021). Regional identity of human neural stem cells determines oncogenic responses to histone H3.3 mutants. Cell stem cell. 28(5). 877–893.e9. 49 indexed citations

6.

Williams, Charles A.C., Abdenour Soufi, & Steven M. Pollard. (2019). Post-translational modification of SOX family proteins: Key biochemical targets in cancer?. Seminars in Cancer Biology. 67(Pt 1). 30–38. 39 indexed citations

7.

Williams, Charles A.C., Nathanael S. Gray, & Greg M. Findlay. (2017). A Simple Method to Identify Kinases That Regulate Embryonic Stem Cell Pluripotency by High-throughput Inhibitor Screening. Journal of Visualized Experiments. 2 indexed citations

8.

Bustos, Francisco, et al.. (2017). Protein Kinases in Pluripotency—Beyond the Usual Suspects. Journal of Molecular Biology. 429(10). 1504–1520. 14 indexed citations

9.

Williams, Charles A.C., et al.. (2016). Erk5 Is a Key Regulator of Naive-Primed Transition and Embryonic Stem Cell Identity. Cell Reports. 16(7). 1820–1828. 34 indexed citations

10.

West, Ryan J. H., et al.. (2015). Neurophysiology ofDrosophilaModels of Parkinson’s Disease. Parkinson s Disease. 2015. 1–11. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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